PATH 370 - Final Exam Questions & Answers
p p p p p p p
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Afterpsurgeryptopremovepaplungptumor,pyourppatientpispscheduledpforpchemotherapy,pwhichpwillpS
electivelypkillptumorpcells
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pAtrophy
Hyperplasiap-pNecrosis
Ap17-year-oldpcollege-
boundpstudentpreceivespapvaccinepagainstpanporganismpthatpcausespmeningitis.pThispispanpe
xamplepof
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econdaryppreventionpT
ertiarypprevention
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hrinepInsulinp-
pinsulin
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p EndemicpCancerpgradingpi
spbasedpon
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calpinvasion
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orrectpresponsepispwhichpofpthepfollowing?
Itpispapprocedurepforpdeterminingpthepextentpofptumorpspread
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spbasedponpexploratorypsurgery
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pItpispapprocedurepforpdeterminingpthepextentpofptumorpspread
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stablished,pispreferredptopas
LatentpSub
clinicalpPro
dromal
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Afterpbronchoscopypandphistologicpexaminationpofpapsuspectedptumor,pyourppatientpispdiagnosedp
withpprimarypbronchialpcarcinoma.pThus,ptheptumor
Ispbenign
Ispmalignant
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Tumorpmarkersp(Selectpallpthatpapply.)
a.parepfoundponlypinpthepblood.pb.
parepproducedpbypnormalpcells.p c.p
helppdeterminepcancerporigin.
d.phelppidentifypprogressionpofpcancer.
e.pincludepprostatic-specificpantigen.p-pANS:pB,pC,pD,pE
Tumorpmarkersparepsubstancespassociatedpwithptumorpcellspthatpmaypbephelpfulpinpidentifyingpthe
irptissuepofporiginpandpprogressionpandpproliferationpofpcancerpcells.pMostptumorpmarkersparepnotpv
erypspecificpforpcancerpsincepnormalpcellspinptheptissuepofporiginpalsopproducepthem.pTumorpmarke
rspincludepprostatic-
specificpantigen.pSomeptumorpcellpmarkersparepreleasedpintopthepcirculation,pbutpotherspmustpbepid
entifiedpthroughpbiopsypofpmetastaticptissue.
Deficitspinpimmunepsystempfunctionpoccurpinpcancerpaspapresultpofp(Selectpallpthatpapply.)pa.
pchemotherapy.
b. cancerpcells.
c. cancerpmetastasisptopbonepmarrow.
d. immunotherapy.
e. malnutrition.p-pANS:pA,pB,pC,pE
, Cancerpcellspsecretepsubstancespthatpsuppresspthepimmunepsystem.pBonepmarrowpsuppressionpmaypb
epbecausepofpinvasionpofpblood-
formingpcellspinpthepbonepmarrow,ppoorpnutrition,pandpchemotherapeuticpagents.pBypcontrast,pimmu
notherapypispapmethodpofpharnessingpthepimmunepsystempinptheptreatmentpofpcancer.
Thepcellularpchangepthatpispconsideredppreneoplasticpisp
Anaplasia
Dysplasiap
Metaplasia
Hyperplasiap-
p dysplasiapApoptosispispthep
processpof
AtrophypDe
athpProlifera
tion
Mutationp-pDeath
Afterpsufferingpapheartpattack,papmiddlepagedpmanpispcounseledptoptakepapcholesterolploweringp
medication.pThispispanpexamplepof
Primaryppreventionp
Secondary
Tertiary
Diseaseptreatmentp-pTertiarypprevention
Manypofpthepresponsesptopstressparepattributedptopactivationpofpthepsympatheticpnervouspsystempa
ndparepmediatedpby
Norepinephrine
pCortisolpGluca
gon
ACTHp-pnorepinephrine
Selye'spthreepphasespofpthepstresspresponsepincludepallpofpthepfollowingpexceptp
Allostasis
Resistance
pAlarm
Exhaustionp-pAllostasis
Sidepeffectpofpchemotherapypincludep(selectpallpthatpapply)p
Anemia
NauseapLeuk
ocytosispBlee
ding
p p p p p p p
Thepcellularpresponsepindicativepofpinjurypdueptopfaultypmetabolismpisp
Hydropicpswelling
Lactatepproductionp
Metaplasia
Intracellularpaccumulationsp-pintracellularpaccumulation
Afterpsurgeryptopremovepaplungptumor,pyourppatientpispscheduledpforpchemotherapy,pwhichpwillpS
electivelypkillptumorpcells
Stimulatepimmunepcellsptopfightpthepcancerp
Havepminimalpsidepeffects
Killprapidlypdividingpcellsp-pkillprapidlypdividingpcells
Allpthesepcellularpresponsespareppotentiallypreversiblepexceptp
Necrosis
Metaplasia
pAtrophy
Hyperplasiap-pNecrosis
Ap17-year-oldpcollege-
boundpstudentpreceivespapvaccinepagainstpanporganismpthatpcausespmeningitis.pThispispanpe
xamplepof
PrimaryppreventionpS
econdaryppreventionpT
ertiarypprevention
Diseaseptreatmentp-pprimarypprevention
Whichpispnotpnormallypsecretedpinpresponseptopstress?p
Norepinephrine
CortisolpEpinep
hrinepInsulinp-
pinsulin
Apdiseasepthatpispnativeptopapparticularpregionpispcalledp
Epidemic
Endemicp
Pandemic
Ethnographicp-
p EndemicpCancerpgradingpi
spbasedpon
,TumorpsizepLo
calpinvasion
Cellpdifferentiation
Metastasisp-pcellpdifferentiation
Yourppatientpispscheduledpforpapstagingpprocedure.pShepwantsptopknowpwhatpthatpmeans.pThepc
orrectpresponsepispwhichpofpthepfollowing?
