TEST BANK: DAVIS ADVANTAG
n n n
E FOR PATHOPHYSIOLOGY 2ND
n n n n
EDITION BY CAPRIOTTI
n n
2020 UPDATE
n
GWU P
n
ATHOPHYSIOLOGY
, TEST BANK FOR DAVIS ADVANTA
n n n nn
GE FOR PATHOPHYSIOLOGY 2ND
n nn n n
EDITION BY CAPRIOTTI n n
Chapter n1,nThenCellninnHealthnandnIllness
MultiplenChoice
Identifynthen choicenthatn bestn completesn then statementn ornanswersn then question.
n 1.n Whichn statement n regardingn then sodium–potassiumn pumpn isn correct?
1. Then cell’sn plasman membranen isn moren solublen ton sodiumn ionsn thann potassiumn ions.
2. Thenconcentrationn of n sodiumn ionsn should n ben highern insiden then celln compartment.
3. Thenconcentrationn of npotassiumn ionsn should nben highernoutsiden then celln
compartment.
4. Thenactiven transport n involvesn pumpingn out n threen sodiumn ionsn and n pumpingn inn twon
potassiumn ions.
n 2.n Innthenabsencen ofn oxygen,nwhichn cellularn functionn createsnthensamen amount nof n energyn asn isn created n inn t
hen presencen of n oxygen?
1. Dissipationn of n pyruvicn acid
2. Initiationn of n then citricn acid n cycle
3. Activationn of n acetyl-coenzymen A
4. Creationn of n acidosisn vian lacticn acid
n 3.n How n manyn adenosinen triphosphatesn (ATPs)n aren produced n inn aerobicn energyn metabolism?
1.n n 2
2.n n 3
3.n n 34 NURSINGTB.COM
4.n n 53
n
4. Whichn celln organellesn differn inn theirn numbern accordingn ton then cell’sn energyn needs?
1. Ribosomes
2. Mitochondria
3. Ribonucleicnacids
4. Deoxyribonucleicn acids
n
5. Whichn optionn best n supportsn then reasonn moren energyn isn produced n whenn an personn isn exercising?
1. Exercisen causesn ann increasen inn then synthesisn of n protein.
2. Therenisn ann increasen inn then productionn of n pyruvicn acid n inn then cells.
3. Thenconversionn of n pyruvicn acid n ton lacticn acid n isn increased n byn exercise.
n
4. Musclencellsn haven moren mitochondrian ton meet n energyn demands.
6. Whenn doesn ribosomaln proteinn synthesisn cease?
1. Duringn endoplasmicn reticulumn stress
, 2. Duringn then synthesisn of n adenosinen triphosphaten (ATP)
3. Duringn an severen hypoxicn state
4. Duringn then processingn of n prohormone
n
7. Whichn cellularn organellesn aren responsiblen forn propellingn mucusn and n inhaled n debrisn out n of n then lungs?
1. Cilia
2. Microfilaments
3. Secretorynvesicles
, DAVIS TESTnBANK
4.n Endoplasmicn reticula
n 8.n Whichn aren then keyn proteinsn inn then contractilen unitsn of n then musclen cells?
1. Actinn and n myosin
2. Prohormonenand n tubulin
3. Tubulinn and n actin
4. Myosinn and n prohormone
n 9.n Whichn deficiencyn causesn Tay–Sachsn disease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomaln enzymes
10.n Whichn isn an characteristicn of n adrenoleukodystrophy?
n
1. Accumulationn of n ganglioside
2. Cessationn of n ribosomaln proteinn synthesis
3. Accelerationn of n cellularnproteasomen activity
4. Accumulationn of n long-chainn fattynacidsn inn then nervousn system
11.n Whichn statement n regardingn endoplasmicn reticulumn (ER)n stressn isn correct?
n
1. Duringn ERn stress,n proteinsn arenrapidlyndegraded.
2. DuringnERn stress,n lipidsn cannot n traveln ton theirn propern intracellularn locations.
3. Duringn ERn stress,n long-chainn fattyn acidsn accumulaten inn then nervousn system.
4. Duringn ERn stress,n nondeNgU
raR
deSdIsN
ubGsT
taB
nc.eC
snO
a cMcu mu late n inn then cells.
12.n A nclient n isn diagnosed nwithntypen1ndiabetesn mellitus.n At nancellularn level,n whichn functionn isnlikelyn ton benin
n
volved?
1. Inabilitynof n ribosomesn ton producen an specificn typen of n protein
2. Incorrect n processingn of n an proteinn bynthen Golgin apparatus
3. Stagnationn of n an previouslyn dynamicn actionn inn microtubules
4. Obstructionn of n then smoothn endoplasmicn reticulum
13.n A nnewbornnpatient nexhibitsn characteristicsn of nseveren physicaln deformities.n Whichn cellularn component n i
n
sn examined n ton determinen then causen and n probabilityn of n then diseasen beingn geneticallyn transferred?
1. Transfern RNA
2. Ribosomaln RNA
3. Doublen helixn of n DNA
4. MitochondrialnDNA
14.n A nhikern experiencesn musclen painn and nacidosisn whilen ascendingn anmountainnduringn an long,nsteepnc
n
limb.n Whichn isn then reasonn forn thesen manifestations?
