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Test Bank for Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives, 3rd Edition by Theresa Capriotti (F.A. Davis Company, 2024) | ISBN: 9781719648592 | All Chapters

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Test Bank for Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives, 3rd Edition by Theresa Capriotti (F.A. Davis Company, 2024) | ISBN: 9781719648592 | All Chapters Test Bank for Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives, 3rd Edition by Theresa Capriotti (F.A. Davis Company, 2024) | ISBN: 9781719648592 | All Chapters Test Bank for Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives, 3rd Edition by Theresa Capriotti (F.A. Davis Company, 2024) | ISBN: 9781719648592 | All Chapters

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Institution
Davis Advantage For Pathophysiology Introductory
Course
Davis Advantage for Pathophysiology Introductory

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i.zIntroductoryConcepts and Clinical Perspectives, 2nd .zi i.z i.z i.z i.z




Edition,TheresaCapriott .zi .zi




Chapter1,TheCellin Healthand Illness wm wm




MultipleChoice
Identify thechoicethatbestcompletesthestatementor answersthequestion.
wm wm




w m 1. Whichstatement regardingthesodium–potassiumpump is correct?
i.z wm m
w




1. Thecell’s plasmamembrane is moresolubletosodium ionsthan potassium ions.
w
m wm w
m wm m
w wm wm




2. Theconcentration ofsodium ions shouldbe higherinside thecellcompartment.
wm wm w
m wm w
m




3. Theconcentration of potassium ionsshould behigheroutsidethecell wm wm wm wm




compartment.
wm




4. Theactivetransportinvolvespumping outthree sodium ionsand pumpingin two i wm wm wm wm wm




potassium ions.
wm wm




2. Inthe absence of oxygen,which cellularfunction creates thesame amount ofenergy as is
w
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m w
m wm i wm




created in the presence of oxygen?
wm wm wm wm wm wm




1. Dissipationofpyruvic acid .zi




2. Initiationofthe citricacid cycle wm wm




3. Activationofacetyl-coenzymeA
4. Creationofacidosis vialactic acid wm wm




3. Howmanyadenosinetriphosphates(ATPs)areproducedinaerobicenergymetabolism? .zi




1. 2
2. 3
3. 34
4. 53

4. Whichcell organelles differintheirnumberaccording tothecell’s energy needs?
wm wm wm wm wm




1. Ribosomes
2. Mitochondria

, 3. Ribonucleicacids .zi




4. Deoxyribonucleic acids wm




5. w mWhichoptionbest supports the reasonmore energy is producedwhen aperson is exercising?
iwm i wm wm wm w
m w
m wm w
m w
m




1. Exercisecausesanincreasein thesynthesis ofprotein. wm wm




2. There is an increase in theproduction ofpyruvicacid inthe cells.
wm w
m wm w
m wm wm wm wm




3. Theconversionof pyruvic acid tolactic acid is increasedby exercise. w
m wm wm i wm wm w
m w
m




4. Musclecells havemore mitochondria tomeet energy demands.iwm wm wm iw m wm




6. w m When does ribosomal proteinsynthesiscease?
.zi i.z wm m
w




1. Duringendoplasmicreticulumstress
2. Duringthesynthesisofadenosinetriphosphate(ATP)
3. Duringaseverehypoxicstate
4. Duringtheprocessing ofprohormone wm




7. w m w m Whichcellular organellesare responsibleforpropellingmucus andinhaled debrisout ofthe lungs?
w
m w
m w
m m
w w
m m
w wm wm




1. Cilia
2. Microfilaments
3. Secretoryvesicles
4. Endoplasmicreticula

8. w m w m Whicharethekey proteinsin thecontractileunits ofthemuscle cells?
wm wm wm wm




