TEST BANK For Davis Advantage for Pathophysiology ... 2nd Edition by Theresa M Capriotti All 1-46 Chapters Covered ,Latest Edition.

TEST BANK FOR DAVIS ADVANTAGE FO
M M M M M




R PATHOPHYSIOLOGY 2ND EDITIONBY
M M M M M




CAPRIOTTI

, Chapter 1, The Cell in Health and Illness
M M M M M M M


MultipleMChoice
IdentifyMtheMchoiceMthatMbestMcompletesMtheMstatementMorManswersMtheMquestion.

M MMM 1.MWhichMstatementMregardingMtheMsodium–potassiumMpumpMisMcorrect?
1. TheMcell’sMplasmaMmembraneMisMmoreMsolubleMtoMsodiumMionsMthanMpotassiumMions.
2. TheMconcentrationMofMsodiumMionsMshouldMbeMhigherMinsideMtheMcellMcompartment.
3. TheMconcentrationMofMpotassiumMionsMshouldMbeMhigherMoutsideMtheM
cellMcompartment.
4. TheMactiveMtransportMinvolvesMpumpingMoutMthreeMsodiumMionsMandMpumpingMinM
twoMpotassiumMions.
M

MMM2.MInMtheMabsenceMofMoxygen,MwhichMcellularMfunctionMcreatesMtheMsameMamountMofMenergyMa

sMisMcreatedMinMtheMpresenceMofMoxygen?
1. DissipationMofMpyruvicMacid
2. InitiationMofMtheMcitricMacidMcycle
3. ActivationMofMacetyl-coenzymeMA
4. CreationMofMacidosisMviaMlacticMacid

M MMM 3.MHowMmanyMadenosineMtriphosphatesM(ATPs)MareMproducedMinMaerobicMenergyMmetabolism?
1.MMM 2
2.MMM 3
3.MMM 34
4.MMM 53

M 4.M WhichMcellMorganellesMdifferMinMtheirMnumberMaccordingMtoMtheMcell’sMenergyMneeds?
MMM

1. Ribosomes
2. Mitochondria
3. RibonucleicMacids
4. DeoxyribonucleicMacids

M 5.M WhichMoptionMbestMsupportsMtheMreasonMmoreMenergyMisMproducedMwhenMaMpersonMisMexercising?
MMM

1. ExerciseMcausesManMincreaseMinMtheMsynthesisMofMprotein.
2. ThereMisManMincreaseMinMtheMproductionMofMpyruvicMacidMinMtheMcells.
3. TheMconversionMofMpyruvicMacidMtoMlacticMacidMisMincreasedMbyMexercise.
4. MuscleMcellsMhaveMmoreMmitochondriaMtoMmeetMenergyMdemands.

M 6.M WhenMdoesMribosomalMproteinMsynthesisMcease?
MMM

1. DuringMendoplasmicMreticulumMstress
2. DuringMtheMsynthesisMofMadenosineMtriphosphateM(ATP)
3. DuringMaMsevereMhypoxicMstate
4. DuringMtheMprocessingMofMprohormone
M

7.M WhichMcellularMorganellesMareMresponsibleMforMpropellingMmucusMandMinhaledMdebrisMoutMofMtheMlungs
MMM

?
1. Cilia
2. Microfilaments

,3. SecretoryMvesicles

, 4. EndoplasmicMreticula

M MMM 8.MWhichMareMtheMkeyMproteinsMinMtheMcontractileMunitsMofMtheMmuscleMcells?
1. ActinMandMmyosin
2. ProhormoneMandMtubulin
3. TubulinMandMactin
4. MyosinMandMprohormone

M MMM 9.M WhichMdeficiencyMcausesMTay–SachsMdisease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. LysosomalMenzymes

M 10.M WhichMisMaMcharacteristicMofMadrenoleukodystrophy?
1. AccumulationMofMganglioside
2. CessationMofMribosomalMproteinMsynthesis
3. AccelerationMofMcellularMproteasomeMactivity
4. AccumulationMofMlong-chainMfattyMacidsMinMtheMnervousMsystem

M 11.M WhichMstatementMregardingMendoplasmicMreticulumM(ER)MstressMisMcorrect?
1. DuringMERMstress,MproteinsMareMrapidlyMdegraded.
2. DuringMERMstress,MlipidsMcannotMtravelMtoMtheirMproperMintracellularMlocations.
3. DuringMERMstress,Mlong-chainMfattyMacidsMaccumulateMinMtheMnervousMsystem.
4. DuringMERMstress,M nondeNgU
raR
deSdIMMsT
taB
nc.eC
sMO
acMM cumulateMinMtheMcesll.
M

12.MAMclientMisMdiagnosedMwithMtypeM1MdiabetesMmellitus.MAtMaMcellularMlevel,MwhichMfunctionMi
sMlikelyMtoMbeMinvolved?
1. InabilityMofMribosomesMtoMproduceMaMspecificMtypeMofMprotein
2. IncorrectMprocessingMofMaMproteinMbyMtheMGolgiMapparatus
3. StagnationMofMaMpreviouslyMdynamicMactionMinMmicrotubules
4. ObstructionMofMtheMsmoothMendoplasmicMreticulum
M

13.MAMnewbornMpatientMexhibitsMcharacteristicsMofMsevereMphysicalMdeformities.MWhichMcellular
McomponentMisMexaminedMtoMdetermineMtheMcauseMandMprobabilityMofMtheMdiseaseMbeingMgenetically
Mtransferred?
1. TransferMRNA
2. RibosomalMRNA
3. DoubleMhelixMofMDNA
4. MitochondrialMDNA
M

14.MAMhikerMexperiencesMmuscleMpainMandMacidosisMwhileMascendingMaMmountainMduringMa
Mlong,MsteepMclimb.MWhichMisMtheMreasonMforMtheseMmanifestations?
1. CellularMhypoxia
2. Autolysis
3. Heterolysis
4. CellularMedema

M 15.M WhichMfactorMprovidesMDNAMtheMuniqueMmolecularMabilityMtoMreplicate?
1. TheMpairingMofMnitrogenousMbases