NURS 8022 EXAM 3 NEWEST 2024 ACTUAL EXAM COMPLETE EXAM QUESTIONS AND CORRECT ANSWERS (VERIFIED ANSWERS) ALREADY GRADED A+BRAND NEW!!.pdf

NURS 8022 EXAM 3 | NEWEST ACTUAL
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EXAM COMPREHENSIVE QUESTIONS A
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ND VERIFIED ANSWERS GRADED A+ | 1
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00% PASS | 2025 UPDATE! m m m m




A person has pneumoconiosis. Which information would the nurse find
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in the history of this
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person?


A. Inhaled inorganic dust particles, resulting in a change in the lungs
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B. Fractured ribs, causing paradoxical movement of the chest with brea
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thing
C. Ruptured visceral pleura, which allows air or gas into the pleural spa
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ce
D. Bronchial inflammation with a persistent abnormal dilation of the br
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onchi
A. Inhaled inorganic dust particles, resulting in a change in the lungs
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A person has pneumococcal pneumonia. Which pathophysiologic proce
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ss has occurred?
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,1. Continual bronchial inflammation causes bronchial edema an
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d increases the size and number of mucous glands and goblet cel
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ls.
2. Abnormal permanent enlargement of the acini is accompanied by th
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e destruction of alveolar walls.
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3. Inflammatory cytokines cause alveolar edema, which creates
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a medium for microorganisms that leads to consolidation
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3. Inflammatory cytokines causes alveolar edema, which creates a me
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dium for microorganisms that leads to consolidation
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Where does second degree Mobitz I heart block occur?
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Level of the AV node
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Where does second degree Mobitz II heart block occur?
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In the bundle of His or lower in the AV conduction system
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A person has a pulmonary embolism. What will the NP find upon assess
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ment?
Sudden pleuritic chest pain, dyspnea, unexplained anxiety
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,An NP recalls that cystic fibrosis is associated with
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1. Defective epithelial chloride ion transport
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2. Increased mucociliary action
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3. Propensity for upper respiratory infections
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1. Defective epithelial chloride ion transport
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Cystic fibrosis causes persistent chronic bronchial inflammation, which
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also causes
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1. Idiopathic pulmonary fibrosis
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2. Pneumoconiosis
3. Bronchiectasis
4. Surfactant impairment m




3. Bronchiectasis
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POPE?
Postobstructive pulmonary edema. Negative pressure pulmonary ede
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ma; rare life threatening complication after relief of upper airway obs
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truction




Acute lung injury (ALI)/Acute respiratory distress syndrome (ARDS)
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, results from an acute, diffuse injury to the alveolocapillary membran
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e and decreased surfactant production, which increases membrane p
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ermeability and causes edema, atelectasis, and hypoxemia.
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ARDS process
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- rapid onset
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- fluid out of blood vessels into alveoli
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- damage impairs gas exchange
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- lung stiffens
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- increased work of breath m m m




- alveolar collapse m




A child has asthma. Which pathophysiologic process occurs in this disea
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se?
1. Acute injury to the alveoli capillary membrane, producing sever
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e pulmonary edema and shunting
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2. Chronic inflammatory disorder, causing mucosal edema an
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d reversible airflow obstruction
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3. Airway obstruction, increasing resistance to airflow and decreasin
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g flow rates, especially inspiratory flow
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2. Chronic inflammatory disorder, causing mucosal edema an
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d reversible airflow obstruction
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