TESTBANK n
NEONATAL & PEDIATRIC RESPIR
n n n
ATORY CARE n
5th Edition, Walsh
n
n
TESTBANK n
,NeonatalnandnPediatricnRespiratorynCare,n5thnEdition,nBriannK.nWalshnTestnBank
Tablenof nContents
Chaptern 1.n Fetaln Lungn Development
Chaptern 2.n Fetaln Gasn Exchangen and n Circulation
Chaptern 3.n Antenataln Assessment n and n High-Riskn Delivery
Chaptern 4.n Examinationn and n Assessment n of n then Neonataln and n Pediatricn Patient
Chaptern 5.n PulmonarynFunctionn Testingn and n Bedsiden Pulmonaryn Mechanics
Chaptern 6.n Radiographicn Assessment
Chaptern 7.n Pediatricn Flexiblen Bronchoscopy
Chaptern 8.n Invasiven Blood n Gasn Analysisn and n Cardiovascularn Monitoring
Chaptern 9.n Noninvasiven Monitoringn inn Neonataln and n Pediatricn Care
Chaptern 10.n Oxygenn Administration
Chaptern 11.n Aerosolsn and n Administrationn of n Inhaled n Medications
Chaptern 12.n AirwaynClearancen Techniquesn and n Hyperinflationn Therapy
Chaptern 13.n AirwaynManagement
Chaptern 14.n Surfactant n Replacement n Therapy
Chaptern 15.n Noninvasiven Mechanicaln Ventilationn and n Continuousn Positiven Pressuren of n then Neonate
Chaptern 16.n Noninvasiven Mechanicaln Ventilationn of n then Infant n and n Child
Chaptern 17.n Invasiven Mechanicaln Ventilationn of n then Neonaten and n Pediatricn Patient
Chaptern 18.n Administrationn of n Gasn Mixtures
Chaptern 19.n Extracorporealn Membranen Oxygenation
Chaptern 20.n Pharmacology
Chaptern 21.n Thoracicn Organn Transplantation
Chaptern 22.n Neonataln Pulmonaryn Disorders
Chaptern 23.n Surgicaln Disordersn inn Childhood n that n Affect n RespiratorynCare
Chaptern 24.n Congenitaln Cardiacn Defects
Chaptern 25.n Pediatricn Sleep-Disordered n Breathing
Chaptern 26.n Pediatricn Airwayn Disordersn and n Parenchymaln Lungn Diseases
Chaptern 27.n Asthma
Chaptern 28.n Cysticn Fibrosis
Chaptern 29.n Acuten Respiratoryn Distressn Syndrome
Chaptern 30.n Shock
Chaptern 31.n Pediatricn Trauma
Chaptern 32.n Disordersn of n then Pleura
Chaptern 33.n Neurologicaln and n Neuromuscularn Disorders
Chaptern 34.n Pediatricn Emergencies
Chaptern 35.n Homen Caren of n then Postpartumn Family
Chaptern 36.n Qualityn and n Safety
,Chaptern1:nFetalnLungnDevelopment
Walsh:nNeonataln&nPediatricnRespiratorynCaren5thnEditionnTestnBankn(2020)
MULTIPLEnCHOICE
1. Whichn of nthen followingnphasesn ofnhumann lungndevelopment n isn characterized nbynthenformationno
f n an capillaryn networkn around n airwayn passages?
a. Pseudoglandular
b. Saccular
c. Alveolar
d. Canalicular
ANS:n D
Then canalicularn phasen followsn then pseudoglandularn phase,n lastingn fromn approximatelyn 17n week
snton26nweeksn of n gestation.nThisn phasenisnson named nbecausenof nthen appearancen of nvascularn chann
els,n orn capillaries,n whichn beginn ton grow n byn formingn an capillaryn networkn around n then airn passages
.n Duringn then pseudoglandularn stage,nwhichn beginsn atn dayn52n andnextendsntonweekn16n of n gestatio
n,n then airwayn systemn subdividesn extensivelyn and n then conductingn airwayn systemn develops,n endi
ngn withn then terminaln bronchioles.n Then saccularn stagen of n development,n whichn takesn placen fromn
weeksn 29n ton 36nofngestation,nisn characterized n bynthendevelopment n of n sacsn that n latern becomen alve
oli.n Duringn then saccularn phase,n an tremendousn increasen inn then potentialn gas-
n exchangingn surfacen arean occurs.n Then distinctionn betweenn then saccularn stagen andn then alveolarn st
agen isn arbitrary.n Then alveolarn stagen stretchesn fromn 39n weeksn of ngestationn ton term.n Thisn stagen isn r
epresented n byn then establishment n of n alveoli.
