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Week 5 NUR 6111 Cardiovascular Disorders | Complete 2026/2027 Study Guide & Notes | William Paterson University

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Ace your NUR 6111 course at William Paterson University with this comprehensive Week 5 study guide focused on cardiovascular disorders. This complete set of notes breaks down complex cardiovascular concepts into clear, concise information tailored for nursing graduate students. Explore pathophysiology, diagnostic methods, treatment protocols, and nursing interventions related to hypertension, myocardial infarction, heart failure, arrhythmias, and more. Crafted to enhance understanding and exam performance, these notes cover essential topics, clinical implications, and evidence-based care strategies. Perfect for quick reviews, test prep, or deep-dive studies in 2025 nursing curriculum. NUR 6111, cardiovascular disorders, nursing study guide, William Paterson University nursing, Week 5 notes, nursing cardiovascular pathophysiology, heart failure nursing, myocardial infarction nursing care, arrhythmia nursing, hypertension nursing management, nursing exam prep, cardiovascular nursing interventions, graduate nursing course, nursing pharmacology cardiovascular, cardiovascular nursing assessment NUR 6111, cardiovascular disorders, nursing notes, William Paterson University, nursing study guide, heart failure, myocardial infarction, hypertension, nursing interventions, graduate nursing

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NUR 6111 WEEK 5
Cardiovascular Disorders
Complete Notes & Study Guide
William Paterson University
Verified Content

What You’ll Get:
Complete Week 5 Lecture Notes
Clinical Guidelines + Case Study Applications
Medications (first-line, second-line, monitoring)
Diagnostic Tools (EKG, Echo, ABI, BNP, ASCVD Calculator)
Nursing & NP Clinical Decision-Making

,●Atrial Fibrillation
○ Impact of Atrial Fibrillation
■ It increases a person’s risk of stroke by 4 to 5 times.
■ AFib causes about 25% of ischemic strokes by causing blockage of blood
flow to the brain.
■ AFib doubles the risk of heart-related deaths.
■ The absence of contraction of the atria can result in a loss of cardiac
output anywhere from 15 - 30% due to the absence of an "atrial kick,"
contributing to heart failure.
○ Risk Factors
■ Advancing age*
■ High blood pressure*
■ Coronary artery disease*
■ Cardiomyopathy
■ Obesity
■ European ancestry
■ Athletes
■ Diabetes
■ Heart failure
■ Hyperthyroidism
■ Chronic kidney disease
■ Heavy alcohol use
■ Rheumatic heart disease
■ Valvular heart disease
■ Enlargement of the chambers on the left side of the heart
■ Pericarditis/myocarditis
■ Sleep Apnea
○ Valvular vs Non-Valvular AFib
■ Valvular: AF in the presence of moderate-to-severe mitral stenosis
(potentially requiring surgical intervention) or in the presence of an
artificial (mechanical) heart valve
■ Non-valvular: AF in the absence of moderate-to-severe mitral stenosis or a
mechanical heart valve
○ Clinical Presentation
■ Many people do not experience any symptoms and are unaware of their diagnosis –
Sometimes it is an incidental finding upon examination
■ Symptomatic presentation:
● General fatigue
● Rapid and irregular heartbeat
● Fluttering or “thumping” in the chest
● Dizziness
● Shortness of breath and anxiety
● Weakness
● Faintness or confusion

, ● Fatigue when exercising
● Sweating
● *Chest pain or pressure- (Call 911)
○ Physical Exam
■ On PE: Irregular heart rhythm – this is the hallmark finding of AFib,
tachycardia (typically 110s-140s), hypotension
■ Possible findings in someone with AF include:
● JVD, rales or effusions from HF
● Murmurs suggest stenosis or regurgitation.
● LE edema (HF or DVT),
● Exophthalmia (*bulging eyes)
● Signs of stroke, including facial droop, arm weakness, and slurred
speech
■ Possible PE findings correspond with diseases coexisting with AF or
being high risk for AF rather than the disease presentation itself
○ EKG
■ There are no visible P waves (no measurable PR interval) & an irregularly
irregular QRS complex. The ventricular rate is frequently fast.
■ *GOLD standard










○ Classifying AFib
■ Paroxysmal AF
● AF that terminates spontaneously or with intervention within 7
days of onset
● Episodes may recur with variable frequency
■ Persistent AF

, ● Continuous AF that is sustained >7 days
■ Long-standing persistent AF
● Continuous AF >12 months in duration
■ Permanent AF
● The term “permanent AF” is used when the patient & clinician
decide to stop further attempts to restore &/or maintain sinus
rhythm.
● Acceptance of AF represents a therapeutic attitude on the part of the
patient and clinician rather than an inherent pathophysiological
attribute of AF
● Acceptance of AF may change as symptoms, efficacy of therapeutic
interventions, & patient & clinician preferences evolve.
■ Nonvalvular AF
● AF in the absence of rheumatic mitral stenosis, a mechanical or
bioprosthetic heart valve, or mitral valve repair.
○ Labs
■ CBC
■ CMP
■ TSH
■ BNP
■ PT/PTT/INR
○ Other Diagnostic Tests
■ Echocardiogram - evaluation of size & function of atria & ventricles;
detection of valvular heart disease, left ventricular hypertrophy, &
pericardial disease.
■ Transesophageal Echocardiogram (TEE) - the most sensitive & specific
technique to detect LA thrombi.
■ Event Recorders (Implantable loop recorders, Holter monitors) - identify
arrhythmia if intermittent, correlate symptoms, or use rate control strategies
■ Stress test
■ EP study
○ Management
■ Goals of Treatment for AF
● Prevention of Thromboembolism – utilize the Chadsvasc score to
determine stroke risk
● Rate Control (preferred over rhythm control)
○ Beta-Adrenergic Receptor Blockers
■ atenolol, metoprolol, nadolol, propranolol, & sotalol
○ Nondihydropyridine Calcium Channel Blockers
■ diltiazem and verapamil
○ Digitalis Glycoside
■ Digoxin
● Rhythm Control

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