DAVISnADVANTAGEnFORnPATHOPHYSIOLOGYn2NDnEDITIONnCAPRIOTTInTESTnBANK
n Chapter n1,nThenCellninnHealthnandnIllness
MultiplenChoice
Identifynthen choice n thatn bestn completesn then statementn orn answersn then question.
n n 1.n Whichn statement n regardingn then sodium–potassiumn pumpn isn correct?
1. Then cell’sn plasman membranen isn moren solublen ton sodiumn ionsn thann potassiumn ions.
2. Then concentrationn of n sodiumn ionsn should n ben highern insiden then celln compartment.
3. Thenconcentrationn of npotassiumn ionsn should n benhighern outsiden then celln
compartment.
4. Then activen transport n involvesn pumpingn out n threen sodiumn ionsn and n pumpingn inn twon
potassiumn ions.
n 2.n Inn thenabsencen ofnoxygen,n whichn cellularn functionn createsnthensamen amount nof nenergynasn isn createdninnth
en presencen of n oxygen?
1. Dissipationn of n pyruvicn acid
2. Initiationn of n then citricn acid n cycle
3. Activationn of n acetyl-coenzymen A
4. Creationn of n acidosisn vian lacticn acid
n n 3.n How n manyn adenosinen triphosphatesn (ATPs)n aren produced n inn aerobicn energyn metabolism?
1.n n 2
2.n n 3
3.n n 34 NURSINGTB.COM
4.n n 53
4. Whichn celln organellesn differn inn theirn numbern accordingn ton then cell’sn energyn needs?
n
1. Ribosomes
2. Mitochondria
3. Ribonucleicn acids
4. Deoxyribonucleicn acids
n
5. Whichn optionn best n supportsn then reasonn moren energyn isn produced n whenn an personn isn exercising?
1. Exercisen causesn ann increasen inn then synthesisn of n protein.
2. Theren isn ann increasen inn then productionn of n pyruvicn acid n inn then cells.
3. Then conversionn of n pyruvicn acid n ton lacticn acid n isn increased n byn exercise.
n
4. Musclen cellsn haven moren mitochondrian ton meet n energyn demands.
6. Whenn doesn ribosomaln proteinn synthesisn cease?
1. Duringn endoplasmicn reticulumn stress
2. Duringn then synthesisn of n adenosinen triphosphaten (ATP)
3. Duringn an severen hypoxicn state
n 4. Duringn then processingn of n prohormone
7. Whichn cellularn organellesn aren responsiblen forn propellingn mucusn and n inhaled n debrisn out n of n then lungs?
1. Cilia
2. Microfilaments
3. Secretoryn vesicles
, DAVISnADVANTAGEnFORnPATHOPHYSIOLOGYn2NDnEDITIONnCAPRIOTTInTESTnBANK
4.n Endoplasmicn reticula
n n 8.n Whichn aren then keyn proteinsn inn then contractilen unitsn of n then musclen cells?
1. Actinn and n myosin
2. Prohormonen and n tubulin
3. Tubulinn and n actin
4. Myosinn and n prohormone
n n 9.n Whichn deficiencyn causesn Tay–Sachsn disease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomaln enzymes
n 10.n Whichn isn an characteristicn of n adrenoleukodystrophy?
1. Accumulationn of n ganglioside
2. Cessationn of n ribosomaln proteinn synthesis
3. Accelerationn of n cellularn proteasomen activity
4. Accumulationn of n long-chainn fattyn acidsn inn then nervousn system
n 11.n Whichn statement n regardingn endoplasmicn reticulumn (ER)n stressn isn correct?
1. Duringn ERn stress,n proteinsn aren rapidlyn degraded.
2. Duringn ERn stress,n lipidsn cannot n traveln ton theirn propern intracellularn locations.
3. Duringn ERn stress,n long-chainn fattyn acidsn accumulaten inn then nervousn system.
4. Duringn ERn stress,n nondeNgU
raR
deSdIsN
ubGsT
taB
nc.eCsn O
acMcu mulate n inn then cells.
n 12.nA nclient n isn diagnosed nwithn typen1n diabetesnmellitus.n At nancellularn level,n whichn functionn isn likelyn tonbenin
volved?