1. Cellularn hypoxia
2. Autolysis
3. Heterolysis
4. Cellularnedema
15.n Whichn factorn providesn DNA n then uniquen molecularn abilityn ton replicate?
n
1. Then pairingn of n nitrogenousn bases
n n n
E FOR PATHOPHYSIOLOGY 2ND
n n n n
EDITION BY CAPRIOTTI
n n
2020 UPDATE
n
GWU P
n
ATHOPHYSIOLOGY
, TEST BANK FOR DAVIS ADVANTA
n n n nn
GE FOR PATHOPHYSIOLOGY 2ND
n nn n n
EDITION BY CAPRIOTTI n n
Chapter n1,nThenCellninnHealthnandnIllness
MultiplenChoice
Identifynthen choicenthatn bestn completesn then statementn ornanswersn then question.
n 1.n Whichn statement n regardingn then sodium–potassiumn pumpn isn correct?
1. Then cell’sn plasman membranen isn moren solublen ton sodiumn ionsn thann potassiumn ions.
2. Thenconcentrationn of n sodiumn ionsn should n ben highern insiden then celln compartment.
3. Thenconcentrationn of npotassiumn ionsn should nben highernoutsiden then celln
compartment.
4. Thenactiven transport n involvesn pumpingn out n threen sodiumn ionsn and n pumpingn inn twon
potassiumn ions.
n 2.n Innthenabsencen ofn oxygen,nwhichn cellularn functionn createsnthensamen amount nof n energyn asn isn created n inn t
hen presencen of n oxygen?
1. Dissipationn of n pyruvicn acid
2. Initiationn of n then citricn acid n cycle
3. Activationn of n acetyl-coenzymen A
4. Creationn of n acidosisn vian lacticn acid
n 3.n How n manyn adenosinen triphosphatesn (ATPs)n aren produced n inn aerobicn energyn metabolism?
1.n n 2
2.n n 3
3.n n 34 NURSINGTB.COM
4.n n 53
n
4. Whichn celln organellesn differn inn theirn numbern accordingn ton then cell’sn energyn needs?
1. Ribosomes
2. Mitochondria
3. Ribonucleicnacids
4. Deoxyribonucleicn acids
n
5. Whichn optionn best n supportsn then reasonn moren energyn isn produced n whenn an personn isn exercising?
1. Exercisen causesn ann increasen inn then synthesisn of n protein.
2. Therenisn ann increasen inn then productionn of n pyruvicn acid n inn then cells.
3. Thenconversionn of n pyruvicn acid n ton lacticn acid n isn increased n byn exercise.
n
4. Musclencellsn haven moren mitochondrian ton meet n energyn demands.
6. Whenn doesn ribosomaln proteinn synthesisn cease?
1. Duringn endoplasmicn reticulumn stress
, 2. Duringn then synthesisn of n adenosinen triphosphaten (ATP)
3. Duringn an severen hypoxicn state
4. Duringn then processingn of n prohormone
n
7. Whichn cellularn organellesn aren responsiblen forn propellingn mucusn and n inhaled n debrisn out n of n then lungs?
1. Cilia
2. Microfilaments
3. Secretorynvesicles
, DAVIS TESTnBANK
4.n Endoplasmicn reticula
n 8.n Whichn aren then keyn proteinsn inn then contractilen unitsn of n then musclen cells?
1. Actinn and n myosin
2. Prohormonenand n tubulin
3. Tubulinn and n actin
4. Myosinn and n prohormone
n 9.n Whichn deficiencyn causesn Tay–Sachsn disease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomaln enzymes
10.n Whichn isn an characteristicn of n adrenoleukodystrophy?
n
1. Accumulationn of n ganglioside
2. Cessationn of n ribosomaln proteinn synthesis
3. Accelerationn of n cellularnproteasomen activity
4. Accumulationn of n long-chainn fattynacidsn inn then nervousn system
11.n Whichn statement n regardingn endoplasmicn reticulumn (ER)n stressn isn correct?
n
1. Duringn ERn stress,n proteinsn arenrapidlyndegraded.
2. DuringnERn stress,n lipidsn cannot n traveln ton theirn propern intracellularn locations.
3. Duringn ERn stress,n long-chainn fattyn acidsn accumulaten inn then nervousn system.
4. Duringn ERn stress,n nondeNgU
raR
deSdIsN
ubGsT
taB
nc.eC
snO
a cMcu mu late n inn then cells.
12.n A nclient n isn diagnosed nwithntypen1ndiabetesn mellitus.n At nancellularn level,n whichn functionn isnlikelyn ton benin
n
volved?
1. Inabilitynof n ribosomesn ton producen an specificn typen of n protein
2. Incorrect n processingn of n an proteinn bynthen Golgin apparatus
3. Stagnationn of n an previouslyn dynamicn actionn inn microtubules
4. Obstructionn of n then smoothn endoplasmicn reticulum
13.n A nnewbornnpatient nexhibitsn characteristicsn of nseveren physicaln deformities.n Whichn cellularn component n i
n
sn examined n ton determinen then causen and n probabilityn of n then diseasen beingn geneticallyn transferred?
1. Transfern RNA
2. Ribosomaln RNA
3. Doublen helixn of n DNA
4. MitochondrialnDNA
14.n A nhikern experiencesn musclen painn and nacidosisn whilen ascendingn anmountainnduringn an long,nsteepnc
n
limb.n Whichn isn then reasonn forn thesen manifestations?
1. Cellularn hypoxia
2. Autolysis
3. Heterolysis
4. Cellularnedema
15.n Whichn factorn providesn DNA n then uniquen molecularn abilityn ton replicate?
n
1. Then pairingn of n nitrogenousn bases
