NR 507 MIDTERM EXAM 2 LATEST VERSIONS (VERSION A& B) / NR 507 WEEK 4
MIDTERM ADVANCED PATHOPHYSIOLOGY MIDTERM EXAM LATEST CHAMBERLAINE
Sickle Cell Anemia - (ANSWER)Involves a single amino acid change on the beta-chain.
Sickle Cell Anemia - (ANSWER)Increased red blood cell (RBC) hemoglobin S concentration, RBC
dehydration, acidosis, and hypoxemia.
Thalassemia - (ANSWER)May have many possible genetic mutations.
Thalassemia - (ANSWER)Ineffective erythropoiesis.
Thalassemia - (ANSWER)Occurs primarily in persons from southeast Asia and China.
Thalassemia - (ANSWER)Inherits an abnormal Hb gene from both parents.
Sickle Cell Anemia - (ANSWER)Autosomal recessive genetic disorder.
Coronary Artery Disease (CAD) - (ANSWER)Mainly the result of longstanding atherosclerosis.
Flow of blood between the heart and lungs - (ANSWER)Oxygenated blood is transported from the lungs
to the heart via the pulmonary veins.
Modifiable risk factor for CAD - (ANSWER)Obesity.
Non-modifiable risk factor for CAD - (ANSWER)Age, gender, family history.
, NR 507 MIDTERM EXAM 2 LATEST VERSIONS (VERSION A& B) / NR 507 WEEK 4
MIDTERM ADVANCED PATHOPHYSIOLOGY MIDTERM EXAM LATEST CHAMBERLAINE
Patient with sickle cell anemia - (ANSWER)At high risk for stroke.
Cells with abnormal hemoglobin - (ANSWER)More susceptible to infection by the parasite that causes
malaria.
Patient with thalassemia - (ANSWER)Not at high risk for stroke.
Geographic prevalence of sickle cell anemia and thalassemia - (ANSWER)More prevalent in Africa.
Coronary Artery Disease (CAD) - (ANSWER)Pumping ability of the heart can be impaired due to the
deprivation of oxygen.
Hypertension - (ANSWER)Has its most immediate effect on afterload.
Cor Pulmonale - (ANSWER)Right ventricular failure secondary to pulmonary hypertension.
Preload Decrease Causes - (ANSWER)Hemorrhage.
Right-Sided Heart Failure Cause - (ANSWER)The most common cause is pulmonary hypertension.
Healthy Heart Response to Increased Preload - (ANSWER)The stroke volume increases.
Stage B Heart Failure - (ANSWER)Patients with structural heart disease who have not yet developed
symptoms of heart failure.
MIDTERM ADVANCED PATHOPHYSIOLOGY MIDTERM EXAM LATEST CHAMBERLAINE
Sickle Cell Anemia - (ANSWER)Involves a single amino acid change on the beta-chain.
Sickle Cell Anemia - (ANSWER)Increased red blood cell (RBC) hemoglobin S concentration, RBC
dehydration, acidosis, and hypoxemia.
Thalassemia - (ANSWER)May have many possible genetic mutations.
Thalassemia - (ANSWER)Ineffective erythropoiesis.
Thalassemia - (ANSWER)Occurs primarily in persons from southeast Asia and China.
Thalassemia - (ANSWER)Inherits an abnormal Hb gene from both parents.
Sickle Cell Anemia - (ANSWER)Autosomal recessive genetic disorder.
Coronary Artery Disease (CAD) - (ANSWER)Mainly the result of longstanding atherosclerosis.
Flow of blood between the heart and lungs - (ANSWER)Oxygenated blood is transported from the lungs
to the heart via the pulmonary veins.
Modifiable risk factor for CAD - (ANSWER)Obesity.
Non-modifiable risk factor for CAD - (ANSWER)Age, gender, family history.
, NR 507 MIDTERM EXAM 2 LATEST VERSIONS (VERSION A& B) / NR 507 WEEK 4
MIDTERM ADVANCED PATHOPHYSIOLOGY MIDTERM EXAM LATEST CHAMBERLAINE
Patient with sickle cell anemia - (ANSWER)At high risk for stroke.
Cells with abnormal hemoglobin - (ANSWER)More susceptible to infection by the parasite that causes
malaria.
Patient with thalassemia - (ANSWER)Not at high risk for stroke.
Geographic prevalence of sickle cell anemia and thalassemia - (ANSWER)More prevalent in Africa.
Coronary Artery Disease (CAD) - (ANSWER)Pumping ability of the heart can be impaired due to the
deprivation of oxygen.
Hypertension - (ANSWER)Has its most immediate effect on afterload.
Cor Pulmonale - (ANSWER)Right ventricular failure secondary to pulmonary hypertension.
Preload Decrease Causes - (ANSWER)Hemorrhage.
Right-Sided Heart Failure Cause - (ANSWER)The most common cause is pulmonary hypertension.
Healthy Heart Response to Increased Preload - (ANSWER)The stroke volume increases.
Stage B Heart Failure - (ANSWER)Patients with structural heart disease who have not yet developed
symptoms of heart failure.
