Test Bank for Rubin's Pathology: Clinicopathologic Foundations of Medicine, 7th Edition

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RUBIN'S PATHOLOGY:b




CLINICOPATHOLOGIC
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FOUNDATIONS OF MEDICINE
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7thEditionByDavidS.Strayer,EmanuelRubin
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,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
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Table of Contents:
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Chapter 1: Cell Adaptation, Injury and Death
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Chapter 2: Inflammation
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Chapter 3: Repair, Regeneration and Fibrosis
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Chapter 4: Immunopathology
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Chapter 5: Neoplasia
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Chapter 6: Developmental and Genetic Diseases
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Chapter 7: Hemodynamic Disorders
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Chapter 8: Environmental and Nutritional Pathology
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Chapter 9: Infectious and Parasitic Diseases
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Section II: Pathogenesis of Systemic Conditions Expandable section
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Chapter 10: Aging
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Chapter 11: Systemic Autoimmune Diseases
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Chapter 12: Sepsis
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Chapter 13: Obesity and Diabetes Mellitus
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Chapter 14: The Pathology of Pregnancy
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Chapter 15: The Amyloidoses
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Section III: Diseases of Individual Organ SystemsExpandable section
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Chapter 16: Blood Vessels
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Chapter 17: The Heart
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Chapter 18: The Respiratory System
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Chapter 19: The Gastrointestinal Tract
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Chapter 20: The Liver and Biliary System
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Chapter 21: The Pancreas
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Chapter 22: The Kidney
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Chapter 23: The Lower Urinary Tract and Male Reproductive System
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Chapter 24: The Female Reproductive System and Peritoneum
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Chapter 25: The Breast
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Chapter 26: Hematopathology
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Chapter 27: The Endocrine System
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Chapter 28: The Skin
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Chapter 29: The Head and Neck
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Chapter 30: Bones, Joints and Soft Tissue
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Chapter 31: Skeletal Muscle and Peripheral Nervous System
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Chapter 32: The Central Nervous System
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Chapter 33: The Eye
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Chapter 34: Forensic Pathology
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,Rubin's Pathology: Clinicopathologic Foundations of Medicine Chapter
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1: Cell Adaptation, Injury and Death
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Ischemia and other toxic injuries increase the accumulation of intracellular calcium as a result of:
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1.
A) release of stored calcium from the mitochondria.
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B) improved intracellular volume regulation. b b b




C) decreased influx across the cell membrane. b b b b b




D) attraction of calcium to fatty infiltrates. b b b b b




The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
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2. Adaptation to the reduced size of the liver leads to _
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A) metaplasia
B) organ atrophy b




C) compensatory hyperplasia b




D) physiologic hypertrophy b




A person eating peanuts starts choking and collapses. His airway obstruction is partially
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cleared, but he remains hypoxic until he reaches the hospital. The prolonged cell hypoxia
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3. caused a cerebral infarction and resulting
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A) caspase activation b




B) coagulation necrosis b




C) rapid phagocytosis
b




D) protein p53 deficiency b b




Bacteria and viruses cause cell damage by
b b b b b b , which is unique from the intracellular
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4. damage caused by other injurious agents.
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A) disrupting the sodium/potassium ATPase pump b b b b




B) interrupting oxidative metabolism processes b b b




C) replicating and producing continued injury b b b b




D) decreasing protein synthesis and function b b b b




The patient has a prolonged interruption in arterial blood flow to his left kidney, causing hypoxic
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5. cell injury and the release of free radicals. Free radicals damage cells by:
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A) destroying phospholipids in the cell membrane. b b b b b




B) altering the immune response of the cell.
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C) disrupting calcium storage in the cell. b b b b b




D) inactivation of enzymes and mitochondria. b b b b

, 6. Injured cells have impaired flow of substances through the cell membrane as a result of:
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A) increased fat load. b b




B) altered permeability. b




C) altered glucose utilization. b b




D) increased surface receptors. b b




7. Reversible adaptive intracellular responses are initiated by:
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A) stimulus overload. b




B) genetic mutations. b




C) chemical messengers. b




D) mitochondrial DNA. b




8. Injured cells become very swollen as a result of:
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A) increased cell protein synthesis. b b b




B) altered cell volume regulation. b b b




C) passive entry of potassium into the cell.
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D) bleb formation in the plasma membrane.
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A diabetic patient has impaired sensation, circulation, and oxygenation of his feet. He steps on a
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piece of glass, the wound does not heal, and the area tissue becomes necrotic. The necrotic cell
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9. death is characterized by:
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A) rapid apoptosis. b




B) cellular rupture. b




C) shrinkage and collapse. b b




D) chronic inflammation. b




A 99-year-old woman has experienced the decline of cell function associated with age. A group
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10. of theories of cellular aging focus on programmed:
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A) changes with genetic influences. b b b




B) elimination of cell receptor sites. b b b b




C) insufficient telomerase enzyme. b b




D) DNA mutation or faulty repair.
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An 89-year-old female patient has experienced significant decreases in her mobility and
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stamina during a 3-week hospital stay for the treatment of a femoral head fracture. Which of the
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following phenomena most likely accounts for the patients decrease in muscle function that
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11. underlies her reduced mobility?
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A) Impaired muscle cell metabolism resulting from metaplasia
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B) Dysplasia as a consequence of inflammation during bone remodeling
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C) Disuse atrophy of muscle cells during a prolonged period of immobility
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