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NURS 5335 VERIFIED MODULE 2 RENAL EXAM QUESTIONS AND ANSWERS LATEST UPDATE

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NURS 5335 VERIFIED MODULE 2 RENAL EXAM QUESTIONS AND ANSWERS LATEST UPDATE

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NURS 5335 VERIFIED MODULE 2 RENAL EXAM QUESTIONS AND
ANSWERS LATEST UPDATE

Enuresis pharm treatment
-Desmopressin for children >6: 0.2-0.6mg at bedtime; restrict fluids 1 hour before admin
and 8 hours after
-Anticholinergics for overactive neurogenic bladder or dysfunctional voiding
Hematuria description
The presence of blood in uncontaminated urine specimen, detected by dipstick,
microsocpic examination, or the naked eye
Hematuria innocent etiology
-Exercise induced
-Mensuration
-Sexual intercourse
-Pelvic exam
-Prostate exam
-Catheterization
-Biopsy of the GU tract
Hematuria adult etiology
-Malignant neoplasms in the GU tract
-Infection
-Renal calculi
-Coagulopathy
-Thrombocytopenia
-Glomerular disease
-Hydronephrosis
-Polycystic kidney disease
-Anticoagulants
-Antiplatelets
-Trauma/recent urologic procedure
-BPH

,Hematuria child etiolog
-UTI
-Perineal or urethral irritation
-Sickle cell disease
-Trauma
-Congenital anomoly
Pseudohematuria
-Myoglobin or hemoglobin can cause positive reactions on tests for hematuria
-If dip is (+) and micro is (-) for RBC, think myoglobin or hemoglobin in urine
-Both are serious and need further eval
Pseudohematuria substance that make urine appear red
-Phenacetin
-Pyridium
-Nitrofurantoin
-Sulfonamides
-Cascara
-Senna
-Doxorubicin
-Beets
-Blackberries
-Rhubarb
-Paprika
-Mercurochrome
-Betadine
Hematuria diagnostics
-UA: RBCs and RBC casts
-Urine C&C
-BUN
-Creatinine
-CMP
-GFR

, -PT, PTT, INR
-Sed rate
-IVP
-Renal US to identify cysts vs solid mass
-Cystoscopy
-Renal biopsy
-CT scan
-MRI
-Sickle cell assessment
Hematuria treatment
Refer to urologist
Bladder Cancer risk factors
-Age >50
-Smoking
-Occupational exposure to aromatic amines, dyes, benzidine, and pain
-Pelvic irradiation
-Cyclophosphamide use
-Schistmoaniasis history from eating snails
-Prolonged use of Phenacetin
-
Poststreptococcal glomerulonephritis description
Immune response to an infection that damages the glomeruli, resulting in diffuse
inflammatory changes in the glomeruli; PSGN produces clinical symtpoms of peripheral
edema, acute renal failure, and gross hematuria
Poststreptococcal glomerulonephritis etiology
-Follows infection of the throat or skin with GAS
-Develops 1-3 weeks after strep infection
Poststreptococcal glomerulonephritis assessment findings
-Pharyngeal strep within the past 2-3 weeks
-Skin strep within the past 2-6 weeks
-Hematuria with abrupt onset

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