NURS 5315 UTA Exam 2 NEWEST 2026 WITH COMPLETE QUESTIONS AND
CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED
A+||BRAND NEW VERSION!!
HIV EIA (3rd generation immunoassay) - (answer)can use urine, saliva, or serum (most accurate), need to
wait until 12 weeks post exposure to see antibodies, >99% accurate
4th generation immunoassay- "gold standard"
measures P24 antigen
can test 10 days post exposure
Mast cell - (answer)Cellular bags of granules located in loose connective tisssue close to blood vessels.
Activation initiates inflammatory process.
Histamine - (answer)Causes vasodilation, increases vascular permeability, increases blood flow to the
site of injury- causes erythema and swelling.
Cytokines - (answer)Soluble factors that contribute to the regulation of innate or adaptive resistance by
affecting other neighboring cells. Can be pro-inflammatory or anti-inflammatory. Can react quickly or be
more delayed.
Leukotrines - (answer)Released when mast cells degranulate, prolong the inflammatory process. Cause
vasodilation, attract neutrophils, monocytes, and eosinophils.target of inhibition for singular.
Prostaglandins - (answer)Released when mast cells degranulate, are produced by the arachidonic
pathway. Cause vasodilation, platelet aggregation at site of injury, pain, and fever.
Chemotactic factors - (answer)Biochemical substance that attracts leukocyte to the site of inflammation
Neutrophils - (answer)Predominant leukocyte at work during the early stages of acute inflammation
Monocytes - (answer)Become macrophages when entering the tissue, responsible for presenting
antigens to the CD4 cell which triggers T-cell immunity and B-cell immunity.
Releases additional cytokines IL1, IL6, TNF.
,NURS 5315 UTA Exam 2 NEWEST 2026 WITH COMPLETE QUESTIONS AND
CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED
A+||BRAND NEW VERSION!!
Cytokine IL1 function - (answer)Causes fever, activates phagocytes & lymphocytes and also increases the
release of IL6a
Cytokine IL6 function - (answer)Stimulates production of acute phase reactants and promotes growth
and stimulation of RBCs
Cytokine TNF function - (answer)Causes fever, increases synthesis of proinflammatory proteins by liver,
causes muscle wasting, induces thrombosis
Cytokine growth factor function - (answer)Promotes production and maturation of neutrophils
Complement - (answer)Functions include bacterial lysis, vasodilation and increased vascular
permeability, triggers mast cell degranulation, chemotaxis, and opsonization.
Kinin - (answer)Converted to bradykinin which is responsible for pain and chemotaxis, and it increases
vascular permeability and vasodilation.
Coagulation cascade - (answer)Factor XII activates kinin. Function is to form fibrin mesh to stop bleeding
and trap micro organisms.
COX1 - (answer)Prostaglandin of arachidonic pathway. Provides gastroprotection, platelet aggregation,
fluid/electrolyte balance
COX2 - (answer)Prostaglandin of arachidonic pathway. Responsible for pain, fever, renal protection,
tissue repair, reproduction development.
COX2 inhibitors- clinical implications - (answer)Protect gastric mucosa- prevent ulcers and bleeding.
Removed from market r/t cardiac events except for Celebrex. Can impair renal function , monitor labs.
, NURS 5315 UTA Exam 2 NEWEST 2026 WITH COMPLETE QUESTIONS AND
CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED
A+||BRAND NEW VERSION!!
Arachidonic pathway purpose - (answer)Synthesis of prostaglandins
Non-selective NSAIDS - (answer)Inhibit COX1 and COX2, risk for gastric ulceration, GI bleeds, edema,
renal impairment
ASA - (answer)Blocks COX1 and COX2, also inhibits Thromboxane A2 and prostaglandins
Corticosteroids - (answer)Inhibit phospholipase A2, preventing formation of prostaglandins,
thromboxane A2, prostacyclin, and leukotrines
Thromboxane - (answer)Vasoconstriction, platelet aggregation
Prostacyclin - (answer)Vasodilation, platelet aggregation (most effective one)
Type 1 hypersensitivity - (answer)E. Immediate response to allergen, food, meds, pollen, asthma, allergic
reactions
P. IgE binds with antigen at 1st exposure. Antigen binds with this complex at 2nd exposure.
Inflammatory cascade initiates.
C.M. Urticaria, rhinitis, conjunctivitis, angioedema, anaphylaxis
Atopic disorders - (answer)Genetic Type 1 hypersensitivity, asthma, hay fever, eczema, urticaria
Type 2 hypersensitivity - (answer)E. Antibodies directed against fixed antigens on the plasma membrane
of cells
C.M. Varies depending on alloimmune or autoimmune
CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED
A+||BRAND NEW VERSION!!
