Types of fractures (a break in the continuity of a bone)
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Many types
Osteomalacia
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softening of the bones, typically through a deficiency of vitamin D or
calcium.
Etiology: small bowel, hepatobiliary and pancreas disorders
INADEQUATE/ DELAYED MINERALIZATION IS THE CAUSE!!
called "Rickets" in children.
pain, bone fractures, vertebral collapse, bone malformation.
Risk factors of gastric ulcers
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H. Pylori infection
duodenal reflux into stomach (acids, bile, and pancreatic fluids)
NSAIDs
Complete
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break all the way through the bone
Clinical manifestations of gastric ulcers
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Pain (located in the upper abdomen)
The pain occurs after eating (w/in the first hour after eating). Eating helps at
first. The food helps until the food moves into the duodenum. Pain happens
, when the stomach is empty!! There is increased acid in stomach when
empty and it causes the pain
Characteristics of gastric ulcers
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-usually occur between 50-70 years of age
-less common than duodenal ulcers
-increased risk of cancer!!
-develop in antral region of the stomach (near the acid-secreting mucosa
of the stomach body)
-primary defect in gastric ulcers is an abnormality that increases the
mucosal barrier's permeability to hydrogen ions. This is a vicious cycle. The
damaged mucosa liberates histamine which stimulates the increase of acid
and pepsinogen production, blood flow, and capillary permeability. The
disrupted mucosa becomes edematous and loses plasma proteins. And the
destruction of the small vessels causes bleeding
avulsion
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fragment of bone connected to a ligament or tendon breaks off the main
bone
closed
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Many types
Osteomalacia
,Give this one a try later!
softening of the bones, typically through a deficiency of vitamin D or
calcium.
Etiology: small bowel, hepatobiliary and pancreas disorders
INADEQUATE/ DELAYED MINERALIZATION IS THE CAUSE!!
called "Rickets" in children.
pain, bone fractures, vertebral collapse, bone malformation.
Risk factors of gastric ulcers
Give this one a try later!
H. Pylori infection
duodenal reflux into stomach (acids, bile, and pancreatic fluids)
NSAIDs
Complete
Give this one a try later!
break all the way through the bone
Clinical manifestations of gastric ulcers
Give this one a try later!
Pain (located in the upper abdomen)
The pain occurs after eating (w/in the first hour after eating). Eating helps at
first. The food helps until the food moves into the duodenum. Pain happens
, when the stomach is empty!! There is increased acid in stomach when
empty and it causes the pain
Characteristics of gastric ulcers
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-usually occur between 50-70 years of age
-less common than duodenal ulcers
-increased risk of cancer!!
-develop in antral region of the stomach (near the acid-secreting mucosa
of the stomach body)
-primary defect in gastric ulcers is an abnormality that increases the
mucosal barrier's permeability to hydrogen ions. This is a vicious cycle. The
damaged mucosa liberates histamine which stimulates the increase of acid
and pepsinogen production, blood flow, and capillary permeability. The
disrupted mucosa becomes edematous and loses plasma proteins. And the
destruction of the small vessels causes bleeding
avulsion
Give this one a try later!
fragment of bone connected to a ligament or tendon breaks off the main
bone
closed
Give this one a try later!