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VASCULAR REGISTRY CCI REVIEW QUESTIONS & ANSWERS 100% CORRECT!!

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Abdominal Aorta waveform(s) - ANSWERLow resistance proximal, Higher resistance beyond renals Celiac Artery supplies - ANSWERLiver, spleen, stomach, & proximal small bowel Branches of the Abdominal AO - ANSWER1st major-Celiac artery (trunk/axis) 2nd major-SMA Renals 3rd major-IMA (after renals) Celiac Axis - ANSWERBranches into Common Hepatic (to right), Splenic, & Left Gastric (off left) Common Hepatic Arteries - ANSWERGives rise to the Gastroduodenal artery in PANC head & divides into Rt & Lt Hepatics Splenic Artery - ANSWERBranches left and posteriosuperior to PANC body/tail SMA/IMA waveforms - ANSWERHigh resistance preprandial/Low resistance postprandial Portal vein is usually formed by the confluence of - ANSWERSMV & Splenic veins *It also receives blood from the inferior mesenteric, gastric, and cystic veins Portals walls/waveforma - ANSWERechogenic walls & phasic waveforms Renal veins are formed by - ANSWERrenal tributaries Left Renal Vein - ANSWERLonger than Rt.; Receives suprarenal/Gonadal vein Left Renal pathway - ANSWERAnterior to AO; Posterior to SMA Right Renal Vein - ANSWERNo tributaries; shorter Hepatic Veins - ANSWERHepatofugal flow; from liver to IVC Patient status for Abdominal Vascular Imaging - ANSWERNPO 8-12 hours Ectasia - ANSWERLocal diameter increase with small bulge (20% increase for Ao 3cm) AAA growth rate - ANSWER1-2mm/year until 3-4cm; 5 mm/yr 4cm Aneurysm classification - ANSWER2-3cm; 3-4cm for AAA SMA supplies - ANSWERBowel from duodenum to prox small bowel IMA supplies - ANSWERBowel descending & rectosigmoid colon Right Renal Artery - ANSWERBranches anterolateral, posterior to IVC Left Renal Artery - ANSWERBranches posterolateral Renal Artery waveform - ANSWERLow resistance AAA Intervention - ANSWER5.5cm (high risk for rupture-catastrophic) Fusiform - ANSWERConcentric enlargement; All 3 layers intact Saccular - ANSWEREccentric enlargement; All 3 layers compromised; Less common (1%); Usually in Thoracic Ao Types of Saccular AAA - ANSWER1-Cannula Placement 2-Mycotic aneurysm (bacterial infection Ao wall) 3-Vasculitis (Inflammatory process) 4-Penetrating ulcer rupture into media Vasculitis/Aortitis - ANSWERInflammatory process in wall of Ao beginning with outer (adventitia) layer and moving inward; ie: Takayasu's Di

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VASCULAR REGISTRY CCI REVIEW
QUESTIONS & ANSWERS 100%
CORRECT!!
Abdominal Aorta waveform(s) - ANSWERLow resistance proximal, Higher resistance
beyond renals

Celiac Artery supplies - ANSWERLiver, spleen, stomach, & proximal small bowel

Branches of the Abdominal AO - ANSWER1st major-Celiac artery (trunk/axis)
2nd major-SMA
Renals
3rd major-IMA (after renals)

Celiac Axis - ANSWERBranches into Common Hepatic (to right), Splenic, & Left
Gastric (off left)

Common Hepatic Arteries - ANSWERGives rise to the Gastroduodenal artery in
PANC head & divides into Rt & Lt Hepatics

Splenic Artery - ANSWERBranches left and posteriosuperior to PANC body/tail

SMA/IMA waveforms - ANSWERHigh resistance preprandial/Low resistance
postprandial


Portal vein is usually formed by the confluence of - ANSWERSMV & Splenic veins
*It also receives blood from the inferior mesenteric, gastric, and cystic veins

Portals walls/waveforma - ANSWERechogenic walls & phasic waveforms

Renal veins are formed by - ANSWERrenal tributaries

Left Renal Vein - ANSWERLonger than Rt.; Receives suprarenal/Gonadal vein

Left Renal pathway - ANSWERAnterior to AO; Posterior to SMA

Right Renal Vein - ANSWERNo tributaries; shorter

Hepatic Veins - ANSWERHepatofugal flow; from liver to IVC

Patient status for Abdominal Vascular Imaging - ANSWERNPO 8-12 hours

Ectasia - ANSWERLocal diameter increase with small bulge
(20% increase for Ao <3cm)

,AAA growth rate - ANSWER1-2mm/year until 3-4cm; 5 mm/yr >4cm

Aneurysm classification - ANSWER2-3cm; 3-4cm for AAA

SMA supplies - ANSWERBowel from duodenum to prox small bowel

IMA supplies - ANSWERBowel descending & rectosigmoid colon

Right Renal Artery - ANSWERBranches anterolateral, posterior to IVC

Left Renal Artery - ANSWERBranches posterolateral

Renal Artery waveform - ANSWERLow resistance

AAA Intervention - ANSWER5.5cm (high risk for rupture-catastrophic)

