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NURS 5354 Final Exam Review 2026/2027 – Pediatric Endocrine Disorders Questions and Verified Answers

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This document provides a comprehensive overview of pediatric endocrine exam content, including growth hormone deficiency, precocious puberty, diagnostic criteria, and clinical manifestations. It summarizes essential definitions, causes, symptoms, testing, and treatment principles aligned with NURS 5354 final exam expectations. The material is structured for rapid review and supports students preparing for advanced nursing endocrine exams.

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Nurs 5354 Final Exam Questions And
Answers With Verified Solutions 100%
Correct!!!
Growth Hormone Deficiency - ANSWER✔✔ Absence or deficiency of growth
hormone produced by the pituitary gland to stimulate the body to grow


Etiology of GH deficiency - ANSWER✔✔ Infections, trauma, brain tumors


Clinical manifestations of GH - ANSWER✔✔ Short height for child's age
Increased amount of fat around waist and in face
Emotional feelings about height or weight
Younger appearance than children of same age
Decreased muscle mass
Delayed skeletal maturation Delayed onset of puberty Delayed tooth development
Hypoglycemia


GH Diagnostics - ANSWER✔✔ Thyroid panel
Evaluate renal and liver function
Insulin like growth factors (will be low)
Growth Hormone Stimulant test
Bone density scan
Brain CT or MRI
Karotyping


GH Treatment - ANSWER✔✔ Most children receive subcutaneous injections

,Injections can be daily or three to four times per week and have increased growth
velocity at bedtime
GH must be refrigerated
Close monitoring of growth with endocrinology visits every 3 to 6 months
Treatment stops when growth plates fuse


Precocious puberty - ANSWER✔✔ The very early onset and rapid progression of
puberty


Before age 8 in girls
Before age 9 in boys


Etiology of Precocious Puberty - ANSWER✔✔ Hormone-secreting tumors
Brain injury caused by head trauma
Infection
Thyroid dysfunction
Ovarian dysfunction
Idiopathic (most cases)


Clinical manifestations of precocious puberty - ANSWER✔✔ Female: Breast
development, axillary hair, pubic hair, body odor, onset of menses, acne


Male: Testicular/Penile enlargement, axillary and chest hair, deepening voice, acne


Diagnostic evaluation of precocious puberty - ANSWER✔✔ Computed
tomographic scan or magnetic resonance imaging

,Bone density scan
Pelvic and adrenal ultrasound
Gonadotropin-releasing hormone stimulation test
Blood work: Testosterone, estrogen, LH, FSH
Treatment involves the suppression of puberty


congenital hypothyroidism - ANSWER✔✔ Condition present at birth that results
in lack of thyroid hormones; results in poor physical and mental development;
formerly called cretinism


Clinical manifestations of hypothyroidism - ANSWER✔✔ Lethargy
Weakness
Dry skin
Cold intolerance
Weight gain
Constipation
Coarse hair


Diagnostic evaluation for hypothyroidism - ANSWER✔✔ State-required
screening: TSH and T4
Low T4, elevated TSH, or both indicate hypothyroidism
Positive test results may be followed by scan for bone age
Blood tests before 48 hours after birth may be falsely interpreted because of the
rise in TSH immediately after birth


Hyperthyroidism - ANSWER✔✔ Excessive production of thyroid hormones

, Signs and symptoms of hyperthyroidism - ANSWER✔✔ Weakness, insomnia,
tachycardia, palpitations, dyspnea, emotional of people and extremes, "everything
speeds up"


Causes of hyperthyroidism - ANSWER✔✔ Graves Disease
Pituitary and thyroid tumors, thyroiditis, congenital due to transfer of
immunoglobulins from thyrotoxic mothers


Treatment of hyperthyroidism - ANSWER✔✔ PTU or methimazole to control the
gland
Radioactive iodine to ablate the gland
Propranolol to treat sympathetic sxs such as tremors/palpitations, etc.


Thyroidectomy


congenital adrenal hyperplasia - ANSWER✔✔ Genetic disease in which the
adrenal gland is overdeveloped, resulting in a deficiency of certain hormones and
an overproduction of others


Salt losing form CAH - ANSWER✔✔ No cortisol = hypoglycemia
No aldosterone = salt and water loss
Increased cortisol precursors
-17-hydroxyprogesterone = salt losing tendency
Increased androgens masculinization
Excess androgen production during fetal life is associated with salt-losing and
simple virilizing CAH and masculinizes the external genitalia of female infants

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