1. Inabilitynof n ribosomesn ton producen an specificn typen of n protein
2. Incorrect n processingn of n an proteinn bynthen Golgin apparatus
3. Stagnationn of n an previouslyn dynamicn actionn inn microtubules
4. Obstructionn of n then smoothn endoplasmicn reticulum
n 13.n Annewbornnpatient nexhibitsn characteristicsn of nseveren physicaln deformities.n Whichn cellularn component n i
sn examined n ton determinen then causen and n probabilityn of n then diseasen beingn geneticallyn transferred?
1. Transfern RNA
2. Ribosomaln RNA
3. Doublen helixn of n DNA
4. Mitochondrialn DNA
n 14.n Anhikern experiencesn musclen painn and nacidosisn whilen ascendingn anmountainn duringnan long,nsteepnc
limb.n Whichn isn then reasonn forn thesen manifestations?
1. Cellularn hypoxia
2. Autolysis
3. Heterolysis
4. Cellularn edema
n 15.n Whichn factorn providesn DNA n then uniquen molecularn abilityn ton replicate?
1. Then pairingn of n nitrogenousn bases
, DAVISnADVANTAGEnFORnPATHOPHYSIOLOGYn2NDnEDITIONnCAPRIOTTInTESTnBANK
2. Then presencen of n pyrimidinen bases
3. Then presencen of n nucleotides
4. Then nitrogenousn basen and n phosphaten bond
n 16.n How n manyn nitrogenousn basesn composen an singlen codon?
1.n n 2
2.n n 3
3.n n 4
4.n n 5
n 17.n Whichn componentsn formn then structuren of n DNA?
1. Nucleotides
2. Aminon acids
3. Fattynacids
4. Phosphates
n 18.n Whichn factorn isn essentialn inn ordern forn proteinn synthesisn ton occur?
1. Free-standingn ribosomesn withinn then cell
2. Proteinn blueprint n fromn then celln of n then DNA
3. Specificn informationn fromn then nucleusn of n then cell
4. Transfern RNA n ton moven then proteinn out n of n then cell
n 19.n Tetracyclinen antibioticn isn prescribed n fornann adult n client n withn chlamydian infection.n Whichn isn then
mechanismn of n actionn of n then drug?
1. Itn preventsn then replicatioNnUoR f n bSaI
ctNerGiaT.nB.COM
2. It n altersn then configurationn of n bacterialn cytoplasm.
3. It n interferesn withn then functionn of n bacterialn ribosomes.
4. It n inhibitsn then functionsn of n bacterialn mitochondria.
n 20.n Wheren doesn then conversionn of n an prohormonen inton an hormonen taken place?
1. Inn then ribosomes
2. Inn then Golgin apparatus
3. Inn then secretoryn granules
4. Inn then endoplasmicn reticulum
n 21.n Whichn isn then cell’sn “mastern mind”?
1. Nucleus
2. Ribosome
3. Golgin apparatus
4. Endoplasmicn reticulum
MultiplenResponse
Identifyn onen orn moren choicesn thatn bestn complete n then statementn orn answern then question.
n 22.n Whichn statementsn regardingn then microtubulesn aren true?n Selectn alln thatn apply.
1. Microtubulesn aren solid.
2. Microtubulesn aren flexible.
3. Microtubulesn aren composed n of n tubulin.
, DAVISnADVANTAGEnFORnPATHOPHYSIOLOGYn2NDnEDITIONnCAPRIOTTInTESTnBANK
4. Microtubulesn aren called n actinn filaments.
5. Microtubulesn haven structuresn involved n inn celln division.
n 23.n Whichn cellularn actionsn aren carried n out n duringn then processn of n proteinn synthesis?n Selectn alln thatn apply.
1. DNA n directsn then celln ton carryn out n then process.
2. Transcriptionn occursn inn then ribosomen of n then cell.
3. Proteinn translationn takesn placen inn then celln nucleus.
4. RNA n isn single-stranded n and n travelsn outsiden then nucleus.
5. Inn RNA,n then pyrimidinen basen thyminen isn replaced n withn uracil.
n 24.n Whichn characteristicsn aren specificn ton RNA n duringn proteinn synthesis?n Selectn alln thatn apply.
1. Presencen of n ribosen pentosen sugar
2. Presencen of n single-stranded n helix
3. Presencen of n double-stranded n helix
4. Presencen of n deoxyribosen pentosen sugar
5. Presencen of n uraciln and n cytosinen asn an pyrimidinen base
n 25.n Whichn aren then purinen basesn found n inn DNA?n Selectn alln thatn apply.
