CORRECT ANSWERS (LATEST UPDATE RATED A+)
1. Dyspnea, sudden onset, sharp stabbing chest pain, Signs and sx of pul-
restlessness, impending doom, cough, Hemopty- monary embolism
sis, Tachypnea, crackles, plural friction rub, s2 or
s4 heart sound, tachycardia, low-grade fever, di-
aphoresis, petechiae over the chest and axillae,
decreased SaO2
2. High D-Dimer DVT, PE, DIC labs all
have
3. BNP, D dimer, metabolic panel, troponin Labs to draw for pul-
monary embolus
4. pulmonary angiography Diagnostic test for
PE
5. Yes. Does the patient with
PE have adequate
tissue perfusion
6. Heparin or enoxaparin, alteplase if shock of hemo- Drug therapy used
dynamic collapse for PE
7. Management for
MASSIVE PE
, NUR 265 EXAM 2 LATEST UPDATE 2022-2023COMPLETE 50 QUESTIONS AND
CORRECT ANSWERS (LATEST UPDATE RATED A+)
8. Sx and management
of submissive PE
9. Phytonadione Injectable vitamin K
10. Clotting Factors, fresh frozen plasma, and Antidote for al-
aminocaproic acid teplase
11. 2.5-3 Therapeutic INR for
PE
12. 0.8-1.1 seconds Normal INR
13. PT: 11-12.5 seconds Normal PT, PTT, and
PTT: 20-30 aPTT
APTT: 30-40
Therapeutic is 1.5 x2 baseline
14. Norepinephrine, epinephrine, dopamine Vasopressors used
when hypertension
occurs in PE
15. Acute Respiratory Failure PaO2 < 60 or PaCO2
> 45 & pH < 7.35
O2 < 90 in both cas-
es
16. acute respiratory distress syndrome (ARDS) ARF with refracto-
ry hypoxemia, lung
infiltrates, noncar-
diac associated pul-
monary edema, larg-
er molecules able to
pass through alve-
oli because of in-
, NUR 265 EXAM 2 LATEST UPDATE 2022-2023COMPLETE 50 QUESTIONS AND
CORRECT ANSWERS (LATEST UPDATE RATED A+)
jured lung tissue.
Can be caused be
shock, burns, aspi-
ration, blood transfu-
sion, etc
17. refractory hypoxemia hypoxemia that does
not respond to O2
therapy
18. Common causes of
ARDS
19. Abnormal lung sounds not heard bc edema hap- Assessing a patient
pens in interstitial spaces first. Assess VS hourly with ARDS
for hypotension, tachycardia, and dysrhythmias,
temperature abnormalities. Better outcome if temp
is elevated instead of lowered.
20. Exudative phase of ARDS Phase of ARDS with
dyspnea and tachy-
cardia. Give O2
21. Fibroproliferative phase of ARDS after inflammatory
injury(fibrosis and
pulmonary HTN) to
the lung is estab-
lished and the initiat-
ing events are con-
trolled, a process of
lung repair begins (3
to 7 days), focus on
O2 and preventing
, NUR 265 EXAM 2 LATEST UPDATE 2022-2023COMPLETE 50 QUESTIONS AND
CORRECT ANSWERS (LATEST UPDATE RATED A+)
complications. Oth-
er organ involvement
can occur
22. Resolution phase of ARDS Usually after 14
days. Fibrosis May
or May not occur.
Many survivors end
up with neuropsy-
chological deficits
23. Intubation, mechanical ventilation with PEEP or Common ARDS in-
CPAP to treat progressive hypoxemia terventions
Sedation or paralysis may be needed to meet oxy-
gen needs. Early mobility and repositioning q2h
helps lung perfusion. Probe position helps breath-
ing. Small amounts of IV fluids along with diuretics
to maintain fluid balance, pt at risk for malnutrition
24. Vitamin C, E, N, and surfactant replacement Reduces oxidative
stress on lungs for
ARDS
25. minimal leak technique: used to air can get around take stethoscope &
endotracheal tube to nose and mouth , pt can't talk put on patients neck
when cuff is inflated. above cuff. Stop in-
flating cuff when no
more air is heard.,
Then take out 1cc of
air so that the cuff is
not on too tight
26. 15-30 seconds How long should in-
tubation attempt last
27. X-ray and check end total CO2 levels. If breath Verifying that endo-
sounds and chest movement is absent on left side, tracheal tube place-
it means the tube may be in right mainstream ment
bronchus, should reposition tube without taking
out. It tube is in stomach, abdomen will be distend-