NSG 233 MED SURG III EXAM 4 STUDY BUNDLE
2026 FINAL REVIEW BUNDLED SOLUTION
PACKAGE
◉ What is most important in poison emergencies?. Answer: Airway
stabilation
◉ Carbon Dioxide Poisioning. Answer: S/S: headache, dizziness,
confusion, palpitations, muscle weakness, intoxication coma, death
Priority - assess carboxyhemoglobin levels
SPO2 will appear normal
◉ Hypothermia puts people at risk for
What to remember with spontaneous v-fib?. Answer: Risk for hypoxia,
acidosis, & dysrhythmias
Spontaneous V-fib: Must rewarm to > 32.2 (90F) before defibrillation
◉ IVP who is screened?. Answer: Must screen every patient for this "Do
you feel safe at home?"
,◉ Compensatory Shock. Answer: Normal BP
>100 bpm
>20 breaths per min
PaCO2 <32
Cold, Clammy
Decreased
Confused and/or agitated
Respiratory Alkalosis
◉ Progressive Shock. Answer: Systolic <90
MAP <65
Requires fluid resuscitation to support blood pressure
>150 bpm
Rapid, Shallow Respirations
Crackles
PaO2 <80 PaCO2 >45
Mottled, Petechiae
< 0.5 mL/kg/hr
Lethargy
Metabolic Acidosis
, ◉ Irreversible Shock. Answer: Requires mechanical or pharmacologic
support
Erratic
Requires intubation and mechanical ventilation and oxygenation
Jaundice
Anuric, Requires Dialysis
Unconscious
Profound Acidosis
◉ Assessing MODS. Answer: Multiple organ dysfunction syndrome
(MODS) is altered organ function in acutely ill patients that requires
medical intervention to support continued organ function. It is another
phase in the progression of shock states. MODS may be a complication
of any form of shock, but it is most commonly seen in patients with
sepsis and is a result of inadequate tissue perfusion.
Clinical Severity Assesment Tools: These clinical assessment tools
include APACHE (Acute Physiology and Chronic Health Evaluation);
SAPS (Simplified Acute Physiology Score); PIRO (Predisposing factors,
the Infection, the host Response, and Organ dysfunction); and SOFA
score
◉ Assessing Hypovolemic Shock. Answer: Hypovolemic shock can be
caused by external fluid losses, as in traumatic blood loss, or by internal
fluid shifts, as in severe dehydration, severe edema, or ascites
Decreased intravascular volume
2026 FINAL REVIEW BUNDLED SOLUTION
PACKAGE
◉ What is most important in poison emergencies?. Answer: Airway
stabilation
◉ Carbon Dioxide Poisioning. Answer: S/S: headache, dizziness,
confusion, palpitations, muscle weakness, intoxication coma, death
Priority - assess carboxyhemoglobin levels
SPO2 will appear normal
◉ Hypothermia puts people at risk for
What to remember with spontaneous v-fib?. Answer: Risk for hypoxia,
acidosis, & dysrhythmias
Spontaneous V-fib: Must rewarm to > 32.2 (90F) before defibrillation
◉ IVP who is screened?. Answer: Must screen every patient for this "Do
you feel safe at home?"
,◉ Compensatory Shock. Answer: Normal BP
>100 bpm
>20 breaths per min
PaCO2 <32
Cold, Clammy
Decreased
Confused and/or agitated
Respiratory Alkalosis
◉ Progressive Shock. Answer: Systolic <90
MAP <65
Requires fluid resuscitation to support blood pressure
>150 bpm
Rapid, Shallow Respirations
Crackles
PaO2 <80 PaCO2 >45
Mottled, Petechiae
< 0.5 mL/kg/hr
Lethargy
Metabolic Acidosis
, ◉ Irreversible Shock. Answer: Requires mechanical or pharmacologic
support
Erratic
Requires intubation and mechanical ventilation and oxygenation
Jaundice
Anuric, Requires Dialysis
Unconscious
Profound Acidosis
◉ Assessing MODS. Answer: Multiple organ dysfunction syndrome
(MODS) is altered organ function in acutely ill patients that requires
medical intervention to support continued organ function. It is another
phase in the progression of shock states. MODS may be a complication
of any form of shock, but it is most commonly seen in patients with
sepsis and is a result of inadequate tissue perfusion.
Clinical Severity Assesment Tools: These clinical assessment tools
include APACHE (Acute Physiology and Chronic Health Evaluation);
SAPS (Simplified Acute Physiology Score); PIRO (Predisposing factors,
the Infection, the host Response, and Organ dysfunction); and SOFA
score
◉ Assessing Hypovolemic Shock. Answer: Hypovolemic shock can be
caused by external fluid losses, as in traumatic blood loss, or by internal
fluid shifts, as in severe dehydration, severe edema, or ascites
Decreased intravascular volume