HIV EIA (3rd generation immunoassay) - (answer)can use urine, saliva, or serum (most accurate), need to
wait until 12 weeks post exposure to see antibodies, >99% accurate
4th generation immunoassay- "gold standard"
measures P24 antigen
can test 10 days post exposure
Mast cell - (answer)Cellular bags of granules located in loose connective tisssue close to blood vessels.
Activation initiates inflammatory process.
Histamine - (answer)Causes vasodilation, increases vascular permeability, increases blood flow to the
site of injury- causes erythema and swelling.
Cytokines - (answer)Soluble factors that contribute to the regulation of innate or adaptive resistance by
affecting other neighboring cells. Can be pro-inflammatory or anti-inflammatory. Can react quickly or be
more delayed.
Leukotrines - (answer)Released when mast cells degranulate, prolong the inflammatory process. Cause
vasodilation, attract neutrophils, monocytes, and eosinophils.target of inhibition for singular.
Prostaglandins - (answer)Released when mast cells degranulate, are produced by the arachidonic
pathway. Cause vasodilation, platelet aggregation at site of injury, pain, and fever.
Chemotactic factors - (answer)Biochemical substance that attracts leukocyte to the site of inflammation
Neutrophils - (answer)Predominant leukocyte at work during the early stages of acute inflammation
Monocytes - (answer)Become macrophages when entering the tissue, responsible for presenting
antigens to the CD4 cell which triggers T-cell immunity and B-cell immunity.
Releases additional cytokines IL1, IL6, TNF.
,NURS 5315 UTA Exam 2 NEWEST 2026 WITH COMPLETE QUESTIONS AND
CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED
A+||BRAND NEW VERSION!!
Cytokine IL1 function - (answer)Causes fever, activates phagocytes & lymphocytes and also increases the
release of IL6a
Cytokine IL6 function - (answer)Stimulates production of acute phase reactants and promotes growth
and stimulation of RBCs
Cytokine TNF function - (answer)Causes fever, increases synthesis of proinflammatory proteins by liver,
causes muscle wasting, induces thrombosis
Cytokine growth factor function - (answer)Promotes production and maturation of neutrophils
Complement - (answer)Functions include bacterial lysis, vasodilation and increased vascular
permeability, triggers mast cell degranulation, chemotaxis, and opsonization.
Kinin - (answer)Converted to bradykinin which is responsible for pain and chemotaxis, and it increases
vascular permeability and vasodilation.
Coagulation cascade - (answer)Factor XII activates kinin. Function is to form fibrin mesh to stop bleeding
and trap micro organisms.
COX1 - (answer)Prostaglandin of arachidonic pathway. Provides gastroprotection, platelet aggregation,
fluid/electrolyte balance
COX2 - (answer)Prostaglandin of arachidonic pathway. Responsible for pain, fever, renal protection,
tissue repair, reproduction development.
COX2 inhibitors- clinical implications - (answer)Protect gastric mucosa- prevent ulcers and bleeding.
Removed from market r/t cardiac events except for Celebrex. Can impair renal function , monitor labs.
, NURS 5315 UTA Exam 2 NEWEST 2026 WITH COMPLETE QUESTIONS AND
CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED
A+||BRAND NEW VERSION!!
Arachidonic pathway purpose - (answer)Synthesis of prostaglandins
Non-selective NSAIDS - (answer)Inhibit COX1 and COX2, risk for gastric ulceration, GI bleeds, edema,
renal impairment
ASA - (answer)Blocks COX1 and COX2, also inhibits Thromboxane A2 and prostaglandins
Corticosteroids - (answer)Inhibit phospholipase A2, preventing formation of prostaglandins,
thromboxane A2, prostacyclin, and leukotrines
Thromboxane - (answer)Vasoconstriction, platelet aggregation
Prostacyclin - (answer)Vasodilation, platelet aggregation (most effective one)
Type 1 hypersensitivity - (answer)E. Immediate response to allergen, food, meds, pollen, asthma, allergic
reactions
P. IgE binds with antigen at 1st exposure. Antigen binds with this complex at 2nd exposure.
Inflammatory cascade initiates.
C.M. Urticaria, rhinitis, conjunctivitis, angioedema, anaphylaxis
Atopic disorders - (answer)Genetic Type 1 hypersensitivity, asthma, hay fever, eczema, urticaria
Type 2 hypersensitivity - (answer)E. Antibodies directed against fixed antigens on the plasma membrane
of cells
C.M. Varies depending on alloimmune or autoimmune