Fusiform - ANSWERConcentric enlargement; All 3 layers intact

Saccular - ANSWEREccentric enlargement; All 3 layers compromised; Less
common (<1%); Usually in Thoracic Ao

Types of Saccular AAA - ANSWER1-Cannula Placement
2-Mycotic aneurysm (bacterial infection Ao wall)
3-Vasculitis (Inflammatory process)
4-Penetrating ulcer rupture into media

Vasculitis/Aortitis - ANSWERInflammatory process in wall of Ao beginning with outer
(adventitia) layer and moving inward; ie: Takayasu's

Dissection - ANSWERIntimal wall compromised resulting in 2 lumens
false>true; flow reversal

Type 1 (a/b) endoleak - ANSWERLeak in anastamosis of graft at (a) prox or (b)
distal end

Type 2 endoleak - ANSWERAorta branch vessel; exhibits retrograde flow; more
dangerous b/c internally bleeding

Type 3/4 endoleak - ANSWER(3) Junction of modular components; (4) Trans graft
flow-graft defect

Chronic Mesenteric Ischemia - ANSWER"Fear of Food" 95% of Bowel Ischemia
cases
Atherosclerotic stenosis/occlusion in main mesenteric arteries: >70% stenosis in 2/3
of principle mesenteric arteries

Ischemia diagnosis criteria via Moneta - ANSWERCeliac >200cm/s
SMA >275cm/s

,Median Arcuate Ligament Syndrome (MALS) - ANSWERArch impedes on Celiac
during EXPIRATION (non-compressed during inhalation)

Measurement(s) of Splenic Vein - ANSWER7-17 cm long; 5-10mm diameter

Portal vein diameter - ANSWER<13mm

Blood supply to liver - ANSWER75% from Portal VEIN; 25% from Hepatic ARTERY

Portal vein carries ____________ to the liver - ANSWERNutrients

Hepatic artery carries ______________ to the liver - ANSWEROxygen

Portal Hypertension - ANSWERExtrahepatic, Hyperdynamic, Intrahepatic (more
common)

Extrahepatic Portal HTN - ANSWERPrehepatic (Portal/splenic vein thrombus,
Extrinsic compression of Potral vein)
Posthepatic (IVC/Hepatic vein obstruction)

Hyperdynamic Portal HTN - ANSWERAV malformation causing arterial portal fistulas

Intrahepatic (within liver) - ANSWERPresinusodial (less common)
Postsinusoidial (more common)
Cirrhosis/Venoclusive disease
Small liver, large spleen, ascites

LaPlace's law - ANSWERLarger vessel radius, larger wall tension to compensate for
extra pressure

Hydrostatic pressure - ANSWERGravitational

Large vessels serve as - ANSWERPressure reservoirs

Vasodilation - ANSWERStretch to absorb

Vasoconstriction - ANSWERshrink/squeeze

Energy and stenosis - ANSWERProx- PE↑, KE↓(highest total energy)
Within-PE↓, KE↑ (lower TE, Bernouille's)
Distal-PE↑, KE↓ (lowest total energy)

A-Early Systole (Forward flow to periphery)
B-Peak Systole (Store PE)
C-Late Systole (Temporary reversal-Peripheral resistance)
D-Early Diastole (Forward-reduced resistance)
E-Late Diastole (Vessel Recoil/Vasoconstrict/PE turns KE) - ANSWER

Brain - ANSWERSupplied by ICA & Vertebrals
2% of Body's weight

, 15% CO
20% Total blood supply

3-8 minutes of oxygen deprivation results in - ANSWERcellular death

Bovine Arch - ANSWERCommon origin of Lt. CCA and Innominate

ICA - ANSWERTerminates into MCA/ACA and feeds the brain, forehead, eyes, &
nose-70-80% from CCA

ECA - ANSWERDoes not feed brain unless needed as collateral circulation

ECA supplies - ANSWERNeck, face, scalp

ECA Branches - ANSWERSuperior Thyroid
Ascending Pharyngeal
Lingual
Facial
Occipital
Posterior Auricular
Maxillary
Superficial Thyroid

Vertebral supply - ANSWERMedulla/Inferior cerebellum

Basilar supply - ANSWERPons/superior anterior cerebellum

Circle of Willis has atleast ____ variations with the most common being
___________________ - ANSWER9; the absence of one or both communicating
arteries

What portion of the population has an incomplete CofW? - ANSWER50%

What portion of the population has a complete Cof W? - ANSWER20-25%

Anatomic interrogation - ANSWERB-mode/2D best

Physiologic/hemodynamic interrogation - ANSWERSpectral/Doppler best

Hemorrhagic Stroke - ANSWERBleed; HTN

Ischemic Stroke - ANSWEROxygen interruption; Blood clot/emboli from
Atherosclerosis

Which stroke is known to be the 3rd leading cause of death? - ANSWERIschemic
Stroke

Small perforating artery obstruction - ANSWEROccurs in elderly/diabetics

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