sliding filament theory
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theory that actin filaments slide toward each other during muscle
contraction, while the myosin filaments are still
what decreases cardiac muscle contraction
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low ATP levels; ischemia hypoxia or acidosis
what influences cardiac contractility (inotropic state)
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levels of electrolytes
High levels of ATP
level of oxygen available
synchronous muscle contraction
What happens in renal failure when the coagulation cascade is activated
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fibrin is deposited in the glomerular structure and decreases capillary
perfusion by causing blockages and further decreases GFR further
How does LV hypertrophy lead to left sided heart failure
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The hypertrophy is secondary to cardiac damage resulting in an enlarged
by weaker structure that holds more blood
,kidney stones
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Solid crystalline masses formed in the kidney, resulting from an excess of
insoluble salts or uric acid crystallizing in the urine; may become trapped
anywhere along the urinary tract.
What causes the semilunar valves to close?
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ventricles relax and intraventricular pressure falls, blood flows back from
the arteries, and fill the cusps of the semilunar valves
Causes of bronchitis
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-long term exposure to environmental irritants
-repeated episodes of acute infection (RSV infection in early infancy)
-Factors affecting gestational childhood lung development (preterm birth)
Erythrocyte function
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, transport oxygen and carbon dioxide
Stenosis of heart valve
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A narrowing of the valve opening, causing turbulent flow and enlargement
of the emptying chamber
Effect of hyperinflation of the alveolar
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-expanded thorax and hypercapnia (retention of CO2)
- respiratory acidosis
Process of high output failure
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- impaired oxygen delivery of excessive tissue oxygen demands cause
tissue hypoxia
- catecholamines initiation increase HR and stroke volume
- increased cardiac output is produced but depletes cardiac muscle
reserve overtime and leads to low output failure over time
Give this one a try later!
theory that actin filaments slide toward each other during muscle
contraction, while the myosin filaments are still
what decreases cardiac muscle contraction
,Give this one a try later!
low ATP levels; ischemia hypoxia or acidosis
what influences cardiac contractility (inotropic state)
Give this one a try later!
levels of electrolytes
High levels of ATP
level of oxygen available
synchronous muscle contraction
What happens in renal failure when the coagulation cascade is activated
Give this one a try later!
fibrin is deposited in the glomerular structure and decreases capillary
perfusion by causing blockages and further decreases GFR further
How does LV hypertrophy lead to left sided heart failure
Give this one a try later!
The hypertrophy is secondary to cardiac damage resulting in an enlarged
by weaker structure that holds more blood
,kidney stones
Give this one a try later!
Solid crystalline masses formed in the kidney, resulting from an excess of
insoluble salts or uric acid crystallizing in the urine; may become trapped
anywhere along the urinary tract.
What causes the semilunar valves to close?
Give this one a try later!
ventricles relax and intraventricular pressure falls, blood flows back from
the arteries, and fill the cusps of the semilunar valves
Causes of bronchitis
Give this one a try later!
-long term exposure to environmental irritants
-repeated episodes of acute infection (RSV infection in early infancy)
-Factors affecting gestational childhood lung development (preterm birth)
Erythrocyte function
Give this one a try later!
, transport oxygen and carbon dioxide
Stenosis of heart valve
Give this one a try later!
A narrowing of the valve opening, causing turbulent flow and enlargement
of the emptying chamber
Effect of hyperinflation of the alveolar
Give this one a try later!
-expanded thorax and hypercapnia (retention of CO2)
- respiratory acidosis
Process of high output failure
Give this one a try later!
- impaired oxygen delivery of excessive tissue oxygen demands cause
tissue hypoxia
- catecholamines initiation increase HR and stroke volume
- increased cardiac output is produced but depletes cardiac muscle
reserve overtime and leads to low output failure over time