Itpispapprocedurepforpdeterminingpthepextentpofptumorpspread
ItpispaphistologicpexaminationpofptissuesptopdeterminepthepdegreepofptumorpdifferentiationpI
spbasedponpexploratorypsurgery
Itpispbiochemicalptestingpofptumorpcellsptopdeterminepthepgeneticpbasispofptheptumorp-
pItpispapprocedurepforpdeterminingpthepextentpofptumorpspread
Thepstagepduringpwhichptheppatientpfunctionspnormally,palthoughpthepdiseasepprocessesparepwellpe
stablished,pispreferredptopas
LatentpSub
clinicalpPro
dromal
Convalescencep-psubclinical
Afterpbronchoscopypandphistologicpexaminationpofpapsuspectedptumor,pyourppatientpispdiagnosedp
withpprimarypbronchialpcarcinoma.pThus,ptheptumor
Ispbenign
Ispmalignant
Ispsecondaryptopcancerpelsewherepinpthepbodyp
Haspspreadp-pIspmalignant
Tumorpmarkersp(Selectpallpthatpapply.)
a.parepfoundponlypinpthepblood.pb.
parepproducedpbypnormalpcells.p c.p
helppdeterminepcancerporigin.
d.phelppidentifypprogressionpofpcancer.
e.pincludepprostatic-specificpantigen.p-pANS:pB,pC,pD,pE
Tumorpmarkersparepsubstancespassociatedpwithptumorpcellspthatpmaypbephelpfulpinpidentifyingpthe
irptissuepofporiginpandpprogressionpandpproliferationpofpcancerpcells.pMostptumorpmarkersparepnotpv
erypspecificpforpcancerpsincepnormalpcellspinptheptissuepofporiginpalsopproducepthem.pTumorpmarke
rspincludepprostatic-
specificpantigen.pSomeptumorpcellpmarkersparepreleasedpintopthepcirculation,pbutpotherspmustpbepid
entifiedpthroughpbiopsypofpmetastaticptissue.
Deficitspinpimmunepsystempfunctionpoccurpinpcancerpaspapresultpofp(Selectpallpthatpapply.)pa.
pchemotherapy.
b. cancerpcells.
c. cancerpmetastasisptopbonepmarrow.
d. immunotherapy.
e. malnutrition.p-pANS:pA,pB,pC,pE
, Cancerpcellspsecretepsubstancespthatpsuppresspthepimmunepsystem.pBonepmarrowpsuppressionpmaypb
epbecausepofpinvasionpofpblood-
formingpcellspinpthepbonepmarrow,ppoorpnutrition,pandpchemotherapeuticpagents.pBypcontrast,pimmu
notherapypispapmethodpofpharnessingpthepimmunepsystempinptheptreatmentpofpcancer.
Thepcellularpchangepthatpispconsideredppreneoplasticpisp
Anaplasia
Dysplasiap
Metaplasia
Hyperplasiap-
p dysplasiapApoptosispispthep
processpof
AtrophypDe
athpProlifera
tion
Mutationp-pDeath
Afterpsufferingpapheartpattack,papmiddlepagedpmanpispcounseledptoptakepapcholesterolploweringp
medication.pThispispanpexamplepof
Primaryppreventionp
Secondary
Tertiary
Diseaseptreatmentp-pTertiarypprevention
Manypofpthepresponsesptopstressparepattributedptopactivationpofpthepsympatheticpnervouspsystempa
ndparepmediatedpby
Norepinephrine
pCortisolpGluca
gon
ACTHp-pnorepinephrine
Selye'spthreepphasespofpthepstresspresponsepincludepallpofpthepfollowingpexceptp
Allostasis
Resistance
pAlarm
Exhaustionp-pAllostasis
Sidepeffectpofpchemotherapypincludep(selectpallpthatpapply)p
Anemia
NauseapLeuk
ocytosispBlee
ding