REF:n pp.n 3-5
2. Regardingnpostnataln lungn growth,nbynapproximatelynwhat nagen donmost nofnthenalveolin thatnwilln b
en present n inn then lungsn forn lifen develop?
a. 6n months
b. 1n year
c. 1.5n years
d. 2n years
ANS:n C
Most n of n then postnataln formationn of nalveolin inn then infant noccursn overn thenfirst n 1.5nyearsn of nlife.n At
n2n yearsn of n age,nthennumbernof nalveolin variesn substantiallynamongnindividuals.nAftern2n yearsn ofn ag
e,n malesn haven moren alveolin thann don females.n Aftern alveolarn multiplicationn ends,n then alveolin cont
inuen ton increasen inn sizen untiln thoracicn growthn isn completed.
REF:n p.n 6
3. Thenrespiratoryntherapist n isn evaluatingn an newbornn withn mild nrespiratoryndistressn duen tontrachealn s
tenosis.n Duringn whichn period n of n lungn development n did n thisn problemn develop?
, a. Embryonal
b. Saccular
c. Canalicular
d. Alveolar
ANS:n A
Then initialn structuresn of n then pulmonaryn treen developn duringn thenembryonaln stage.n Errorsn inn deve
lopment n duringn thisn timen mayn result n inn laryngeal,n tracheal,n orn esophagealn atresian orn stenosis.nPu
lmonarynhypoplasia,n ann incompleten development n of nthenlungsn characterized n bynann abnormallyn l
ow n numbern and/orn sizen of n bronchopulmonaryn segmentsn and/orn alveoli,n cann developn duringn then
pseudoglandularn phase.n If n then fetusn isn bornn duringn then canalicularn phasen (i.e.,n prematurely),n sev
eren respiratoryn distressn cann ben expected n becausen theninadequatelyn developed nairways,n alongn wit
hn insufficient n and n immaturen surfactant nproductionn byn alveolarn typen II n cells,n givesn risen ton then co
nstellationn of n problemsn knownn asn infantn respiratoryn distressn syndrome.
REF:n n p.n 6
4. Whichnof nthen followingn mechanismsn isn(are)n responsiblen fornthenpossiblen associationn betweenn
oligohydramniosn and n lungn hypoplasia?
I. Abnormalncarbohydraten metabolism
II. Mechanicalnrestrictionn of n then chest n wall
III. Interferencenwithn fetalnbreathing
IV. Failurenton producen fetaln lungn liquid
a. I n and n III n only
b. II nand n III n only
c. I,n II,n and n IV nonly
d. II,n III,n and n IVnonly
ANS:n D
Oligohydramnios,n an reduced n quantitynof namnioticn fluid npresent n forn annextended n period n of n time,n
withn orn without n renaln anomalies,n isn associated n withnlungn hypoplasia.n Then mechanismsn byn whichn
amnioticnfluid n volumeninfluencesnlungn growthnremainn unclear.nPossiblen explanationsn forn reduce
d n quantityn ofn amnioticn fluid n includenmechanicaln restrictionn of n thenchest nwall,n interferencen withnf
etaln breathing,n orn failuren ton producen fetaln lungn liquid.n Thesen clinicaln and n experimentaln observati
onsn possiblyn point n ton an commonn denominator,n lungn stretch,n asn beingn an majorn growthn stimulant.
