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BNKN602 - NURSING THE PERSON WITH COPD EXAM QUESTIONS AND ANSWERS WITH COMPLETE SOLUTIONS

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BNKN602 - NURSING THE PERSON WITH COPD EXAM QUESTIONS AND ANSWERS WITH COMPLETE SOLUTIONS What are the two main characteristics of COPD emphysema and chronic bronchitis What is emphysema loss of elasticity allows alveolu to expand with inspiration but reduces its ability to recoil with exiration. this results in co2 being trapped and increases alveolar pco2 What results due to an increased PCO2 co2 cannot diffuse readily from pulmonary capillaries causing a build up of co2 in arterial blood and results in hypercapnia. accumulation of damage causes large spaces to develop which reduces surface area and decreases gas exchange between alveoli and pulmonary capillaries Expected airway findings of exacerbation of copd airway patent but potentially under threat if decreased level of conciousness due to hypercapnia and hypoxaemia Expected breathing findings of exacerbation of copd increased WOB due to hypercapnia, pursed lip breathing, tachypnoeic, sp02 90% due to hypoxaemia, upright or tripod position to expand thoracic cavity and maximise air entry for gas exchange, adventitious sounds - wheeze due to bronchoconstriction, possible decreased air entry due to alveolar collapse, resonant percussion due to hyperinflation Expected circulation findings of acute exacerbation of COPD cool pale peripheries and/or cyanosis due to hypoxaemia, CRT 3 seconds due to peripheral vasoconstriction, possible elevated temperature due to increased metabolic

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BNKN602 - NURSING THE PERSON WITH COPD EXAM

QUESTIONS AND ANSWERS WITH COMPLETE

SOLUTIONS


What are the two main characteristics of COPD

emphysema and chronic bronchitis

What is emphysema

loss of elasticity allows alveolu to expand with inspiration but reduces its ability to recoil

with exiration. this results in co2 being trapped and increases alveolar pco2

What results due to an increased PCO2

co2 cannot diffuse readily from pulmonary capillaries causing a build up of co2 in

arterial blood and results in hypercapnia. accumulation of damage causes large spaces

to develop which reduces surface area and decreases gas exchange between alveoli

and pulmonary capillaries

Expected airway findings of exacerbation of copd

airway patent but potentially under threat if decreased level of conciousness due to

hypercapnia and hypoxaemia

Expected breathing findings of exacerbation of copd

increased WOB due to hypercapnia, pursed lip breathing, tachypnoeic, sp02 <90% due

to hypoxaemia, upright or tripod position to expand thoracic cavity and maximise air

entry for gas exchange, adventitious sounds - wheeze due to bronchoconstriction,

, possible decreased air entry due to alveolar collapse, resonant percussion due to

hyperinflation

Expected circulation findings of acute exacerbation of COPD

cool pale peripheries and/or cyanosis due to hypoxaemia, CRT >3 seconds due to

peripheral vasoconstriction, possible elevated temperature due to increased metabolic

rate or infection, diaphoresis due to increased WOB or hypercapnia, tachycardia due to

hypoxaemia or bronchodilators, possible HTN dye to hypoxaemia or systemic

inflammatory response

Expected disability findings of acute exacerbation of hypoxaemia

possible disorientation or confusion to dye hypoxaemia/hypoxia, reduced level of

consciousness dye to hypercapnia and hypoxaemia, anxiety or fear of dying due to

dyspnoea

expected environment findings of acute exacerbation of copd

elevated EWS due to >RR, HR and <spo2, increased falls risk due to dyspnoea,

dizziness and/or confusion, decreased braden score due to corticosteroids, reduced

mobility and possible oedema, ex/current smoker, COLDSPA or irritants/triggers,

identify allergies or drug reactions

Airway nursing interventions for a pt with copd

ensure correct positioning to open airway and maintain patency

Breathing nursing interventions for a pt with copd

ensure patient is upright to optimise air entry and facilitate gas exchange, encourage

pursed lip breathing to facilitate exhalation of CO2 and reduce dyspnoea, administer

bronchodilators as prescribed for relaxation of smooth muscle in airways to facilitate gas

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