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Relias - Fetal Heart Monitoring | With complete questions and answers | 2025/26

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Relias - Fetal Heart Monitoring | With complete questions and answers | 2025/26 /. uterine blood supply - Answer-- uterine arteries deliver oxygenated blood to spiral arteries which bring oxygen rich blood to intervillous space of placenta that has fetal capillaries - fetal capillaries carry the O2 rich blood to umbilical VEIN that goes to fetus -in contrast, the umbilical ARTERIES return waste products to that intervillous space that go into mother's venous system /.Potential issues that negatively affect fetal oxygenation - Answer-*Maternal Oxygenation:* asthma, hyper- or hypo- ventilation *Maternal Circulation:* decreased maternal cardiac output, hypotension, decreased Hgb *Placental O2 and CO2 Exchange:* postterm, abruption, HTN, hypotension, uterine tachysystole *Fetal circulation:* cord compression or occlusion /.Fetal hypoxemia - Answer-- can occur d/t reduced fetal O2 reserves, excessive uterine activity, or reduced uteroplacental blood flow - worsening fetal hypoxemia can lead to abnormal FHR patterns, mostly minimal or absent variability from acidemia /.(1) hypoxemia vs. (2) hypoxia - Answer-1 - reduce O2 in blood 2 - reduced O2 delivery at tissue level /.Fetal anaerobic metabolism - Answer-- occurs when long term O2 delivery is insufficient to meet cellular needs of tissues - results in production of lactic acid and other noncarbonic acids - ACIDOSIS is the presence of excessive acids in tissues /.acidosis - Answer-pH below 7.35 pH is low (acidosis is the process that leads to low blood pH, or acidemia) /.alkalosis - Answer-pH above 7.45 pH is high /.buffers - Answer-- help maintain acid base homeostasis - 2 major fetal buffers are plasma bicarbonate and hgb /.base excess and base deficit - Answer-- base deficit is expressed as a positive number - base excess is expressed as a negative number ~ they are equivalent and terms are used interchangeably ~ /.fetal acidosis - Answer-- when O2 is decreased to fetus, tissue hypoxia results in acidosis, which then shows a drop in pH, a loss of bicarb, and increase in base deficit /.acidemia - Answer-assoc w/ widespread, deleterious effects on vital organ and body function /.fetal hypoxia during birth - Answer-assoc w/ neonatal depression, low apgars, neonatal encephalopathy, and cerebral palsy /.respiratory acidosis - Answer-*low pH ( 7.10), high pCO2 ( 60), normal base deficit ( 12)* - increase of pCO2 for fetus that lowers pH but doesn't affect base deficit /.factors that contribute to resp acidosis - Answer-- sudden decrease in placental or cord perfusion - uterine tachysystole - maternal hypoventilation /.metabolic acidosis - Answer-*ph 7.10 , normal pCO2 (60), high base deficit (12)* - a higher base deficit (such as 12) has been assoc w/higher risk for severe neonatal complications

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Relias - Fetal Heart Monitoring | With
complete questions and answers |
2025/26

/. uterine blood supply - Answer-✅- uterine arteries deliver oxygenated blood to spiral
arteries which bring oxygen rich blood to intervillous space of placenta that has fetal
capillaries

- fetal capillaries carry the O2 rich blood to umbilical VEIN that goes to fetus

-in contrast, the umbilical ARTERIES return waste products to that intervillous space
that go into mother's venous system

/.Potential issues that negatively affect fetal oxygenation - Answer-✅*Maternal
Oxygenation:* asthma, hyper- or hypo- ventilation

*Maternal Circulation:* decreased maternal cardiac output, hypotension, decreased Hgb

*Placental O2 and CO2 Exchange:* postterm, abruption, HTN, hypotension, uterine
tachysystole

*Fetal circulation:* cord compression or occlusion

/.Fetal hypoxemia - Answer-✅- can occur d/t reduced fetal O2 reserves, excessive
uterine activity, or reduced uteroplacental blood flow

- worsening fetal hypoxemia can lead to abnormal FHR patterns, mostly minimal or
absent variability from acidemia

/.(1) hypoxemia vs. (2) hypoxia - Answer-✅1 - reduce O2 in blood

2 - reduced O2 delivery at tissue level

/.Fetal anaerobic metabolism - Answer-✅- occurs when long term O2 delivery is
insufficient to meet cellular needs of tissues

- results in production of lactic acid and other noncarbonic acids

- ACIDOSIS is the presence of excessive acids in tissues

, /.acidosis - Answer-✅pH below 7.35

pH is low

(acidosis is the process that leads to low blood pH, or acidemia)

/.alkalosis - Answer-✅pH above 7.45

pH is high

/.buffers - Answer-✅- help maintain acid base homeostasis

- 2 major fetal buffers are plasma bicarbonate and hgb

/.base excess and base deficit - Answer-✅- base deficit is expressed as a positive
number

- base excess is expressed as a negative number

~ they are equivalent and terms are used interchangeably ~

/.fetal acidosis - Answer-✅- when O2 is decreased to fetus, tissue hypoxia results in
acidosis, which then shows a drop in pH, a loss of bicarb, and increase in base deficit

/.acidemia - Answer-✅assoc w/ widespread, deleterious effects on vital organ and body
function

/.fetal hypoxia during birth - Answer-✅assoc w/ neonatal depression, low apgars,
neonatal encephalopathy, and cerebral palsy

/.respiratory acidosis - Answer-✅*low pH (< 7.10), high pCO2 (> 60), normal base
deficit ( < 12)*

- increase of pCO2 for fetus that lowers pH but doesn't affect base deficit

/.factors that contribute to resp acidosis - Answer-✅- sudden decrease in placental or
cord perfusion
- uterine tachysystole
- maternal hypoventilation

/.metabolic acidosis - Answer-✅*ph < 7.10 , normal pCO2 (<60), high base deficit
(>12)*

- a higher base deficit (such as > 12) has been assoc w/higher risk for severe neonatal
complications

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