REF:n pp.n6-7
5. What n isn then purposen of n thensubstancen secreted n bynthentypen II npneumocyte?
a. Tonincreasenthen gasn exchangen surfacenarea
b. Tonreducensurfacentension
c. Tonmaintainn lungnelasticity
d. Tonpreserven thenvolumen of nthen amnioticn fluid
NEONATAL & PEDIATRIC RESPIR
n n n
ATORY CARE n
5th Edition, Walsh
n
n
TESTBANK n
,NeonatalnandnPediatricnRespiratorynCare,n5thnEdition,nBriannK.nWalshnTestnBank
Tablenof nContents
Chaptern 1.n Fetaln Lungn Development
Chaptern 2.n Fetaln Gasn Exchangen and n Circulation
Chaptern 3.n Antenataln Assessment n and n High-Riskn Delivery
Chaptern 4.n Examinationn and n Assessment n of n then Neonataln and n Pediatricn Patient
Chaptern 5.n PulmonarynFunctionn Testingn and n Bedsiden Pulmonaryn Mechanics
Chaptern 6.n Radiographicn Assessment
Chaptern 7.n Pediatricn Flexiblen Bronchoscopy
Chaptern 8.n Invasiven Blood n Gasn Analysisn and n Cardiovascularn Monitoring
Chaptern 9.n Noninvasiven Monitoringn inn Neonataln and n Pediatricn Care
Chaptern 10.n Oxygenn Administration
Chaptern 11.n Aerosolsn and n Administrationn of n Inhaled n Medications
Chaptern 12.n AirwaynClearancen Techniquesn and n Hyperinflationn Therapy
Chaptern 13.n AirwaynManagement
Chaptern 14.n Surfactant n Replacement n Therapy
Chaptern 15.n Noninvasiven Mechanicaln Ventilationn and n Continuousn Positiven Pressuren of n then Neonate
Chaptern 16.n Noninvasiven Mechanicaln Ventilationn of n then Infant n and n Child
Chaptern 17.n Invasiven Mechanicaln Ventilationn of n then Neonaten and n Pediatricn Patient
Chaptern 18.n Administrationn of n Gasn Mixtures
Chaptern 19.n Extracorporealn Membranen Oxygenation
Chaptern 20.n Pharmacology
Chaptern 21.n Thoracicn Organn Transplantation
Chaptern 22.n Neonataln Pulmonaryn Disorders
Chaptern 23.n Surgicaln Disordersn inn Childhood n that n Affect n RespiratorynCare
Chaptern 24.n Congenitaln Cardiacn Defects
Chaptern 25.n Pediatricn Sleep-Disordered n Breathing
Chaptern 26.n Pediatricn Airwayn Disordersn and n Parenchymaln Lungn Diseases
Chaptern 27.n Asthma
Chaptern 28.n Cysticn Fibrosis
Chaptern 29.n Acuten Respiratoryn Distressn Syndrome
Chaptern 30.n Shock
Chaptern 31.n Pediatricn Trauma
Chaptern 32.n Disordersn of n then Pleura
Chaptern 33.n Neurologicaln and n Neuromuscularn Disorders
Chaptern 34.n Pediatricn Emergencies
Chaptern 35.n Homen Caren of n then Postpartumn Family
Chaptern 36.n Qualityn and n Safety
,Chaptern1:nFetalnLungnDevelopment
Walsh:nNeonataln&nPediatricnRespiratorynCaren5thnEditionnTestnBankn(2020)
MULTIPLEnCHOICE
1. Whichn of nthen followingnphasesn ofnhumann lungndevelopment n isn characterized nbynthenformationno
f n an capillaryn networkn around n airwayn passages?
a. Pseudoglandular
b. Saccular
c. Alveolar
d. Canalicular
ANS:n D
Then canalicularn phasen followsn then pseudoglandularn phase,n lastingn fromn approximatelyn 17n week
snton26nweeksn of n gestation.nThisn phasenisnson named nbecausenof nthen appearancen of nvascularn chann
els,n orn capillaries,n whichn beginn ton grow n byn formingn an capillaryn networkn around n then airn passages
.n Duringn then pseudoglandularn stage,nwhichn beginsn atn dayn52n andnextendsntonweekn16n of n gestatio
n,n then airwayn systemn subdividesn extensivelyn and n then conductingn airwayn systemn develops,n endi
ngn withn then terminaln bronchioles.n Then saccularn stagen of n development,n whichn takesn placen fromn
weeksn 29n ton 36nofngestation,nisn characterized n bynthendevelopment n of n sacsn that n latern becomen alve
oli.n Duringn then saccularn phase,n an tremendousn increasen inn then potentialn gas-
n exchangingn surfacen arean occurs.n Then distinctionn betweenn then saccularn stagen andn then alveolarn st
agen isn arbitrary.n Then alveolarn stagen stretchesn fromn 39n weeksn of ngestationn ton term.n Thisn stagen isn r
epresented n byn then establishment n of n alveoli.
