NSG 3850 PATHO EXAM 4 | 105 REAL EXAM QUESTIONS & ANSWERS | 2026
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cholelithiasis - (answer)three phases: supersaturation of bile with cholesterol causing precipitation of
cholesterol, nucleation of crystals, hypermobility (stasis of bile) allowing stone growth
true - (answer)T/F: crystals of cholesterol may ignite gallstone formation
true - (answer)T/F: excessive somatostatin stimulates the gallbladder
true - (answer)T/F: crystals of cholesterol may initiate gallstone formation
cholelithiasis - (answer)risk factors of this include prolonged fasting, rapid weight loss, oral
contraceptives, diabetes, spinal cord injury, TPN, pregnancy
cholecystitis - (answer)inflammation of the gallbladder wall related to continued presence of
gallstones
,acute cholecystitis - (answer)if untreated, escalates; gangrene may occur. can rupture gallbladder and
cause peritonitis
chronic cholecystitis - (answer)predisposing factors: diabetes and obesity
chronic cholcystitis - (answer)can lead to complications like biliary sepsis and scarring/calcified
(porcelain gallbladder); causes higher risk for cancer.
cholecystitis - (answer)diagnostics: typically CT, US, or MRI, but can use HIDA, MRCA, ERCP
liver disease - (answer)S/S: jaundice (from impaired bilirubin metabolism), decreased clotting
factors, hypoalbuminemia (from ascites/edema related to low serum oncotic pressure), portal HTN,
muscle wasting, ascites, impaired absorption of vit A, D, E, K, hypertriglyceridemia
liver disease - (answer)can disrupt lipoprotein metabolism causing dyslipidemias.
"anti" - (answer)with hepatitis, what term means immunity or forming immunity to the specific hep
virus
, hep A and E - (answer)heps that are fecal oral route
Hep A - (answer)2-7 week incubation
hep A - (answer)treatment is supportive and avoiding hepatoyoxins
hep A - (answer)clinical manifestations: rashes, glomerulonephritis, angioedema, jaundice, abd pain
A and B - (answer)can get immunized from these heps
IgG(previous infection) and IgM (acute infection) - (answer)antibodies the body makes against hep
A
hep A - (answer)treatment is supportive and avoiding hepatotoxins like ETOH (alcohol)
B, C, D - (answer)heps transmitted through sex, blood, contaminated needles. mother can pass to
baby
LATEST UPDATED| GET A+
cholelithiasis - (answer)three phases: supersaturation of bile with cholesterol causing precipitation of
cholesterol, nucleation of crystals, hypermobility (stasis of bile) allowing stone growth
true - (answer)T/F: crystals of cholesterol may ignite gallstone formation
true - (answer)T/F: excessive somatostatin stimulates the gallbladder
true - (answer)T/F: crystals of cholesterol may initiate gallstone formation
cholelithiasis - (answer)risk factors of this include prolonged fasting, rapid weight loss, oral
contraceptives, diabetes, spinal cord injury, TPN, pregnancy
cholecystitis - (answer)inflammation of the gallbladder wall related to continued presence of
gallstones
,acute cholecystitis - (answer)if untreated, escalates; gangrene may occur. can rupture gallbladder and
cause peritonitis
chronic cholecystitis - (answer)predisposing factors: diabetes and obesity
chronic cholcystitis - (answer)can lead to complications like biliary sepsis and scarring/calcified
(porcelain gallbladder); causes higher risk for cancer.
cholecystitis - (answer)diagnostics: typically CT, US, or MRI, but can use HIDA, MRCA, ERCP
liver disease - (answer)S/S: jaundice (from impaired bilirubin metabolism), decreased clotting
factors, hypoalbuminemia (from ascites/edema related to low serum oncotic pressure), portal HTN,
muscle wasting, ascites, impaired absorption of vit A, D, E, K, hypertriglyceridemia
liver disease - (answer)can disrupt lipoprotein metabolism causing dyslipidemias.
"anti" - (answer)with hepatitis, what term means immunity or forming immunity to the specific hep
virus
, hep A and E - (answer)heps that are fecal oral route
Hep A - (answer)2-7 week incubation
hep A - (answer)treatment is supportive and avoiding hepatoyoxins
hep A - (answer)clinical manifestations: rashes, glomerulonephritis, angioedema, jaundice, abd pain
A and B - (answer)can get immunized from these heps
IgG(previous infection) and IgM (acute infection) - (answer)antibodies the body makes against hep
A
hep A - (answer)treatment is supportive and avoiding hepatotoxins like ETOH (alcohol)
B, C, D - (answer)heps transmitted through sex, blood, contaminated needles. mother can pass to
baby
