NSGl 3850/l NSG3850l Examl 3l –l
Pathophysiologyl forl Nursesl IIl Review|l
Galenl (Latestl 2026/l 2027l Update)l 100%l
Verifiedl Questionsl &l Answersl |l Gradel A
Q:l S/Sl ofl interstitiall cystitis?
Answer:
-l painl withl reliefl w/l voidingl
-l notl infectionl butl duel tol inflammation
-l "chronicl bladderl painl syndrome"l
-l don'tl knowl cause
Q:l Whatl arel riskl factorsl forl kidneyl stones?
Answer:
-l dehydration
-l urinaryl retentionl (anythingl thatl sitsl inl thel bladder)
Q:l Acutel infectionl onl chronicl kidneyl disease.l Whatl arel causesl ofl this?
Answer:
Systemicl disease/infection
,Q:l Whatl isl thel pathol ofl acutel glomerulonephritis?
Answer:
-l strepl infection
-l antigen-antibodyl responsel >l causesl damagel tol glomelularl duel tol inflammation
-l lostl itsl permeability
Q:l S/Sl ofl acutel glomerulonephritis?
Answer:
-l coffee/darkl coloredl urine
-l hematuria
Q:l Whyl arel peoplel withl CKDl atl increasedl riskl forl anemia?
Answer:
-l decreasedl productionl ofl EPO
-l whichl isl neededl forl bonel marrowl productionl ofl RBCs
Q:l S/Sl ofl nephroticl syndrome?
Answer:
-l proteinuria
-l hypoalbuminemia
-l hyperlipidemia
-l increasedl glomerularl permeabilityl (allowingl alll proteinsl tol getl through)
-l hematurial sometimes
, Q:l Lowl seruml albuminl willl resultl inl ___________l r/tl nephroticl syndrome.
Answer:
edema
*decreasedl pressurel allowsl proteinsl andl fluidl tol escape
Q:l Howl dol wel differentiatel b/wl thel 2l formsl ofl PKD?
Answer:
Liverl biopsy
Q:l Whatl %l ofl nephronl lossl isl therel inl ESRD?
Answer:
75%l nephronl loss
Q:l Whyl dol nephronsl becomel non-functionall isl ESRD/kidneyl disease?
Answer:
Bcl ofl continuedl enlargement
Q:l Whatl arel prerenall causesl ofl AKI?
Pathophysiologyl forl Nursesl IIl Review|l
Galenl (Latestl 2026/l 2027l Update)l 100%l
Verifiedl Questionsl &l Answersl |l Gradel A
Q:l S/Sl ofl interstitiall cystitis?
Answer:
-l painl withl reliefl w/l voidingl
-l notl infectionl butl duel tol inflammation
-l "chronicl bladderl painl syndrome"l
-l don'tl knowl cause
Q:l Whatl arel riskl factorsl forl kidneyl stones?
Answer:
-l dehydration
-l urinaryl retentionl (anythingl thatl sitsl inl thel bladder)
Q:l Acutel infectionl onl chronicl kidneyl disease.l Whatl arel causesl ofl this?
Answer:
Systemicl disease/infection
,Q:l Whatl isl thel pathol ofl acutel glomerulonephritis?
Answer:
-l strepl infection
-l antigen-antibodyl responsel >l causesl damagel tol glomelularl duel tol inflammation
-l lostl itsl permeability
Q:l S/Sl ofl acutel glomerulonephritis?
Answer:
-l coffee/darkl coloredl urine
-l hematuria
Q:l Whyl arel peoplel withl CKDl atl increasedl riskl forl anemia?
Answer:
-l decreasedl productionl ofl EPO
-l whichl isl neededl forl bonel marrowl productionl ofl RBCs
Q:l S/Sl ofl nephroticl syndrome?
Answer:
-l proteinuria
-l hypoalbuminemia
-l hyperlipidemia
-l increasedl glomerularl permeabilityl (allowingl alll proteinsl tol getl through)
-l hematurial sometimes
, Q:l Lowl seruml albuminl willl resultl inl ___________l r/tl nephroticl syndrome.
Answer:
edema
*decreasedl pressurel allowsl proteinsl andl fluidl tol escape
Q:l Howl dol wel differentiatel b/wl thel 2l formsl ofl PKD?
Answer:
Liverl biopsy
Q:l Whatl %l ofl nephronl lossl isl therel inl ESRD?
Answer:
75%l nephronl loss
Q:l Whyl dol nephronsl becomel non-functionall isl ESRD/kidneyl disease?
Answer:
Bcl ofl continuedl enlargement
Q:l Whatl arel prerenall causesl ofl AKI?
