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NR507 MIDTERM EXAM 2 QUESTIONS AND ANSWERS WITH COMPLETE SOLUTIONS VERIFIED

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NR507 MIDTERM EXAM 2 QUESTIONS AND ANSWERS WITH COMPLETE SOLUTIONS VERIFIED Practice questions for this set Terms in this set (145) Sickle Cell Anemia Involves a single amino acid change on the beta-chain. Sickle Cell Anemia Increased red blood cell (RBC) hemoglobin S concentration, RBC dehydration, acidosis, and hypoxemia. Thalassemia May have many possible genetic mutations. Thalassemia Ineffective erythropoiesis. Thalassemia Occurs primarily in persons from southeast Asia and China. Thalassemia Inherits an abnormal Hb gene from both parents. Sickle Cell Anemia Autosomal recessive genetic disorder. Coronary Artery Disease (CAD) Mainly the result of longstanding atherosclerosis. Flow of blood between the heart and lungs Oxygenated blood is transported from the lungs to the heart via the pulmonary veins. Modifiable risk factor for CAD Obesity. Non-modifiable risk factor for CAD Age, gender, family history. Patient with sickle cell anemia At high risk for stroke. Cells with abnormal hemoglobin More susceptible to infection by the parasite that causes malaria. Patient with thalassemia Not at high risk for stroke. Geographic prevalence of sickle cell anemia and thalassemia More prevalent in Africa. Coronary Artery Disease (CAD) Pumping ability of the heart can be impaired due to the deprivation of oxygen. Hypertension Has its most immediate effect on afterload. Cor Pulmonale Right ventricular failure secondary to pulmonary hypertension. Preload Decrease Causes Hemorrhage. Right-Sided Heart Failure Cause The most common cause is pulmonary hypertension. Healthy Heart Response to Increased Preload The stroke volume increases. Stage B Heart Failure Patients with structural heart disease who have not yet developed symptoms of heart failure. Stage D Heart Failure Patients with refractory heart failure that require advanced intervention (e.g., pacemaker). Stage A Heart Failure Patients at risk for heart failure who have not yet developed structural heart changes. Stage C Heart Failure Patients who have developed clinical heart failure. Class II Heart Failure There is a slight limitation of physical activity; the patient is comfortable at rest, but ordinary physical activity results in symptoms of HF. Class IV Heart Failure The patient is unable to carry on any physical activity without symptoms of HF, or they have symptoms of HF at rest. Class I Heart Failure There is no limitation of physical activity; ordinary physical activity does not cause symptoms of HF. Class III Heart Failure There is marked limitation of physical activity; the patient is comfortable at rest, but less than ordinary activity causes symptoms of HF. Right-Sided Heart Failure Symptoms Jugular vein distention, hepatosplenomegaly, peripheral edema, cor pulmonale, tricuspid valve damage. Left-Sided Heart Failure Symptoms Increased left ventricular afterload, decreased ejection fraction, increased left ventricular preload, pulmonary edema, dyspnea. Aortic Regurgitation Presentation An early high-pitched diastolic murmur heard at the left lower sternal border, a diastolic rumbling murmur heard at the apex of the heart, a systolic crescendo- decrescendo murmur heard at the left upper sternal border. Mitral regurgitation A condition where a patient would most likely present with a blowing, holosystolic murmur. Mitral stenosis A condition where a patient would most likely present with a rumbling, decrescendo diastolic murmur heard at the apex of the heart. Aortic stenosis A condition where a patient would most likely present with a mid-systolic crescendo-decrescendo murmur. Rheumatic fever A common cause for mitral stenosis. Forced Vital Capacity (FVC) A patient with normal lungs should be able to exhale 80% of the forced vital capacity within the first second. FEV1/FVC ratio A severely reduced ratio indicates chronic asthma. Restrictive lung disease Reflected by normal FEV1, FVC, and FEV1/FVC ratio with reduced total lung capacity. Simple spirometry Can measure all of the following EXCEPT residual volume.

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3/24/25, 6:00 NR507 Midterm Exam 2 Flashcards |
PM
NR507 MIDTERM EXAM 2 QUESTIONS AND ANSWERS WITH COMPLETE
SOLUTIONS VERIFIED
Practice questions for this set


Learn 1/7 Study using Learn




Facial rash confined to the cheeks can be used to diagnose.



