NURS 5315 Endocrine EXAM 2025 LATEST UPDATED | COMPREHENSIVE QUESTIONS WITH 100%
RATED ANSWERS | GUARANTEED TO PASS!!
Thyroid hormones - correct answer T3, T4 and calcitonin
Pancreas hormones - correct answer Insulin and glucagon
Adrenal Glands hormones - correct answer Made up of the cortex which secretes steroids such
as cortisone and aldosterone and the medulla which secretes catecholamines such as epi and
norepi
Primary thyroid disorders - correct answer result in alterations of thyroid hormone (TH) levels
with secondary feedback effects on pituitary thyroid stimulating hormone (TSH)
Subclinical Thyroid disease - correct answer Thyroid disease that presents with minimal to no
symptoms but with abnormal lab values
Secondary Thyroid disorders - correct answer conditions that results from the dysfunction of
the pituitary gland TSH production
Thyrotoxicosis - correct answer (hyperthyroidism) a condition that results from any cause of
increased TH levels. Will how low TSH levels and high T4 level. S&S: increased metabolic rate,
heat intolerance, goiter, menstrual irrregularities, weight loss, diaphoresis, fine tremor,
tachycardia, frequent bowel movements, restlessness, short attention span, hair loss, anorexia,
exophthalmos, pretibial edema, and heart failure.
Thyrotoxic Crisis(thyroid storm) - correct answer worsening hyperthyroid state triggered by an
igniting even such as infection, trauma, cardiopulmonary disorder, burns, seizures surgery, or
spontaneously. S&S: extreme restlessness and agitation, delirium, seizures, coma, severe
tachycardia, heart failure, hyperthermia, delirium, volume depletion, NVD and death if not
treated.
Grave's disease - correct answer Most common cause of hyperthyroidism and is an
autoimmune disorder. Antibodies attach to the thyroid cells and mimic the function of TSH
which results in an increased secretion of T3 and T4 and overrides the negative feedback
mechanisms which regulate TSH secretion. The stimulation of the receptors by the antibodies
results in the development of goiter. May also experience exophthalmos, periorbital edema,
and extraocular muscle weakness leading to strabismus and diplopia
Hyperthyroidism from nodular thyroid disease - correct answer Follicular hypertrophy of the
thyroid cells is responsible for the formation of the thyroid nodules which secrete extra
hormones. Nodules develop bc of normal changes during pregnancy or puberty or as a result of
an autoimmune issue, viral infection or genetic influence. Symptoms develop slowly and will
not display exophthalmos or pretibial myxedema
Primary hypothyroidism - correct answer Defect is in the thyroid gland itself which causes
insufficient amounts of thyroid hormone. Causes include congenital defects, thyroidectomy,
thyroid radiation, iodine deficiency, anti-thyroid medications, or impairment in thyroid
hormone synthesis
, NURS 5315 Endocrine EXAM 2025 LATEST UPDATED | COMPREHENSIVE QUESTIONS WITH 100%
RATED ANSWERS | GUARANTEED TO PASS!!
Secondary hypothyroidism - correct answer Malfunction in the pituitary or hypothalamus
glands which leads to a lack of TSH. Most common cause is pituitary tumors. Other causes
include TBI, subarachnoid hemorrhage, or pituitary infarction
Subclinical hypothyroidism - correct answer mild thyroid failure. defined by elevated TSH level
with normal TH level.
Hypothyroid S&S - correct answer confusion, syncope, slow speech and thinking, anemia,
bradycardia, reduced stroke volume and cardiac output, dyspnea, hypoventilation, decreased
appetite, weight gain, dry hair, cold intolerant, constipation, hyperlipidemia, periorbital edema,
peripheral edema, myxedema(puffy face), increased total body water, hyponatremia, reduced
renal blood flow
Myxedema coma - correct answer thyroid emergency which has the opposite effect of thyroid
storm. Results in decreased LOC and is usually precipitated by an event such as infection,
discontinuation of thyroid medications, narcotic or sedative use. other S&S hypotension,
hypoventilation, shivering, hypothermia, lactic acidosis, coma, and hypoglycemia.
Type 1 DM - correct answer 3 types: 1A is autoimmune, 1B is idiopathic and 3c is associated
with chronic pancreatitis. in 1A: autoimmune response destroys the beta cells in the pancreas
which leads to apoptosis. Beta cell destruction is what causes a lack of insulin to be produced.
Type 1 clinical manifestations - correct answer polydipsia, polyuria, polyphagia, weight loss and
fatigue.
Typer 2 DM - correct answer pathlogical defect is insulin resistance. A decrease in number of
insulin cell receptors or insufficient amounts of insulin secretion to meet metabolic needs are
characteristic of DM 2.
Type 2 DM clinical manifestations - correct answer recurrent infections, genital pruritus, visual
changes, paresthesias, fatigue, and acanthosis nigricans(brown to black pigmentation in body
folds)
Gestational DM - correct answer develops when hyperglycemia appears during pregnancy and
usually resolves after birth. All women should be screened during their first prenatal visit and
again between 24-28 weeks. Risk factors include obesity, family history, and high maternal age.
upon birth the child may have hyperplasia of the pancreatic islet cells and hypoglycemia. 3w2
Hemoglobin A1c - correct answer A test that measures the level of hemoglobin A1c in the blood
as a means of determining the average blood sugar concentrations for the preceding two to
three months. DM diagnostic is greater than or equal to 6.5. Increased risk for DM level 5.7-
6.4%
Fasting plasma glucose - correct answer DM diagnostic criteria is greater or equal to 126. Risk
for DM level at 100-125
RATED ANSWERS | GUARANTEED TO PASS!!