1. Actinandmyosin
2. Prohormoneandtubulin
3. Tubulinandactin m
w




4. Myosinand prohormone w
m




9. WhichdeficiencycausesTay–Sachsdisease? .zi




1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomal enzymes wm




w m 10. w m w mWhichisa characteristic ofadrenoleukodystrophy?
wm i wm




1. Accumulationofganglioside
2. Cessationofribosomalproteinsynthesis
3. Accelerationofcellularproteasomeactivity
4. Accumulationoflong-chainfattyacidsinthenervoussystem i.z




w m 11. w m Whichstatementregardingendoplasmicreticulum(ER)stressiscorrect? .zi




1. DuringERstress,proteinsarerapidlydegraded.
2. DuringERstress,lipids cannot traveltotheir properintracellular locations. i wm i.z wm wm




3. DuringERstress,long-chainfattyacidsaccumulateinthe nervoussystem. w m




4. DuringERstress,nondegradedsubstancesaccumulateinthecells.
.zi

,12. Aclient is diagnosedwith type 1diabetes mellitus. Atacellular level,which function is likelyto be
wm w
m wm w
m w
m wm wm wm wm i wm wm




involved?
wm




1. Inability of ribosomes toproduceaspecific type ofprotein
wm w
m wm wm w
m




2. Incorrect processing ofa protein bythe Golgiapparatus
i wm w
m wm w
m w
m




3. Stagnationofapreviouslydynamicactioninmicrotubules m
w m
w m
w m
w




4. Obstructionofthe smoothendoplasmic reticulum
m
w w
m w
m




13. A newborn patient exhibits characteristics of severe physical deformities. Which
wm wm wm wm wm wm wm wm wm




cellular componentis examinedtodeterminethecauseandprobability of thediseasebeing
wm wm wm wm wm




genetically transferred?
wm wm




1. TransferRNA
2. RibosomalRNA
3. Doublehelix ofDNA wm




4. Mitochondrial DNA wm




14. Ahiker experiences muscle painandacidosis whileascending amountain duringa long,steep
wm wm w
m wm wm wm wm




climb. Which is the reason for these manifestations?
wm wm wm wm wm wm wm wm




1. Cellularhypoxia
2. Autolysis
3. Heterolysis
4. Cellularedema

, 15. Which factor provides DNA the unique molecular ability to replicate?
wm wm wm wm wm wm wm wm w
m




1. Thepairing ofnitrogenous bases w
m wm




2. Thepresenceof pyrimidine bases w
m wm




3. Thepresence of nucleotides w
m wm




4. Thenitrogenousbaseand phosphatebond m
w




16. How many nitrogenous bases compose a single codon?
wm wm wm wm wm wm wm




1. 2
2. 3
3. 4
4. 5

17. Which components form the structure of DNA?
wm wm wm wm wm wm




1. Nucleotides
2. Aminoacids
3. Fatty acids wm




4. Phosphates

18. Which factor is essential in order for protein synthesis to occur?
wm wm wm wm wm wm wm wm wm wm




1. Free-standingribosomeswithinthe cell .zi wm




2. Proteinblueprintfromthe cell of theDNA wm w
m w
m m
w




3. Specific information fromthenucleusofthecell
i.z wm




4. TransferRNA tomovetheproteinout of thecell i.z wm wm




19. Tetracyclineantibioticisprescribedforanadultclient with chlamydiainfection.Whichisthe .zi .z .z wm




mechanism of action of the drug?
wm wm wm wm wm wm




1. Itpreventsthereplicationofbacteria.
2. Italterstheconfigurationofbacterialcytoplasm. .zi




3. Itinterfereswiththe function ofbacterialribosomes. wm wm




4. It inhibitsthefunctions ofbacterialmitochondria.
wm wm




20. Where doestheconversionofa prohormone intoahormone take place?
wm wm wm i wm




1. Inthe ribosomes
m
w wm




2. Inthe Golgiapparatus
m
w w
m




3. Inthesecretorygranules
4. Inthe endoplasmic reticulum
w
m wm




21. Whichisthe cell’s “mastermind”?
m
w wm m
w




1. Nucleus
2. Ribosome
3. Golgiapparatus
4. Endoplasmicreticulum

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Institution
Davis Advantage for Pathophysiology Introductory
Course
Davis Advantage for Pathophysiology Introductory

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