8/17/25, 8:30
AM
ENDOCRINE DISORDERS: NRS 340 EXAM 5
CONTENT EXAM QUESTIONS AND ANSWERS
WITH COMPLETE SOLUTIONS VERIFIED LATEST
UPDATE GRADED A++ 2025/2026
Terms in this set (65)
-regulates blood calcium and phosphate levels
(inverse relationship between the two)
parathyroid glands -parathyroid hormone (PTH) is stimulated by low serum
calcium levels
-PTH helps regulate levels by stimulating bone
resorption (calcium out of bones), renal tubular
conservation of calcium, and stimulates
intestinal absorption
1. parathyroid: PTH controls calcium levels
2. thyroid: controls T3 and T4 responsible for metabolism
and temp
function of organ 3. adrenal: stress response, control blood sugar, burn fat
review: parathyroid, for energy
thyroid, adrenal 4. pituitary: master gland controls parathyorid,
cortex, pituitary, thyroid, adrenals. secretes TSH, ADH, GH, LH
hypothalamus, pineal body and FSH
5. hypothalamus: sends singals to the
pitutary so need for control of any of the
1/
23
,8/17/25, 8:30
AM
following
6. pineal body: responsible for melatonin and sleep
-increased secretion of PTH
-so have hypercaclemia (calcium calms so increased
hyperparathyroidism calm effect)
-effects more women than men
-50% of pediatric patients show clinical manifestations
while 50% have no s/s
-loss of calcium IN the bones so osteoporosis and bone
pain
-children will complain of bone or joint pain
-kidney stones
-neuromuslcar abnormalities (weakness, bc calcium
calms)
clinical
-hypo-reflexia
manifestation
-loss of appetite
s of
hyperparath -constipation
yroidism -increased need for sleep
-shortened attention span
-more serious is pancreatitis, renal failure, cardiac
changes fractures
**all RT to increased calcium
2/
23
, 8/17/25, 8:30
AM
-x rays find bone demineralization and bone cysts
-serum calcium exceeds 10-11
-serum phosphorus below 3
diagnostic tests for -urine calcium, serum chloride, uric acid,
hyperparathyroidism creatinine, amylase and alkaline phosphatase
increased
-loss of lamina dura around teeth so need dental visits,
check for dental changes
nursing consideration for -hypercalcemia causes sensitivity to digitalis so toxicity
hyperparathyroidism quickly develops
-increased susceptibility to digitalis induced
arrhythmias
-s/s are vomiting, yellow halo vision
-ambulation, avoid immobility
-high fluids and moderate calcium intake (acidic juices
non surgical and 3000 ml/day)
treatment for -can use drugs like alendronate (fosamax), estrogen or
hyperparathy progestin, oral phosphates or pamidronate, diuretics ,
roidism and calcimimetic agnets (Sensipar)
-take a multivitamin
-surgical removal of the gland causing the
hypersecretion
-early surgical intervention may reduce bone damage
surgical management of but not renal disease
hyperparathyrodisim -post op care is to monitor for s/s of
hypocalcemia because of the sudden decrease
in calcium levels
3/
23
AM
ENDOCRINE DISORDERS: NRS 340 EXAM 5
CONTENT EXAM QUESTIONS AND ANSWERS
WITH COMPLETE SOLUTIONS VERIFIED LATEST
UPDATE GRADED A++ 2025/2026
Terms in this set (65)
-regulates blood calcium and phosphate levels
(inverse relationship between the two)
parathyroid glands -parathyroid hormone (PTH) is stimulated by low serum
calcium levels
-PTH helps regulate levels by stimulating bone
resorption (calcium out of bones), renal tubular
conservation of calcium, and stimulates
intestinal absorption
1. parathyroid: PTH controls calcium levels
2. thyroid: controls T3 and T4 responsible for metabolism
and temp
function of organ 3. adrenal: stress response, control blood sugar, burn fat
review: parathyroid, for energy
thyroid, adrenal 4. pituitary: master gland controls parathyorid,
cortex, pituitary, thyroid, adrenals. secretes TSH, ADH, GH, LH
hypothalamus, pineal body and FSH
5. hypothalamus: sends singals to the
pitutary so need for control of any of the
1/
23
,8/17/25, 8:30
AM
following
6. pineal body: responsible for melatonin and sleep
-increased secretion of PTH
-so have hypercaclemia (calcium calms so increased
hyperparathyroidism calm effect)
-effects more women than men
-50% of pediatric patients show clinical manifestations
while 50% have no s/s
-loss of calcium IN the bones so osteoporosis and bone
pain
-children will complain of bone or joint pain
-kidney stones
-neuromuslcar abnormalities (weakness, bc calcium
calms)
clinical
-hypo-reflexia
manifestation
-loss of appetite
s of
hyperparath -constipation
yroidism -increased need for sleep
-shortened attention span
-more serious is pancreatitis, renal failure, cardiac
changes fractures
**all RT to increased calcium
2/
23
, 8/17/25, 8:30
AM
-x rays find bone demineralization and bone cysts
-serum calcium exceeds 10-11
-serum phosphorus below 3
diagnostic tests for -urine calcium, serum chloride, uric acid,
hyperparathyroidism creatinine, amylase and alkaline phosphatase
increased
-loss of lamina dura around teeth so need dental visits,
check for dental changes
nursing consideration for -hypercalcemia causes sensitivity to digitalis so toxicity
hyperparathyroidism quickly develops
-increased susceptibility to digitalis induced
arrhythmias
-s/s are vomiting, yellow halo vision
-ambulation, avoid immobility
-high fluids and moderate calcium intake (acidic juices
non surgical and 3000 ml/day)
treatment for -can use drugs like alendronate (fosamax), estrogen or
hyperparathy progestin, oral phosphates or pamidronate, diuretics ,
roidism and calcimimetic agnets (Sensipar)
-take a multivitamin
-surgical removal of the gland causing the
hypersecretion
-early surgical intervention may reduce bone damage
surgical management of but not renal disease
hyperparathyrodisim -post op care is to monitor for s/s of
hypocalcemia because of the sudden decrease
in calcium levels
3/
23
