NBME Surgery Exam Questions And Answers
Verified 100% Correct
Question 1
Answer to the Previous Question
-A
-The pathogenesis of HS is not fully understood; however, new evidence is
emerging [13]. As opposed to early theories that implicated apocrine glands as
the primary contributors to HS, most authors now support follicle-centered
theories for the pathogenesis of HS. Follicular occlusion, follicular rupture,
and an associated immune response appear to be important events in the
development of the clinical manifestations of HS. The basic principles that
underlie follicle-centered theories are reviewed below.
Source: UPTD.
Probably not will be asked in the real deal.
Question 2
Answer to the Previous Question
-B
,Question 3
Answer to the Previous Question
-A
-His bacterial overgrowth started after his Billroth II gastrectomy because
decreased gastric acid/decreased gastric motility can lead to bacterial
overgrowth. The treatment for bacterial overgrowth is antibiotics, so it got
better after tetracycline.
Question 4
Answer to the Previous Question
-B
-given the young age of patient and the fact that the vignette tells you that
there are absent "skin" finding of chronic hepatic disease (i.e. caput medusa,
spider telangiectasia, palmar erythema), the likely hood of this being eso
varices secondary to cirrhosis is relatively low. more than likely going to be a
mallory-wise tear with hypotension secondary to multiple bouts of emesis
more so than the volume of actual blood loss. regardless, you would
resuscitate and stbalizes your ABCs before proceding with UGE.
,Question 5
Answer to the Previous Question
-C
-ectopic if ruptured leads to hypotension, pain and peritoneal irritation (right
shoulder pain).
Question 6
Answer to the Previous Question
-A
-contrast enema to un-invert the intussusception. The currant jelly stools and
drawing up knees are classic, tell-tale signs
Question 7
, Answer to the Previous Question
-D
-cicatrization of the duodenal ulcer by fibrosis causing gastric outlet
obstruction evidenced by borborgymi, succusion splash.
Question 8
Answer to the Previous Question
-D
Question 9
Answer to the Previous Question
-D
Verified 100% Correct
Question 1
Answer to the Previous Question
-A
-The pathogenesis of HS is not fully understood; however, new evidence is
emerging [13]. As opposed to early theories that implicated apocrine glands as
the primary contributors to HS, most authors now support follicle-centered
theories for the pathogenesis of HS. Follicular occlusion, follicular rupture,
and an associated immune response appear to be important events in the
development of the clinical manifestations of HS. The basic principles that
underlie follicle-centered theories are reviewed below.
Source: UPTD.
Probably not will be asked in the real deal.
Question 2
Answer to the Previous Question
-B
,Question 3
Answer to the Previous Question
-A
-His bacterial overgrowth started after his Billroth II gastrectomy because
decreased gastric acid/decreased gastric motility can lead to bacterial
overgrowth. The treatment for bacterial overgrowth is antibiotics, so it got
better after tetracycline.
Question 4
Answer to the Previous Question
-B
-given the young age of patient and the fact that the vignette tells you that
there are absent "skin" finding of chronic hepatic disease (i.e. caput medusa,
spider telangiectasia, palmar erythema), the likely hood of this being eso
varices secondary to cirrhosis is relatively low. more than likely going to be a
mallory-wise tear with hypotension secondary to multiple bouts of emesis
more so than the volume of actual blood loss. regardless, you would
resuscitate and stbalizes your ABCs before proceding with UGE.
,Question 5
Answer to the Previous Question
-C
-ectopic if ruptured leads to hypotension, pain and peritoneal irritation (right
shoulder pain).
Question 6
Answer to the Previous Question
-A
-contrast enema to un-invert the intussusception. The currant jelly stools and
drawing up knees are classic, tell-tale signs
Question 7
, Answer to the Previous Question
-D
-cicatrization of the duodenal ulcer by fibrosis causing gastric outlet
obstruction evidenced by borborgymi, succusion splash.
Question 8
Answer to the Previous Question
-D
Question 9
Answer to the Previous Question
-D
