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NR 546 Midterm Exam 2023/2024 – A+ Answer Key | PMHNP Legal, Ethics, and Brain Anatomy Study Guide

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Comprehensive NR 546 Midterm Exam 2023/2024 with A+ graded answers. Covers chemical restraints, genetics, brain anatomy, legal/ethical issues, and psychotropic medication guidelines—an essential study guide for PMHNP students. BRAND NEW!!

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NR 546 MIDTERM EXAM
WITH ANSWERS GRADED A+
BRANDNEW!!
Update 2023/2024
1. What should the PMHNP consider when prescribing chemical restraints?

-allergy status

-prior med hx for adverse drug reactions r/t the meds ordered in the chemical restraint

-state regulations regarding chemical restrains must be reviewed

2. Are the PMHNP and other staff liable if the client has an allergic reaction or adverse

side effects to the drugs used for chemical restraint?

No.

The client has been court-ordered to take the prescribed medications and the standing order

for chemical restraints is approved. The PMHNP and other staff are not liable if

the patient has an allergic reaction or adverse side effects.

3. How does reviewing the genetic makeup of a client help guide the PMHNP in selecting

medication for clients?

-Genetic testing can assist by providing more information on how clients may respond to
certain psychotropic medications

-provides information on how a client may break down and metabolize medications

based on the cytochrome P450 system.

,4. Tanrıkulu and Erbaş (2020) investigated identical twins to determine the presence of

an inherited link for schizophrenia and why one twin may develop schizophrenia when

the other does not. When two people have

100% identical DNA, why don't both persons develop the exact illnesses?

Studies of identical Danish twins found that if one twin had schizophrenia, the other

twin had a 50% lifetime risk of developing schizophrenia

(Lemvigh et al., 2020). Why is there only half the risk?

Both environmental and psychosocial stressors can impact mental health. Although twins

may have identical genes, their gene expression may be different.



There may be an environmental exposure that turned a gene "on" that should have been

"off" for one twin to develop schizophrenia and not the other.

5. central sulcus

separates the frontal lobe from the parietal lobe

6. frontal lobe

associated with movement, intelligence, abstract thinking

7. broca's area

speech production

8. temporal lobe

involves object identification and auditory signals

9. cerebellum

coordination

,10.wernicke's area

speech comprehension

11.occipital lobe

primary visual area

12.parietal lobe

keeps us alert to what is going on around us

13.sensory cortex

pain, heat, and other sensations

14.motor cortex

movement

15.hippocampus

involved in both memory and anxiety

16.nucleus accumbens

involved in the reward process

17.thalamus

involved in sensory organ and motor command processing

18.striatum involved in complex motor actions, also links cognition to motor

actions

19.limbic system

includes circuits that are associated with pleasure and reward

20.basal ganglia

group of structures involved in voluntary motor movements

, 21.amygdala

involved in emotional regulation and perception of odors

22.corpus callosum controls the communication between the two brain

hemispheres

23.white matter contains nerve fibers that connect neurons from different regions into

functional circuits


24.grey matter

contains nerve cells and dendrites

25.brain tissue

made up of grey matter and white matter

26.dorsal striatum involved in complex motor actions and linkage of cognition to

motor actions

-main input area for basal ganglia

*activated when anticipating or engaging in pleasure

27.The field of epigenetics is rapidly growing and can help explain how gene expression is:

influenced by environmental factors and how epigenetics contributes to the

manifestation of mental illness

28.How does epigenetics impact a person's mental health?

internal or external factors activate portions of the genome that result in the

manifestation of mental health symptoms

-activation is often a result of a stressful event, which, when combined with the genetic

risk, results in the disease

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