Nurs 615 Exam IV Broad Questions with Correct and
Confirmed Graded A+ Answers. Assured Pass.
Terms in this set (81)
Patients with cystic fibrosis are often prescribed enzyme
How will you prescribe lipase,
replacement for pancreatic secretions each replacement drug has
protease, and amylase
amylase, lipase and protease components, however the drug is
components?
prescribed in units of lipase
Surgical resection of the tumor is the first treatment of choice
either my open laparotomy or laparoscopy either surgical option
What is the medication of choice requires prior treatment of
for hypertensive crisis with nonspecific irreversible adrenergic adraonoreceptor blocker
pheochromocytoma?
phenoxybenzamine or a shorter acting alpha antagonists,
prazosin, terazosin, and doxazosin. Mainly use
phenozibenamine in practice. Doing so promotes the surgery to
proceed while
minimizing the likelihood of severe intraoperative hypertension
which is likely when the tumor is manipulated.
, (Intermediate Acting) NPH
Onset-60-90 min after
administration, Peak 48 hrs
Duration 10-18 hrs.
(Short Acting) Regular Onset 30-
60 min Peak 2-4 hrs
Duration 6-10 hrs
What is the onset of action, peak
(Long Acting) Aspart, Lispro,
of action, and duration of action
Glulisine Onset less than 15
of each insulin
preparation? min
Peak 1-2 hrs
Duration 3-6 hrs
(Long Acting) Glargine,
Detemir Onset 1-2 hrs
Peak NO PEAK
Duration 24 hrs
Hypoglycemia- dizziness, confusion, diaphoresis, tachycardia
Identify the symptoms of Hyperglycemia- polyphagia, polydipsia, polyuria, blurred vision, and fatigue
hypoglycemia, hyperglycemia, Ketoacidosis- hallmark symptoms include acetone breath like nail
and ketoacidosis. polish remover or fruity breath. Also abdominal pain, nausea,
vomiting and sob.
When changing from NPH to The initial dose of glargine is reduced by 20% to prevent hypoglycemia.
glargine
insulin, how will you adjust the
patient's dose?
Decreases hyperglycemia by decreasing hepatic glucose production
called hepatic gluconeogenesis. The average person with type 2
diabetes has three times the rate of gluconeogenesis, metformin
treatment reduces this by over 1/3rd. The molecular mechanism
of metformin isn't completely understood. In addition to
How does metformin work?
suppressing
hepatic glucose production, metformin increases insulin
sensitivity, enhances peripheral glucose uptake by inducing the
, phosphorilization of glu4 enhancer
factor, decreases insulin induced suppression of fatty acid
oxidation, and decreases absorption of glucose from the GI
tract. Also of note* Metformin helps reduce LDL cholesterol
and triglyceride levels and is not associated with weight gain,
in some people it helps promote weight loss*
Metformin is not metabolized, it is cleared from the body by
What diagnostic testing is tubular secretion and is secreted unchanged in the urine.
required before and throughout Metformin is undetectable in blood plasma within 24 hrs of a
therapy with metformin? single oral dose the average elimination half-life in plasma is 6.2
hrs as it is
secreted in the urine you should check a serum crt to assess renal function.
The mechanism of DDP-4 inhibitors is to increase incretin levels
What is the action of gliptin? incretin are GLP1 and GIP which inhibit glucagon release in
which in turn increases insulin secretion,
decreases gastric emptying, and decreases blood glucose levels
They bind directly to a receptor in the pancreatic beta cell. These
How do GLP agonists work? agents work in the same pathway as the DPP-4 inhibitors as
mentioned above but are generally
considered more potent.
When should exenatide be 60 minutes prior to the morning and evening meal
administered?
Confirmed Graded A+ Answers. Assured Pass.
Terms in this set (81)
Patients with cystic fibrosis are often prescribed enzyme
How will you prescribe lipase,
replacement for pancreatic secretions each replacement drug has
protease, and amylase
amylase, lipase and protease components, however the drug is
components?
prescribed in units of lipase
Surgical resection of the tumor is the first treatment of choice
either my open laparotomy or laparoscopy either surgical option
What is the medication of choice requires prior treatment of
for hypertensive crisis with nonspecific irreversible adrenergic adraonoreceptor blocker
pheochromocytoma?
phenoxybenzamine or a shorter acting alpha antagonists,
prazosin, terazosin, and doxazosin. Mainly use
phenozibenamine in practice. Doing so promotes the surgery to
proceed while
minimizing the likelihood of severe intraoperative hypertension
which is likely when the tumor is manipulated.
, (Intermediate Acting) NPH
Onset-60-90 min after
administration, Peak 48 hrs
Duration 10-18 hrs.
(Short Acting) Regular Onset 30-
60 min Peak 2-4 hrs
Duration 6-10 hrs
What is the onset of action, peak
(Long Acting) Aspart, Lispro,
of action, and duration of action
Glulisine Onset less than 15
of each insulin
preparation? min
Peak 1-2 hrs
Duration 3-6 hrs
(Long Acting) Glargine,
Detemir Onset 1-2 hrs
Peak NO PEAK
Duration 24 hrs
Hypoglycemia- dizziness, confusion, diaphoresis, tachycardia
Identify the symptoms of Hyperglycemia- polyphagia, polydipsia, polyuria, blurred vision, and fatigue
hypoglycemia, hyperglycemia, Ketoacidosis- hallmark symptoms include acetone breath like nail
and ketoacidosis. polish remover or fruity breath. Also abdominal pain, nausea,
vomiting and sob.
When changing from NPH to The initial dose of glargine is reduced by 20% to prevent hypoglycemia.
glargine
insulin, how will you adjust the
patient's dose?
Decreases hyperglycemia by decreasing hepatic glucose production
called hepatic gluconeogenesis. The average person with type 2
diabetes has three times the rate of gluconeogenesis, metformin
treatment reduces this by over 1/3rd. The molecular mechanism
of metformin isn't completely understood. In addition to
How does metformin work?
suppressing
hepatic glucose production, metformin increases insulin
sensitivity, enhances peripheral glucose uptake by inducing the
, phosphorilization of glu4 enhancer
factor, decreases insulin induced suppression of fatty acid
oxidation, and decreases absorption of glucose from the GI
tract. Also of note* Metformin helps reduce LDL cholesterol
and triglyceride levels and is not associated with weight gain,
in some people it helps promote weight loss*
Metformin is not metabolized, it is cleared from the body by
What diagnostic testing is tubular secretion and is secreted unchanged in the urine.
required before and throughout Metformin is undetectable in blood plasma within 24 hrs of a
therapy with metformin? single oral dose the average elimination half-life in plasma is 6.2
hrs as it is
secreted in the urine you should check a serum crt to assess renal function.
The mechanism of DDP-4 inhibitors is to increase incretin levels
What is the action of gliptin? incretin are GLP1 and GIP which inhibit glucagon release in
which in turn increases insulin secretion,
decreases gastric emptying, and decreases blood glucose levels
They bind directly to a receptor in the pancreatic beta cell. These
How do GLP agonists work? agents work in the same pathway as the DPP-4 inhibitors as
mentioned above but are generally
considered more potent.
When should exenatide be 60 minutes prior to the morning and evening meal
administered?
