Inultiple Myeloma
plasma cell neoplasm asst
BRAH lytis Bone lesions
Renal insufficiency Acg immune abnormalities
Hypercalcemia
Plasma
Yanomal ab
4ᵗʰ normal
y
Fat
protein
mm
it i M
a
abnormalab
poi
IF hair
Sathogenesis
mutations are seen in I Btsprotein
19H locus
proto oncogenes
cellcycleregulators cyclin D 3
tumor suppressor deletion TP 53
MYC
GoFm in NF KB pathway
level of 11 6 Cgf for plasmacells
MI
0Pa
Ellie cell
growthsur
at Erik
jp
RI
mgn
C.IE
RANKL binding activates
osteoclast
it is inhibited
Eiatiigfj
byOPGfrom 0blast
plasma cell neoplasm asst
BRAH lytis Bone lesions
Renal insufficiency Acg immune abnormalities
Hypercalcemia
Plasma
Yanomal ab
4ᵗʰ normal
y
Fat
protein
mm
it i M
a
abnormalab
poi
IF hair
Sathogenesis
mutations are seen in I Btsprotein
19H locus
proto oncogenes
cellcycleregulators cyclin D 3
tumor suppressor deletion TP 53
MYC
GoFm in NF KB pathway
level of 11 6 Cgf for plasmacells
MI
0Pa
Ellie cell
growthsur
at Erik
jp
RI
mgn
C.IE
RANKL binding activates
osteoclast
it is inhibited
Eiatiigfj
byOPGfrom 0blast