Thyroid hormones - correct answer T3, T4 and calcitonin
Pancreas hormones - correct answer Insulin and glucagon
Adrenal Glands hormones - correct answer Made up of the cortex which secretes steroids such
as cortisone and aldosterone and the medulla which secretes catecholamines such as epi and
norepi
Primary thyroid disorders - correct answer result in alterations of thyroid hormone (TH) levels
with secondary feedback effects on pituitary thyroid stimulating hormone (TSH)
Subclinical Thyroid disease - correct answer Thyroid disease that presents with minimal to no
symptoms but with abnormal lab values
Secondary Thyroid disorders - correct answer conditions that results from the dysfunction of
the pituitary gland TSH production
Thyrotoxicosis - correct answer (hyperthyroidism) a condition that results from any cause of
increased TH levels. Will how low TSH levels and high T4 level. S&S: increased metabolic rate,
heat intolerance, goiter, menstrual irrregularities, weight loss, diaphoresis, fine tremor,
tachycardia, frequent bowel movements, restlessness, short attention span, hair loss, anorexia,
exophthalmos, pretibial edema, and heart failure.
Thyrotoxic Crisis(thyroid storm) - correct answer worsening hyperthyroid state triggered by an
igniting even such as infection, trauma, cardiopulmonary disorder, burns, seizures surgery, or
spontaneously. S&S: extreme restlessness and agitation, delirium, seizures, coma, severe
tachycardia, heart failure, hyperthermia, delirium, volume depletion, NVD and death if not
treated.
Grave's disease - correct answer Most common cause of hyperthyroidism and is an
autoimmune disorder. Antibodies attach to the thyroid cells and mimic the function of TSH
which results in an increased secretion of T3 and T4 and overrides the negative feedback
mechanisms which regulate TSH secretion. The stimulation of the receptors by the antibodies
results in the development of goiter. May also experience exophthalmos, periorbital edema,
and extraocular muscle weakness leading to strabismus and diplopia
Hyperthyroidism from nodular thyroid disease - correct answer Follicular hypertrophy of the
thyroid cells is responsible for the formation of the thyroid nodules which secrete extra
hormones. Nodules develop bc of normal changes during pregnancy or puberty or as a result of
an autoimmune issue, viral infection or genetic influence. Symptoms develop slowly and will
not display exophthalmos or pretibial myxedema
Primary hypothyroidism - correct answer Defect is in the thyroid gland itself which causes
insufficient amounts of thyroid hormone. Causes include congenital defects, thyroidectomy,
thyroid radiation, iodine deficiency, anti-thyroid medications, or impairment in thyroid
hormone synthesis
, NURS 5315 Endocrine EXAM 2025 LATEST UPDATED | COMPREHENSIVE QUESTIONS WITH 100%
RATED ANSWERS | GUARANTEED TO PASS!!
Secondary hypothyroidism - correct answer Malfunction in the pituitary or hypothalamus
glands which leads to a lack of TSH. Most common cause is pituitary tumors. Other causes
include TBI, subarachnoid hemorrhage, or pituitary infarction
Subclinical hypothyroidism - correct answer mild thyroid failure. defined by elevated TSH level
with normal TH level.
Hypothyroid S&S - correct answer confusion, syncope, slow speech and thinking, anemia,
bradycardia, reduced stroke volume and cardiac output, dyspnea, hypoventilation, decreased
appetite, weight gain, dry hair, cold intolerant, constipation, hyperlipidemia, periorbital edema,
peripheral edema, myxedema(puffy face), increased total body water, hyponatremia, reduced
renal blood flow
Myxedema coma - correct answer thyroid emergency which has the opposite effect of thyroid
storm. Results in decreased LOC and is usually precipitated by an event such as infection,
discontinuation of thyroid medications, narcotic or sedative use. other S&S hypotension,
hypoventilation, shivering, hypothermia, lactic acidosis, coma, and hypoglycemia.
Type 1 DM - correct answer 3 types: 1A is autoimmune, 1B is idiopathic and 3c is associated
with chronic pancreatitis. in 1A: autoimmune response destroys the beta cells in the pancreas
which leads to apoptosis. Beta cell destruction is what causes a lack of insulin to be produced.
Type 1 clinical manifestations - correct answer polydipsia, polyuria, polyphagia, weight loss and
fatigue.
Typer 2 DM - correct answer pathlogical defect is insulin resistance. A decrease in number of
insulin cell receptors or insufficient amounts of insulin secretion to meet metabolic needs are
characteristic of DM 2.
Type 2 DM clinical manifestations - correct answer recurrent infections, genital pruritus, visual
changes, paresthesias, fatigue, and acanthosis nigricans(brown to black pigmentation in body
folds)
Gestational DM - correct answer develops when hyperglycemia appears during pregnancy and
usually resolves after birth. All women should be screened during their first prenatal visit and
again between 24-28 weeks. Risk factors include obesity, family history, and high maternal age.
upon birth the child may have hyperplasia of the pancreatic islet cells and hypoglycemia. 3w2
Hemoglobin A1c - correct answer A test that measures the level of hemoglobin A1c in the blood
as a means of determining the average blood sugar concentrations for the preceding two to
three months. DM diagnostic is greater than or equal to 6.5. Increased risk for DM level 5.7-
6.4%
Fasting plasma glucose - correct answer DM diagnostic criteria is greater or equal to 126. Risk
for DM level at 100-125
