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NR 507 CARDIOVASCULAR EXAM WITH CORRECT QUESTIONS AND ANSWES 2025

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NR 507 CARDIOVASCULAR EXAM WITH CORRECT QUESTIONS AND ANSWES 2025

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NR 507

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NR 507 CARDIOVASCULAR EXAM WITH
CORRECT QUESTIONS AND ANSWES
2025

Cardiovascular disorders - CORRECT-ANSWERSCardiovascular disorders

are prevalent in primary care. Many of the disorders develop over several

years, due to the risk factors to which individuals have been exposed. For

each disorder covered in this unit, a discussion of risk factors will be

included. For the concepts covered below, clinical application of each disease

will be provided so that students can understand the importance of

pathophysiology in diagnosing and treating the disease.



Prerequisite knowledge:

For this content, you should have a basic knowledge of cardiac anatomy;

know the differences between the right and left sides of the heart, in terms

of structure and function. You should also possess solid knowledge of the

unidirectional blood flow through the heart. For example, deoxygenated

blood arrives to the right side of the heart, travels to the pulmonary arteries

to release CO2 and pick up oxygen. At this point, the oxygenated blood is

carried from the lungs through the pulmonary veins to the left side of the

heart where it eventually reaches the aorta to carry oxygenated blood out to

the body organs. The cellular physiology related to cardiac contraction is

another important basic concept to know, as electrolytes (sodium, potassium

, and calcium) play a major role in muscle contraction. Finally, the concepts of

preload, afterload, and contractility are essential to understand, as all of

these can be affected in some way when a person has cardiovascular

disease.

What is Coronary Artery Disease (CAD)? - CORRECT-ANSWERSCAD is

considered the leading cause of death in the United States (U.S.). It is the

result of longstanding atherosclerosis.



Atherosclerosis begins with damage to the endothelium. It is the

endothelium, under normal functioning that maintains balance between the

vasoconstrictive and vasodilation actions, prevents platelets from

aggregating and control of the production of fibrin. When the endothelium

becomes damaged, our familiar inflammatory processes occur. Macrophages

attach to the endothelium, setting up phagocytosis; plaque formation and

vasoconstriction also occurs marking the beginning of atherosclerosis. The

plaque lesions located in the vessels become enlarged which allows the

plaque to progress within the enlarged vessel lumen. The plaque lesion

disrupts normal blood flow and causes thrombus formation which can be

triggered by cardiac risk factors such as elevated LDL, cholesterol, smoking

and diabetes.



So, why is this a problem?

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