Select the correct term




1Rheumatoid Arthritis (RA) 2Psoriatic arthritis




Acute cutaneous lupus erythematosus
3 4Systemic Lupus Erythematosus (SLE)
(acle)



Don't know?




Terms in this set (145)


Sickle Cell Anemia Involves a single amino acid change on the beta-chain.

Increased red blood cell (RBC) hemoglobin S concentration, RBC dehydration,
Sickle Cell Anemia
acidosis, and hypoxemia.

Thalassemia May have many possible genetic mutations.

Thalassemia Ineffective erythropoiesis.

Thalassemia Occurs primarily in persons from southeast Asia and China.

Thalassemia Inherits an abnormal Hb gene from both parents.

Sickle Cell Anemia Autosomal recessive genetic disorder.

Coronary Artery Disease (CAD) Mainly the result of longstanding atherosclerosis.

Oxygenated blood is transported from the lungs to the heart via the pulmonary
Flow of blood between the heart and lungs
veins.

Modifiable risk factor for CAD Obesity.

Non-modifiable risk factor for CAD Age, gender, family history.

Patient with sickle cell anemia At high risk for stroke.

Cells with abnormal hemoglobin More susceptible to infection by the parasite that causes malaria.

Patient with thalassemia Not at high risk for stroke.




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5

, 3/24/25, 6:00 NR507 Midterm Exam 2 Flashcards |
PM
Geographic prevalence of sickle cell More prevalent in Africa.
anemia and thalassemia

Coronary Artery Disease (CAD) Pumping ability of the heart can be impaired due to the deprivation of oxygen.

Hypertension Has its most immediate effect on afterload.

Cor Pulmonale Right ventricular failure secondary to pulmonary hypertension.

Preload Decrease Causes Hemorrhage.

Right-Sided Heart Failure Cause The most common cause is pulmonary hypertension.

Healthy Heart Response to Increased The stroke volume increases.
Preload

Patients with structural heart disease who have not yet developed symptoms of
Stage B Heart Failure
heart failure.

Patients with refractory heart failure that require advanced intervention (e.g.,
Stage D Heart Failure
pacemaker).

Stage A Heart Failure Patients at risk for heart failure who have not yet developed structural heart changes.

Stage C Heart Failure Patients who have developed clinical heart failure.

There is a slight limitation of physical activity; the patient is comfortable at rest, but
Class II Heart Failure
ordinary physical activity results in symptoms of HF.

The patient is unable to carry on any physical activity without symptoms of HF, or
Class IV Heart Failure
they have symptoms of HF at rest.

There is no limitation of physical activity; ordinary physical activity does not cause
Class I Heart Failure
symptoms of HF.

There is marked limitation of physical activity; the patient is comfortable at rest, but
Class III Heart Failure
less than ordinary activity causes symptoms of HF.

Jugular vein distention, hepatosplenomegaly, peripheral edema, cor pulmonale,
Right-Sided Heart Failure Symptoms
tricuspid valve damage.

Increased left ventricular afterload, decreased ejection fraction, increased left
Left-Sided Heart Failure Symptoms
ventricular preload, pulmonary edema, dyspnea.

An early high-pitched diastolic murmur heard at the left lower sternal border,
Aortic Regurgitation Presentation a diastolic rumbling murmur heard at the apex of the heart, a systolic
crescendo- decrescendo murmur heard at the left upper sternal border.

A condition where a patient would most likely present with a blowing, holosystolic
Mitral regurgitation
murmur.



A condition where a patient would most likely present with a rumbling, decrescendo
Mitral stenosis
diastolic murmur heard at the apex of the heart.

A condition where a patient would most likely present with a mid-
Aortic stenosis
systolic crescendo-decrescendo murmur.

Rheumatic fever A common cause for mitral stenosis.

A patient with normal lungs should be able to exhale 80% of the forced vital
Forced Vital Capacity (FVC)
capacity within the first second.

FEV1/FVC ratio A severely reduced ratio indicates chronic asthma.

Restrictive lung disease Reflected by normal FEV1, FVC, and FEV1/FVC ratio with reduced total lung capacity.

Simple spirometry Can measure all of the following EXCEPT residual volume.

Chronic bronchitis Anticipated spirometry findings include decreased forced expiratory flow (FEV1).

Restrictive lung disease indicators FEV1, FVC, and total lung capacity reduced; FEV1/FVC ratio normal.


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