advanced pathophysiology exam 1
Terms in this set (21)
-study of human physiologic
dysfunction in disease
(disruption of homeostasis and
feedback loops) 1110 specific
diseases have been identified
1. Etiology= causative mechanisms "why"
2. Epidemiology= risk factors
discuss the five essential and distribution in populations
components of "patterns" incidence and
pathophysiology prevalence of disease.
Incidence: #of new cases in a
given population within a
given time. Prevalence: # of
cases existing (both old and
new) in a given time
3. Pathogenesis= disease
mechanisms, sequence of
events that occurs from stimulus
to disease
4. Clinical manifestations=
signs (result of
exam/assessment),
symptoms and diagnostic
criteria
5. Outcomes= cure, remission, chronicity, death
, - atrophy: reduction in cell size
d/t decrease in work demands
or adverse environmental
conditions. physiologic:
disappear when stressor is
removed (thymus gland and
child development).
pathologic: remain when
stressor is removed d/t
decreased workload (brain
tissue + Alzheimer's disease)
discuss the causes--
physiologic and -hypertrophy: increase in tissue
pathologic--of atrophy, size d/t increase in work
hypertrophy, and demands. physiologic: muscles
hyperplasia and give w/ increased lifting, heart
examples muscles with
exercise/pregnancy- can return
to norm size. pathologic: after
donating a kidney to keep up
with workload, dilated
cardiomyopathy- never return
in size
-hyperplasia: increase in the
number of cells in an
organ/tissue that is still capable
of mitotic division. physiologic:
uterus with pregnancy.
pathologic: endometriosis
-Compensatory hyperplasia is
an adaptive mechanism that
enables certain organs to
regenerate. For example,
removal of part of the liver
leads to hyperplasia of the
remaining liver cells, callus=
thickening of skin d/t stimulus
-atrophy: cell shrink's d/t
decreased
workload/stimulus/nutrients
Terms in this set (21)
-study of human physiologic
dysfunction in disease
(disruption of homeostasis and
feedback loops) 1110 specific
diseases have been identified
1. Etiology= causative mechanisms "why"
2. Epidemiology= risk factors
discuss the five essential and distribution in populations
components of "patterns" incidence and
pathophysiology prevalence of disease.
Incidence: #of new cases in a
given population within a
given time. Prevalence: # of
cases existing (both old and
new) in a given time
3. Pathogenesis= disease
mechanisms, sequence of
events that occurs from stimulus
to disease
4. Clinical manifestations=
signs (result of
exam/assessment),
symptoms and diagnostic
criteria
5. Outcomes= cure, remission, chronicity, death
, - atrophy: reduction in cell size
d/t decrease in work demands
or adverse environmental
conditions. physiologic:
disappear when stressor is
removed (thymus gland and
child development).
pathologic: remain when
stressor is removed d/t
decreased workload (brain
tissue + Alzheimer's disease)
discuss the causes--
physiologic and -hypertrophy: increase in tissue
pathologic--of atrophy, size d/t increase in work
hypertrophy, and demands. physiologic: muscles
hyperplasia and give w/ increased lifting, heart
examples muscles with
exercise/pregnancy- can return
to norm size. pathologic: after
donating a kidney to keep up
with workload, dilated
cardiomyopathy- never return
in size
-hyperplasia: increase in the
number of cells in an
organ/tissue that is still capable
of mitotic division. physiologic:
uterus with pregnancy.
pathologic: endometriosis
-Compensatory hyperplasia is
an adaptive mechanism that
enables certain organs to
regenerate. For example,
removal of part of the liver
leads to hyperplasia of the
remaining liver cells, callus=
thickening of skin d/t stimulus
-atrophy: cell shrink's d/t
decreased
workload/stimulus/nutrients
