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NR 507 ADVANCED PATHOPHYSIOLOGY FINAL EXAM LATEST ACTUAL EXAM WITH COMPLETE QUESTIONS AND CORRECT DETAILED ANSWERS (100% VERIFIED ANSWERS) |ALREADY GRADED A+| ||PROFESSOR VERIFIED|| ||BRANDNEW!!!||

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NR 507 ADVANCED PATHOPHYSIOLOGY FINAL EXAM LATEST ACTUAL EXAM WITH COMPLETE QUESTIONS AND CORRECT DETAILED ANSWERS (100% VERIFIED ANSWERS) |ALREADY GRADED A+| ||PROFESSOR VERIFIED|| ||BRANDNEW!!!||

Instelling
NR 507 ADVANCED PATHOPHYSIOLOGY
Vak
NR 507 ADVANCED PATHOPHYSIOLOGY

Voorbeeld van de inhoud

1|Page


NR 507 ADVANCED PATHOPHYSIOLOGY FINAL EXAM LATEST 2026-2027
ACTUAL EXAM WITH COMPLETE QUESTIONS AND CORRECT DETAILED
ANSWERS (100% VERIFIED ANSWERS) |ALREADY GRADED A+| ||PROFESSOR
VERIFIED|| ||BRANDNEW!!!||

A healthcare professional student is learning about fungal
infections. What information should the student use to help
another student understand?

a. Fungal infections occur only on skin, hair, and nails.

b. Phagocytes and T lymphocytes control fungal infections.

c. Fungal infections release endotoxins.

d. Vaccines prevent fungal infections. - ANSWER-B.

The host defense against fungal infection includes the
fungistatic properties of neutrophils and macrophages. T
lymphocytes are crucial in limiting the extent of infection and
producing cytokines to further activate macrophages. Fungi
infect other tissue types such as vaginal or gastrointestinal.
Fungi do not release endotoxins; they reside in the cell walls
of gram-negative bacteria. Fungal infections are not
prevented by current vaccines.



Cytokines are thought to cause fevers by stimulating the
synthesis of which chemical mediator?

,2|Page


a. Leukotriene

b. Histamine

c. Prostaglandin

d. Bradykinin - ANSWER-C.

Cytokines seem to raise the thermoregulatory set point
through stimulation of prostaglandin synthesis and turnover
in thermoregulatory (brain) and nonthermoregulatory
(peripheral) tissues. Leukotrienes, histamine, and bradykinin
are not directly related to fever production



After sexual transmission of HIV, how soon can lab results detect
the infection?

a. 1 to 2 days

b. 4 to 10 days

c. 4 to 8 weeks

d. 2 to 4 months - ANSWER-B.

HIV RNA may be detected in the plasma by about 4 to 10
days after an acute infection and HIV.



Which cells are primary targets for HIV?

,3|Page


a. CD4+ Th cells only

b. CD4+ Th cells, macrophages, and dendritic cells

c. CD8-positive cytotoxic T (Tc) cells and plasma cells

d. CD8-positive Tc cells only - ANSWER-B.

The primary cellular targets for HIV include CD4+ Th cells,
macrophages, and dendritic cells. The other cell types are
not the primary target cells of HIV.



Which hormone prompts increased anxiety, vigilance, and arousal
during a stress response?

a. Norepinephrine

b. Epinephrine

c. Cortisol

d. Adrenocorticotropic hormone (ACTH) - ANSWER-A.

The release of norepinephrine promotes arousal, increased
vigilance, increased anxiety, and other protective emotional
responses. Epinephrine's effects are primarily on the
cardiovascular system. Cortisol's chief effects involve
metabolic processes. By inhibiting the use of metabolic
substances while promoting their formation, cortisol

, 4|Page


mobilizes glucose, amino acids, lipids, and fatty acids and
delivers them to the bloodstream. ACTH binds with specific
receptors on the adrenal glands which causes the release of
the glucocorticoids.



A student asks the healthcare professional how immunity is
decreased by stress. The professional responds that during a
stress response, the helper T (Th) 1 response is suppressed by
which hormone?

a. ACTH

b. Cortisol

c. Prolactin

d. Growth hormone - ANSWER-B.

Cortisol acts to suppress the activity of Th1 cells, which
leads to a decrease in innate immunity and the
proinflammatory response. Cortisol also stimulates the
activity of Th2 cells, which increases adaptive immunity and
the antiinflammatory response. ACTH binds with specific
receptors on the adrenal glands which causes the release of
the glucocorticoids. Prolactin is secreted in response to a
variety of stressful stimuli and acts as a second messenger

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Instelling
NR 507 ADVANCED PATHOPHYSIOLOGY
Vak
NR 507 ADVANCED PATHOPHYSIOLOGY

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