AM
AHN 572 NEPHROLOGY EXAM QUESTIONS AND ANSWERS
WITH COMPLETE SOLUTIONS VERIFIED LATEST UPDATE
2025
Terms in this set (51)
diabetes type 1 deficiency of insulin secretion due to
autoimmune b cell destruction or latent
autoimmune disease of adulthood
diabetes type 2 non auto immune progressive loss of adequate
b cell insulin secretion frequently associated
with insulin resistance
stomach: accelerated gastric emptying and
carbohydrates metabolized into glucose
how dues each body pancreas : defective b- cell secretion
part contribute to type liver : excessive glucose production
2 dm
Clinical manifestations of polyuria, polydipsia, polyphagia, blurred vision,
type 1 DM include weight loss, muscle catabolism, paresthesia,
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and DKA
Clinical manifestations initially asymptomatic, polyuria, polydipsia,
of type 2 DM include polyphagia, skin infections, pruritis, vaginitis,
overweight/obese, acanthosis nigricans and
HHNK
Diagnosis DM - plasma > 126 on more than one occasion after an 8 hour
glucose fast, > 200 random check with symptoms of
DM
Diagnosis DM- impaired BG 100-125
fasting glucose
Diagnosis DM - oral 75mg glucose in 300cc H20 w/in 5 minutes,
glucose tolerance test performed in the AM, obtain samples at 0-120
min, the 2 hour value >140-199 impaired
glucose tolerance, > 200 DM
HgbA1C gold standard in diagnosis , reflect the state of
glycemia over 8-12 weeks, measure ev 3-4
months
What Hgb a1c value > 6.5
indicates diabetes
islet autoantbodies proteins produced by the immune system that
have been shown to be associated with type 1
diabetes. They are key markers for the
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diagnosis of type 1 diabetes
adults overweight or obesity, 1st degree
relative, high race ethnicity, hx of CVD,
Who should be screened hypertension, HDL cholesterol <34 and / or TGL
for DM > 250, pcos, physical inactivity. Ppl with
prediabetes, ppl with GDM should have lifelong
testing q 3 years, for everyone else start at 35
years
How often should DM every 3 years, more frequently if needed
testing occur
Pre-diabetes diagnosis FPG 100- 125, 2 hr plasma glucose 140-199, A1C 5.7-6.4
link between insulin resistance and both
hyperthyroidism and hypothyroidism.
Because thyroid hormones are essential for
carbohydrate metabolism, thyroid
TSH and diabetes dysfunction can affect
relationship insulin and lead to the development of type 2 diabetes.
Insulin resistance may occur due to
hyperthyroidism because of an increase in
fatty free acids in the bloodstream. In body fat,
the thyroid hormone stimulates lipolysis, the
breakdown of body fat into fatty acids.
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