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EEG Board Exam 2025 – Complete Study Material with Correct Answers and Exam-Style Practice

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This document offers a comprehensive collection of study material for the 2025 EEG Board Exam, including exam-style questions paired with correct answers. It covers the core topics assessed on the exam, such as EEG interpretation, instrumentation, patient safety, and clinical applications. The content is structured to support efficient learning and help candidates understand the reasoning behind each answer.

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EEG BOARD EXAM 2025 WITH 100% CORRECT
ANSWERS,GUARANTEED SUCCESS
Terms in this set (96)


SSS/BETS (small sharp waves / Low voltage, short duration, diphasic spikes with a steep
benign epileptiform transients of descending limb. Usually seen in drowsiness and light sleep.
sleep)
SREDA (subclinical rhythmic Sharply contoured theta activity in the posterior head region. A
electrographic discharges of normal variant in older adults during wakefullness.
adults)
1-2 seconds of sharply contoured discharges in the posterior
14 and 6 positive spikes
head regions in light sleep. Presents in adolescents.
Midparietal low amplitude discharges. Occurs in young adults in
6 hz spike and wave
drowsiness and disappears in sleep.
Oscillating 10 hz waves seen in leads overylying the
My rhythm senserimotor cortex in the absence of movement. If a patient
moves or thinks about moving their contralateral limb, this
rhythm will be suppressed.
Symmetric monophasic sharp wave occuring predominantly in
Wickets
older adults during light sleep in temporal leads without
disruption of the background.
3 hz waves without an associated spike which can be seen during
3 hz slow wave activity
hyperventilation in childhood
RTTBD (rhythmic temporal theta 5-6 hz rhythmic waves in the temporal lobe. Seen in young adults during
bursts of drowsiness) drowsiness.

Unilateral high voltage iregular wave rhythms due to alteration of
Breach rhythm
conductance commonly seen in patients with a skull defect.
Positive downward deflection, maximal in the frontopolar leads,
Anterior eye blinks (bells
phenomenon) followed by a negative deflection from eye opening. Disappears
in sleep.
Rhythmic electropositive discharges in one or multiple
EKG
leads, most often in the occipital leads. Time locked and
synchronous with the EKG tracing.

, Rhythmic slow waves in a single lead due to a close pulsating
Pulse
vessel. Time locked but delayed after each QRS sample.
Very slow out of phase derivations involving anterior electrodes
Lateral eye movements due to movement of the positively charged cornea. Best
appreciated in drowsiness and early sleep when patient
experience rolling eye movements.
Extremely high frequency waves often generated from the
Muscle
frontalis and temporalis muscles. Usually spares central leads.
Disappears in sleep.
Diffuse, low frequency discharges produced by movements of
Glossokinetic
the negative tip of the tongue. Can be induced by saying "la la la
la", chewing, or sucking.
Single or multiple sharp waves localized to a single electrode
Electrode pop
without a surrounding field. Disappears by reapplying an
electrode.
Generalized in all leads, typically signifies global cerebral
GRDA (generalized rhythmic delta
activity) dysfunction, such as in a severe encephalopathy, but is not to be
a risk factor for seizure or seizure tendency.
Can be seen with a variety of pathologies including posterior
Frontally dominant GRDA
fossa lesions, intracranial lesions, and increased
intraventricular pressure.
Can be seen with focal lesions such a hemorrhage, tumor, or
LRDA (lateralized rhythmic delta)
stroke. Is associated with increased seizure risk/seizure
tendency.
Often seen with focal acute or subacute cerebral dysfunction,
LPDs (Lateralized periodic
discharges) such as with herpes simplex enchephalitis, stroke, abscess, or
subdural hematoma.
Felt to have highest seizure tendency of the 'ictal-interictal'
GPDs (Generalized periodic patterns. If seen clinically with rapidly progressive dementia it
discharges)
can be strongly suggestive of Creutzfeldt-Jakob Disease.
This disease can present with hyperammonemia and generalized
periodic waves with triphasic morphology. They are bilaterally
End stage liver disease
synchronous and usually frontally predominant and exhibit
three phases (i.e. negative, positive, negative). Triphasic waves
can also be seen in ESRD and other forms of metabolic

, encephalopathy.



Hemispheric asymmetry with the lower amplitude discharges
Subdural Hematoma
localizing to the affected hemisphere.
Due to the predilection for the temporal lobes, patients will
HSV Encephalitis
present with lateralized periodic discharges (LPDs) most
prominent in temporal leads.
Stroke Shows focal irregular theta/delta activity with LRDA or LPDs.
Generalized periodic discharges, spikes, and spike-waves with a
Creuztfeldt-Jakob Disease (CJD)
disorganized background.
Fatal familial insomnia (FFI) Loss of sleep spindles.
periodic high-amplitude complexes with high-amplitude
Subacute sclerosing bisynchronous delta waves, frontal rhythmic delta activity,
panencephalitis
generalized periodic discharges, electrodecremental periods
following EEG complexes, and focal spike and slow-waves.
Tay-Sachs disease Slow background with or without multifocal epileptiform discharges.
Alzheimer's disease (AD) Slow background with or without multifocal epileptiform discharges.
Angelman Syndrome Notched delta activity.
Lithium toxicity Generalized delta/theta activity with multifocal spikes.

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