1. Uracil
2. Adenine
3. Guanine
4. Thymine
5. Cytosine
NURSINGTB.COM
n Chapter n1,nThenCellninnHealthnandnIllness
MultiplenChoice
Identifynthen choice n thatn bestn completesn then statementn orn answersn then question.
n n 1.n Whichn statement n regardingn then sodium–potassiumn pumpn isn correct?
1. Then cell’sn plasman membranen isn moren solublen ton sodiumn ionsn thann potassiumn ions.
2. Then concentrationn of n sodiumn ionsn should n ben highern insiden then celln compartment.
3. Thenconcentrationn of npotassiumn ionsn should n benhighern outsiden then celln
compartment.
4. Then activen transport n involvesn pumpingn out n threen sodiumn ionsn and n pumpingn inn twon
potassiumn ions.
n 2.n Inn thenabsencen ofnoxygen,n whichn cellularn functionn createsnthensamen amount nof nenergynasn isn createdninnth
en presencen of n oxygen?
1. Dissipationn of n pyruvicn acid
2. Initiationn of n then citricn acid n cycle
3. Activationn of n acetyl-coenzymen A
4. Creationn of n acidosisn vian lacticn acid
n n 3.n How n manyn adenosinen triphosphatesn (ATPs)n aren produced n inn aerobicn energyn metabolism?
1.n n 2
2.n n 3
3.n n 34 NURSINGTB.COM
4.n n 53
4. Whichn celln organellesn differn inn theirn numbern accordingn ton then cell’sn energyn needs?
n
1. Ribosomes
2. Mitochondria
3. Ribonucleicn acids
4. Deoxyribonucleicn acids
n
5. Whichn optionn best n supportsn then reasonn moren energyn isn produced n whenn an personn isn exercising?
1. Exercisen causesn ann increasen inn then synthesisn of n protein.
2. Theren isn ann increasen inn then productionn of n pyruvicn acid n inn then cells.
3. Then conversionn of n pyruvicn acid n ton lacticn acid n isn increased n byn exercise.
n
4. Musclen cellsn haven moren mitochondrian ton meet n energyn demands.
6. Whenn doesn ribosomaln proteinn synthesisn cease?
1. Duringn endoplasmicn reticulumn stress
2. Duringn then synthesisn of n adenosinen triphosphaten (ATP)
3. Duringn an severen hypoxicn state
n 4. Duringn then processingn of n prohormone
7. Whichn cellularn organellesn aren responsiblen forn propellingn mucusn and n inhaled n debrisn out n of n then lungs?
1. Cilia
2. Microfilaments
3. Secretoryn vesicles
, DAVISnADVANTAGEnFORnPATHOPHYSIOLOGYn2NDnEDITIONnCAPRIOTTInTESTnBANK
4.n Endoplasmicn reticula
n n 8.n Whichn aren then keyn proteinsn inn then contractilen unitsn of n then musclen cells?
1. Actinn and n myosin
2. Prohormonen and n tubulin
3. Tubulinn and n actin
4. Myosinn and n prohormone
n n 9.n Whichn deficiencyn causesn Tay–Sachsn disease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomaln enzymes
n 10.n Whichn isn an characteristicn of n adrenoleukodystrophy?
1. Accumulationn of n ganglioside
2. Cessationn of n ribosomaln proteinn synthesis
3. Accelerationn of n cellularn proteasomen activity
4. Accumulationn of n long-chainn fattyn acidsn inn then nervousn system
n 11.n Whichn statement n regardingn endoplasmicn reticulumn (ER)n stressn isn correct?
1. Duringn ERn stress,n proteinsn aren rapidlyn degraded.
2. Duringn ERn stress,n lipidsn cannot n traveln ton theirn propern intracellularn locations.
3. Duringn ERn stress,n long-chainn fattyn acidsn accumulaten inn then nervousn system.
4. Duringn ERn stress,n nondeNgU
raR
deSdIsN
ubGsT
taB
nc.eCsn O
acMcu mulate n inn then cells.
n 12.nA nclient n isn diagnosed nwithn typen1n diabetesnmellitus.n At nancellularn level,n whichn functionn isn likelyn tonbenin
volved?
1. Inabilitynof n ribosomesn ton producen an specificn typen of n protein
2. Incorrect n processingn of n an proteinn bynthen Golgin apparatus
3. Stagnationn of n an previouslyn dynamicn actionn inn microtubules
4. Obstructionn of n then smoothn endoplasmicn reticulum
n 13.n Annewbornnpatient nexhibitsn characteristicsn of nseveren physicaln deformities.n Whichn cellularn component n i
sn examined n ton determinen then causen and n probabilityn of n then diseasen beingn geneticallyn transferred?
