WGU Biochemistry C624 , WGU C785 Biochemistry OA & Lipid Structure
COMBINATION examination 2025 -2026 QUESTIONS WITH CORRECT
ANSWERS VERIFIED 100% GRADED A+
WGU C785 Biochemistry OA
During gluconeogenesis, how many ATP molecules are consumed for each
glucose molecule produced?
4
2
1
6
6
It takes 6 ATP molecules to produce glucose through gluconeogenesis.
Which molecule signals the liver to release glucose by breaking down
glycogen?
Insulin
Eicosanoids
ATP
Glucagon
Glucagon
Insulin release increases glycogenesis which forms new glucose.
Which molecule is regenerated with the production of lactate during anaerobic
metabolism?
FAD
NAD+
ADH
FADH2
NAD+
Fermentation regenerates NAD+ so that it can be used by glycolysis.
,During aerobic metabolism, acetyl-CoA is produced from which molecule?
Glycerol
Lactate
Pyruvate
Oxaloacetate
Pyruvate
Acetyl-COA is produced from the breakdown of pyruvate after glycolysis.
Why would a marathon runner consume a large meal full of complex
carbohydrates the night before her run?
To replenish cellular stores of protein
To replenish cellular stores of ketone bodies
To replenish cellular stores of glycogen
To replenish cellular stores of DNA
To replenish cellular stores of glycogen
A large amount of carbohydrates can be stored as glycogen and used during
exercise.
Why must red blood cells rely solely on glycolysis to make ATP?
Red blood cells lack mitochondria.
Red blood cells absorb too much oxygen.
Red blood cells lack cytoplasm.
Red blood cells cannot absorb oxygen.
Red blood cells lack mitochondria
Red blood cells lack mitochondria and must depend upon anaerobic metabolism to
produce ATP.
What are advanced glycation end products (AGEs)?
The end products of glycogenolysis
Glucose molecules linked to lipids or proteins without the need for enzymes
The end products of glycolysis
Glucose molecules linked to DNA or RNA without the need for enzymes
,Glucose molecules linked to DNA or RNA without the need for enzymes
The end product of glycogenolysis is glucose.
Why are blood glucose levels high in patients with unmanaged Diabetes
Mellitus?
Cells move too many GLUT4 transporters to their cell membranes.
Cells do not move enough GLUT4 transporters to their cell membranes.
Ketogenesis is suppressed in the liver.
Ketogenesis is stimulated in the liver.
Cells do not move enough GLUT4 transporters to their cell membranes.
GLUT4 transporters allow blood glucose to enter cells which then lowers blood
glucose levels.
What kind of molecule is glycogen?
A simple carbohydrate
A triglyceride
A protein
A complex carbohydrate
A complex carbohydrate
What happens in the muscle cells when they respond to insulin?
Beta-oxidation of fatty acids is increased.
Glycogenolysis is increased.
GLUT4 transporters move to the surface of the cell.
Gluconeogenesis is increased.
GLUT4 transporters move to the surface of the cell.
GLUT4 transporters move to the surface of the cell to allow glucose to enter the cell.
What is the function of fermentation during anaerobic metabolism?
Fermentation consumes the lactic acid produced by glycolysis.
Fermentation regenerates the NADH needed for glycolysis.
Fermentation consumes the NAD+ produced by glycolysis.
Fermentation regenerates the NAD+ needed for glycolysis.
, Fermentation regenerates the NAD+ needed for glycolysis.
Fermentation produces NAD+. NAD+ is needed as a substrate for glycolysis.
How does the proton gradient affect the pH of the mitochondrial matrix and the
intermembrane space?
The concentration of protons in the matrix is higher than the concentration in
the intermembrane space.
The concentration of protons in the intermembrane space is higher than the
concentration in the matrix.
The pH of the intermembrane space is higher than the pH of the matrix.
The pH of the intermembrane space is higher than the pH of the cytoplasm.
The concentration of protons in the intermembrane space is higher than the
concentration in the matrix.
The intermembrane space of mitochondria has a high concentration of hydrogen
ions (H+) which makes the pH very low compared to the matrix.
Which diet listed below would result in the production of ketone bodies in a
healthy individual?
A diet rich in fats and very low in carbohydrates
A diet rich in carbohydrates and very low in fat
A diet rich in protein and very low in fat
A diet rich in carbohydrates and very low in protein
A diet rich in fats and very low in carbohydrates
Many of our cell- and tissue-types cannot use fatty acids as a fuel source for making
ATP because they lack the capacity to carry out beta-oxidation of fatty acids.When
our only (or major) source of energy is fatty acids, the liver will do the job of breaking
them down into acetyl CoA for those cells that cannot do it for themselves. However,
owing to differences between the chemistry of the blood versus the chemistry of the
insides of our cells, we cannot ship acetyl CoA through the bloodstream. Therefore,
the liver will assemble the excess acetyl CoA it produces (from beta-oxidation of fatty
acids) into the ketone bodies, acetoacetate and 3-hydroxybutyrate. You can think of
ketone bodies as transport forms of acetyl units that can travel through the
bloodstream and be taken up by those cells that cannot break down fatty
acids.Those cells will convert the ketone bodies back to acetyl CoA and use that to
keep their citric acid cycles going so they can keep making ATP and stay alive. This,
after all, is the whole point of this exercise - to enable us to survive when our only
energy source is fatty acids.
