NSG 430 Adult Health Nursing II
Questions with Answers, 100% Guarantee Pass
Grand Canyon University
NSG 430 Final Exam Study Guide
1. where can spider like angiomas be seen
Answer> checks, upper neck, shoulders
2. what are complications of liver cirrhosis
Answer> Esophageal varices Ascities
Hepatic encehalopathy
Hepatorenal syndrome
3. what are esophageal varices
Answer> enlarged veins at the lower end of the esophagus
4. what are esophageal varices important to monitor
Answer> fragile, do NOT tolerate HIGH PRESSURE
(risk for bursting.. leading to hemorrhage)
5. Ascities
,6. how to do we treat peripheral edema
Answer> Tx exogenous albumin
7. why is it important to manage peripheral edema
Answer> leads to increased BP, more importantly increase portal pressure!
8. What is gross ascites
Answer> increase in and pressure leading to an eversion of the umbilicus
Decrease in osmotic pressure
dehydration
dry skin, sunken eyes, weakness
9. what is gross ascites at risk for
Answer> spontaneous bacterial peritonitis -- due to alternations in the fluid
space (Ecoli)
10. what is hepatic encephalopathy
Answer> Neurotoxic effects of ammonia
Liver is unable to convert ammonia ( as it crosses the BBB)
11. what is the cause of ammonia
Answer> when the liver is unable to convert ammonia into urea.. leading to toxic
levels in the blood stream
,13. how soon can hepatic encephalopathy occur
Answer> can occur sunddely!
-important to do your neuro checks
14. what are the prime characteristics of hepatic encephalopathy
Answer> Asterixis , Fetor hepaticicus, apraxia
15. what is asterixis and where is it from
Answer> hand flapping tremor(occurs when hand are held out and pulled back)
16. what is hepaticicus
Answer> musty, sweet odor from breath
17. what is aprixia
Answer> difficult to perform tasks, or movement, even though they
understand what needs to be done
18. Cirrhosis
Answer> End stage liver disease
19. what is cirrhosis
Answer> tensive degeneration and destruction of the liver cells
- replaced with fibrosis and regenerative nodules
20. What is the MC cause of cirrhosis
Answer> MC is hep C ( chronic hep C
, 22. what are the late symptoms of LATE manifestations of cirrhosis
Answer> Jaundice, Dark urine,
LOW serum albumin
Clay colored stool ( indications of bile abnormality
23. Other unique s/s (late) liver cirrhosis
Answer> spider angiomas (small bright red center branches)
- palmar erythema
- hematologic disorders
( low WBC, RBC, platelets) --splenomegaly
- peripheral neuropathies
- gynecomastia
24. What is used to treat high levels of ammonia
Answer> Lactulose! & antibiotics (rifax- imin)
25. what is hepatorenal syndrome
Answer> Renal failure due to decreased blood flow to the kidneys ( from
constricted BV in the kidneys)
26. Aztemia
Answer> unable to concentrate urine and get rid of urea/ammonia via urination
Questions with Answers, 100% Guarantee Pass
Grand Canyon University
NSG 430 Final Exam Study Guide
1. where can spider like angiomas be seen
Answer> checks, upper neck, shoulders
2. what are complications of liver cirrhosis
Answer> Esophageal varices Ascities
Hepatic encehalopathy
Hepatorenal syndrome
3. what are esophageal varices
Answer> enlarged veins at the lower end of the esophagus
4. what are esophageal varices important to monitor
Answer> fragile, do NOT tolerate HIGH PRESSURE
(risk for bursting.. leading to hemorrhage)
5. Ascities
,6. how to do we treat peripheral edema
Answer> Tx exogenous albumin
7. why is it important to manage peripheral edema
Answer> leads to increased BP, more importantly increase portal pressure!
8. What is gross ascites
Answer> increase in and pressure leading to an eversion of the umbilicus
Decrease in osmotic pressure
dehydration
dry skin, sunken eyes, weakness
9. what is gross ascites at risk for
Answer> spontaneous bacterial peritonitis -- due to alternations in the fluid
space (Ecoli)
10. what is hepatic encephalopathy
Answer> Neurotoxic effects of ammonia
Liver is unable to convert ammonia ( as it crosses the BBB)
11. what is the cause of ammonia
Answer> when the liver is unable to convert ammonia into urea.. leading to toxic
levels in the blood stream
,13. how soon can hepatic encephalopathy occur
Answer> can occur sunddely!
-important to do your neuro checks
14. what are the prime characteristics of hepatic encephalopathy
Answer> Asterixis , Fetor hepaticicus, apraxia
15. what is asterixis and where is it from
Answer> hand flapping tremor(occurs when hand are held out and pulled back)
16. what is hepaticicus
Answer> musty, sweet odor from breath
17. what is aprixia
Answer> difficult to perform tasks, or movement, even though they
understand what needs to be done
18. Cirrhosis
Answer> End stage liver disease
19. what is cirrhosis
Answer> tensive degeneration and destruction of the liver cells
- replaced with fibrosis and regenerative nodules
20. What is the MC cause of cirrhosis
Answer> MC is hep C ( chronic hep C
, 22. what are the late symptoms of LATE manifestations of cirrhosis
Answer> Jaundice, Dark urine,
LOW serum albumin
Clay colored stool ( indications of bile abnormality
23. Other unique s/s (late) liver cirrhosis
Answer> spider angiomas (small bright red center branches)
- palmar erythema
- hematologic disorders
( low WBC, RBC, platelets) --splenomegaly
- peripheral neuropathies
- gynecomastia
24. What is used to treat high levels of ammonia
Answer> Lactulose! & antibiotics (rifax- imin)
25. what is hepatorenal syndrome
Answer> Renal failure due to decreased blood flow to the kidneys ( from
constricted BV in the kidneys)
26. Aztemia
Answer> unable to concentrate urine and get rid of urea/ammonia via urination