1. Transfern RNA
2. Ribosomaln RNA
3. Doublen helixn of n DNA
4. Mitochondrialn DNA
n 14.n Anhikern experiencesn musclen painn and nacidosisn whilen ascendingn anmountainn duringnan long,nsteepnc
limb.n Whichn isn then reasonn forn thesen manifestations?
1. Cellularn hypoxia
2. Autolysis
3. Heterolysis
4. Cellularn edema
n 15.n Whichn factorn providesn DNA n then uniquen molecularn abilityn ton replicate?
1. Then pairingn of n nitrogenousn bases
, DAVISnADVANTAGEnFORnPATHOPHYSIOLOGYn2NDnEDITIONnCAPRIOTTInTESTnBANK
2. Then presencen of n pyrimidinen bases
3. Then presencen of n nucleotides
4. Then nitrogenousn basen and n phosphaten bond
n 16.n How n manyn nitrogenousn basesn composen an singlen codon?
1.n n 2
2.n n 3
3.n n 4
4.n n 5
n 17.n Whichn componentsn formn then structuren of n DNA?
1. Nucleotides
2. Aminon acids
3. Fattynacids
4. Phosphates
n 18.n Whichn factorn isn essentialn inn ordern forn proteinn synthesisn ton occur?
1. Free-standingn ribosomesn withinn then cell
2. Proteinn blueprint n fromn then celln of n then DNA
3. Specificn informationn fromn then nucleusn of n then cell
4. Transfern RNA n ton moven then proteinn out n of n then cell
n 19.n Tetracyclinen antibioticn isn prescribed n fornann adult n client n withn chlamydian infection.n Whichn isn then
mechanismn of n actionn of n then drug?
1. Itn preventsn then replicatioNnUoR f n bSaI
ctNerGiaT.nB.COM
2. It n altersn then configurationn of n bacterialn cytoplasm.
3. It n interferesn withn then functionn of n bacterialn ribosomes.
4. It n inhibitsn then functionsn of n bacterialn mitochondria.
n 20.n Wheren doesn then conversionn of n an prohormonen inton an hormonen taken place?
1. Inn then ribosomes
2. Inn then Golgin apparatus
3. Inn then secretoryn granules
4. Inn then endoplasmicn reticulum
n 21.n Whichn isn then cell’sn “mastern mind”?
1. Nucleus
2. Ribosome
3. Golgin apparatus
4. Endoplasmicn reticulum
MultiplenResponse
Identifyn onen orn moren choicesn thatn bestn complete n then statementn orn answern then question.
n 22.n Whichn statementsn regardingn then microtubulesn aren true?n Selectn alln thatn apply.
1. Microtubulesn aren solid.
2. Microtubulesn aren flexible.
3. Microtubulesn aren composed n of n tubulin.
, DAVISnADVANTAGEnFORnPATHOPHYSIOLOGYn2NDnEDITIONnCAPRIOTTInTESTnBANK
4. Microtubulesn aren called n actinn filaments.
5. Microtubulesn haven structuresn involved n inn celln division.
n 23.n Whichn cellularn actionsn aren carried n out n duringn then processn of n proteinn synthesis?n Selectn alln thatn apply.
1. DNA n directsn then celln ton carryn out n then process.
2. Transcriptionn occursn inn then ribosomen of n then cell.
3. Proteinn translationn takesn placen inn then celln nucleus.
4. RNA n isn single-stranded n and n travelsn outsiden then nucleus.
5. Inn RNA,n then pyrimidinen basen thyminen isn replaced n withn uracil.
n 24.n Whichn characteristicsn aren specificn ton RNA n duringn proteinn synthesis?n Selectn alln thatn apply.
1. Presencen of n ribosen pentosen sugar
2. Presencen of n single-stranded n helix
3. Presencen of n double-stranded n helix
4. Presencen of n deoxyribosen pentosen sugar
5. Presencen of n uraciln and n cytosinen asn an pyrimidinen base
n 25.n Whichn aren then purinen basesn found n inn DNA?n Selectn alln thatn apply.
1. Uracil
2. Adenine
3. Guanine
4. Thymine
5. Cytosine
NURSINGTB.COM