What stimulates beta-oxidation of fatty acids?
The insulin signal
COMBINATION examination 2025 -2026 QUESTIONS WITH CORRECT
ANSWERS VERIFIED 100% GRADED A+
WGU C785 Biochemistry OA
During gluconeogenesis, how many ATP molecules are consumed for each
glucose molecule produced?
4
2
1
6
6
It takes 6 ATP molecules to produce glucose through gluconeogenesis.
Which molecule signals the liver to release glucose by breaking down
glycogen?
Insulin
Eicosanoids
ATP
Glucagon
Glucagon
Insulin release increases glycogenesis which forms new glucose.
Which molecule is regenerated with the production of lactate during anaerobic
metabolism?
FAD
NAD+
ADH
FADH2
NAD+
Fermentation regenerates NAD+ so that it can be used by glycolysis.
,During aerobic metabolism, acetyl-CoA is produced from which molecule?
Glycerol
Lactate
Pyruvate
Oxaloacetate
Pyruvate
Acetyl-COA is produced from the breakdown of pyruvate after glycolysis.
Why would a marathon runner consume a large meal full of complex
carbohydrates the night before her run?
To replenish cellular stores of protein
To replenish cellular stores of ketone bodies
To replenish cellular stores of glycogen
To replenish cellular stores of DNA
To replenish cellular stores of glycogen
A large amount of carbohydrates can be stored as glycogen and used during
exercise.
Why must red blood cells rely solely on glycolysis to make ATP?
Red blood cells lack mitochondria.
Red blood cells absorb too much oxygen.
Red blood cells lack cytoplasm.
Red blood cells cannot absorb oxygen.
Red blood cells lack mitochondria
Red blood cells lack mitochondria and must depend upon anaerobic metabolism to
produce ATP.
What are advanced glycation end products (AGEs)?
The end products of glycogenolysis
Glucose molecules linked to lipids or proteins without the need for enzymes
The end products of glycolysis
Glucose molecules linked to DNA or RNA without the need for enzymes
,Glucose molecules linked to DNA or RNA without the need for enzymes
The end product of glycogenolysis is glucose.
Why are blood glucose levels high in patients with unmanaged Diabetes
Mellitus?
Cells move too many GLUT4 transporters to their cell membranes.
Cells do not move enough GLUT4 transporters to their cell membranes.
Ketogenesis is suppressed in the liver.
Ketogenesis is stimulated in the liver.
Cells do not move enough GLUT4 transporters to their cell membranes.
GLUT4 transporters allow blood glucose to enter cells which then lowers blood
glucose levels.
What kind of molecule is glycogen?
A simple carbohydrate
A triglyceride
A protein
A complex carbohydrate
A complex carbohydrate
What happens in the muscle cells when they respond to insulin?
Beta-oxidation of fatty acids is increased.
Glycogenolysis is increased.
GLUT4 transporters move to the surface of the cell.
Gluconeogenesis is increased.
GLUT4 transporters move to the surface of the cell.
GLUT4 transporters move to the surface of the cell to allow glucose to enter the cell.
What is the function of fermentation during anaerobic metabolism?
Fermentation consumes the lactic acid produced by glycolysis.
Fermentation regenerates the NADH needed for glycolysis.
Fermentation consumes the NAD+ produced by glycolysis.
Fermentation regenerates the NAD+ needed for glycolysis.
, Fermentation regenerates the NAD+ needed for glycolysis.
Fermentation produces NAD+. NAD+ is needed as a substrate for glycolysis.
How does the proton gradient affect the pH of the mitochondrial matrix and the
intermembrane space?
The concentration of protons in the matrix is higher than the concentration in
the intermembrane space.
The concentration of protons in the intermembrane space is higher than the
concentration in the matrix.
The pH of the intermembrane space is higher than the pH of the matrix.
The pH of the intermembrane space is higher than the pH of the cytoplasm.
The concentration of protons in the intermembrane space is higher than the
concentration in the matrix.
The intermembrane space of mitochondria has a high concentration of hydrogen
ions (H+) which makes the pH very low compared to the matrix.
Which diet listed below would result in the production of ketone bodies in a
healthy individual?
A diet rich in fats and very low in carbohydrates
A diet rich in carbohydrates and very low in fat
A diet rich in protein and very low in fat
A diet rich in carbohydrates and very low in protein
A diet rich in fats and very low in carbohydrates
Many of our cell- and tissue-types cannot use fatty acids as a fuel source for making
ATP because they lack the capacity to carry out beta-oxidation of fatty acids.When
our only (or major) source of energy is fatty acids, the liver will do the job of breaking
them down into acetyl CoA for those cells that cannot do it for themselves. However,
owing to differences between the chemistry of the blood versus the chemistry of the
insides of our cells, we cannot ship acetyl CoA through the bloodstream. Therefore,
the liver will assemble the excess acetyl CoA it produces (from beta-oxidation of fatty
acids) into the ketone bodies, acetoacetate and 3-hydroxybutyrate. You can think of
ketone bodies as transport forms of acetyl units that can travel through the
bloodstream and be taken up by those cells that cannot break down fatty
acids.Those cells will convert the ketone bodies back to acetyl CoA and use that to
keep their citric acid cycles going so they can keep making ATP and stay alive. This,
after all, is the whole point of this exercise - to enable us to survive when our only
energy source is fatty acids.
What stimulates beta-oxidation of fatty acids?
The insulin signal
