NUR 552 Exam 1 questions with verified answers
5A's of smoking cessation Ans✓✓✓ 1. Ask
2. Advise
3. Assess
4. Assist
5. Arrange
Acamprosate (Campral) Ans✓✓✓ Likely modulates glutamate transmission
First line trx in maintaining abstinence after detox
Used for relapse prevention(post detoxification)
Can be used in liver disease-not metabolized by liver
Can be administered to patients with hepatitis, liver dz and those who continue to
drink alcohol
Contraindicated in severe renal disease
Decreases craving
ADHD nonstimulant medications Ans✓✓✓ Atomoxetine (Strattera); Bupropion
(Wellbutrin) ; Alpha 2 adrenergic agonist (Clonidine, Guanfacine)
ADHD treatment Ans✓✓✓ •2nd line: Alpha-2 agonists (Clonidine, guanfacine)
•****Monitor Height, weight, BP, CBC w/ diff; Pulse quarterly (Height and weight
d/t risk of growth restriction)
•In healthy individuals, it is not necessary to obtain an EKG prior to initiating a
stimulant
•Prescription Monitoring Program should be checked
,•Note: With stimulants, evidence of growth suppression is not clear, seems
transient and resolves in mid-adolescence
•
•Note: If a child is taking their medication twice daily (i.e. at home and school)
and parents request to solely administer the medication= consider switching to an
extended release form (e.g. Methylphenidate CD= extended release)
ADHD treatment: Considering non-stimulant(factors to consider) Ans✓✓✓
•Stimulants are not working well to control ADHD symptoms
•Stimulants cause too many side effects (often intolerable) - e.g. anxiety
•The child or teen has problems with substance abuse
•The child or teen has a medical condition for which stimulants cannot be used -
e.g. tic disorder
•Adjunctive therapy for stimulants
Aggression/Agitation/Psychosis Ans✓✓✓ Consider atypical antipsychotics
zyprexa, seroquel, risperdal,haldol
Note: Reserve Benzos for short-term and acute episodes
Alchohol withdrawal Ans✓✓✓ Mild: Insomnia, irritability, Hand tremor
Moderate: Autonomic hyperactivity(diaphoresis, tachy, HTN), HTN
Severe: Seizures(12-48 hours consumption); hallucinations; delium tremens(48-96
hrs after last drink)
Anxiety
Anorexia
Nausea/Vomiting
, Psychomotor agitation
Use CIWA to monitor withdrawal
Alcohol intoxication Ans✓✓✓ Impaired fine motor control
Impaired judgment and coordination
Ataxic gait and poor balance
Lethargic, difficulty sitting upright, difficulty with memory
Nausea/Vomiting
Coma=Levels 300mg/dl and over
Respiratory depression and death possible
Alcohol Intoxication/withdrawal treatments Ans✓✓✓ Benzos(lorazepam,
diazepam, chlordiazepoxide-librium)= to keep patient and lightly sedated
MOA: enhances the effects of GABA
Alzheimer's disease Ans✓✓✓ -Gradual progressive decline
-The most common type
-Affects memory, learning, and language
-Aphasia(difficulty with speech
-Apraxia(inability to perform previous learned tasks)
-Agnosia(inability to recognize an object)
Etiology: Accumulation of beta-amyloid plaques and intraneuronal tau protein
tangles
Txt: Cholinesterase inhibitors
NMDA receptor antagonists
5A's of smoking cessation Ans✓✓✓ 1. Ask
2. Advise
3. Assess
4. Assist
5. Arrange
Acamprosate (Campral) Ans✓✓✓ Likely modulates glutamate transmission
First line trx in maintaining abstinence after detox
Used for relapse prevention(post detoxification)
Can be used in liver disease-not metabolized by liver
Can be administered to patients with hepatitis, liver dz and those who continue to
drink alcohol
Contraindicated in severe renal disease
Decreases craving
ADHD nonstimulant medications Ans✓✓✓ Atomoxetine (Strattera); Bupropion
(Wellbutrin) ; Alpha 2 adrenergic agonist (Clonidine, Guanfacine)
ADHD treatment Ans✓✓✓ •2nd line: Alpha-2 agonists (Clonidine, guanfacine)
•****Monitor Height, weight, BP, CBC w/ diff; Pulse quarterly (Height and weight
d/t risk of growth restriction)
•In healthy individuals, it is not necessary to obtain an EKG prior to initiating a
stimulant
•Prescription Monitoring Program should be checked
,•Note: With stimulants, evidence of growth suppression is not clear, seems
transient and resolves in mid-adolescence
•
•Note: If a child is taking their medication twice daily (i.e. at home and school)
and parents request to solely administer the medication= consider switching to an
extended release form (e.g. Methylphenidate CD= extended release)
ADHD treatment: Considering non-stimulant(factors to consider) Ans✓✓✓
•Stimulants are not working well to control ADHD symptoms
•Stimulants cause too many side effects (often intolerable) - e.g. anxiety
•The child or teen has problems with substance abuse
•The child or teen has a medical condition for which stimulants cannot be used -
e.g. tic disorder
•Adjunctive therapy for stimulants
Aggression/Agitation/Psychosis Ans✓✓✓ Consider atypical antipsychotics
zyprexa, seroquel, risperdal,haldol
Note: Reserve Benzos for short-term and acute episodes
Alchohol withdrawal Ans✓✓✓ Mild: Insomnia, irritability, Hand tremor
Moderate: Autonomic hyperactivity(diaphoresis, tachy, HTN), HTN
Severe: Seizures(12-48 hours consumption); hallucinations; delium tremens(48-96
hrs after last drink)
Anxiety
Anorexia
Nausea/Vomiting
, Psychomotor agitation
Use CIWA to monitor withdrawal
Alcohol intoxication Ans✓✓✓ Impaired fine motor control
Impaired judgment and coordination
Ataxic gait and poor balance
Lethargic, difficulty sitting upright, difficulty with memory
Nausea/Vomiting
Coma=Levels 300mg/dl and over
Respiratory depression and death possible
Alcohol Intoxication/withdrawal treatments Ans✓✓✓ Benzos(lorazepam,
diazepam, chlordiazepoxide-librium)= to keep patient and lightly sedated
MOA: enhances the effects of GABA
Alzheimer's disease Ans✓✓✓ -Gradual progressive decline
-The most common type
-Affects memory, learning, and language
-Aphasia(difficulty with speech
-Apraxia(inability to perform previous learned tasks)
-Agnosia(inability to recognize an object)
Etiology: Accumulation of beta-amyloid plaques and intraneuronal tau protein
tangles
Txt: Cholinesterase inhibitors
NMDA receptor antagonists