REF:n pp.n 3-5
2. Regardingnpostnataln lungn growth,nbynapproximatelynwhat nagen donmost nofnthenalveolin thatnwilln b
en present n inn then lungsn forn lifen develop?
a. 6n months
b. 1n year
c. 1.5n years
d. 2n years
ANS:n C
Most n of n then postnataln formationn of nalveolin inn then infant noccursn overn thenfirst n 1.5nyearsn of nlife.n At
n2n yearsn of n age,nthennumbernof nalveolin variesn substantiallynamongnindividuals.nAftern2n yearsn ofn ag
e,n malesn haven moren alveolin thann don females.n Aftern alveolarn multiplicationn ends,n then alveolin cont
inuen ton increasen inn sizen untiln thoracicn growthn isn completed.
REF:n p.n 6
3. Thenrespiratoryntherapist n isn evaluatingn an newbornn withn mild nrespiratoryndistressn duen tontrachealn s
tenosis.n Duringn whichn period n of n lungn development n did n thisn problemn develop?
, a. Embryonal
b. Saccular
c. Canalicular
d. Alveolar
ANS:n A
Then initialn structuresn of n then pulmonaryn treen developn duringn thenembryonaln stage.n Errorsn inn deve
lopment n duringn thisn timen mayn result n inn laryngeal,n tracheal,n orn esophagealn atresian orn stenosis.nPu
lmonarynhypoplasia,n ann incompleten development n of nthenlungsn characterized n bynann abnormallyn l
ow n numbern and/orn sizen of n bronchopulmonaryn segmentsn and/orn alveoli,n cann developn duringn then
pseudoglandularn phase.n If n then fetusn isn bornn duringn then canalicularn phasen (i.e.,n prematurely),n sev
eren respiratoryn distressn cann ben expected n becausen theninadequatelyn developed nairways,n alongn wit
hn insufficient n and n immaturen surfactant nproductionn byn alveolarn typen II n cells,n givesn risen ton then co
nstellationn of n problemsn knownn asn infantn respiratoryn distressn syndrome.
REF:n n p.n 6
4. Whichnof nthen followingn mechanismsn isn(are)n responsiblen fornthenpossiblen associationn betweenn
oligohydramniosn and n lungn hypoplasia?
I. Abnormalncarbohydraten metabolism
II. Mechanicalnrestrictionn of n then chest n wall
III. Interferencenwithn fetalnbreathing
IV. Failurenton producen fetaln lungn liquid
a. I n and n III n only
b. II nand n III n only
c. I,n II,n and n IV nonly
d. II,n III,n and n IVnonly
ANS:n D
Oligohydramnios,n an reduced n quantitynof namnioticn fluid npresent n forn annextended n period n of n time,n
withn orn without n renaln anomalies,n isn associated n withnlungn hypoplasia.n Then mechanismsn byn whichn
amnioticnfluid n volumeninfluencesnlungn growthnremainn unclear.nPossiblen explanationsn forn reduce
d n quantityn ofn amnioticn fluid n includenmechanicaln restrictionn of n thenchest nwall,n interferencen withnf
etaln breathing,n orn failuren ton producen fetaln lungn liquid.n Thesen clinicaln and n experimentaln observati
onsn possiblyn point n ton an commonn denominator,n lungn stretch,n asn beingn an majorn growthn stimulant.
REF:n pp.n6-7
5. What n isn then purposen of n thensubstancen secreted n bynthentypen II npneumocyte?
a. Tonincreasenthen gasn exchangen surfacenarea
b. Tonreducensurfacentension
c. Tonmaintainn lungnelasticity
d. Tonpreserven thenvolumen of nthen amnioticn fluid
