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Regulation of Cell Cycle Entry and G1 Progression by CSF-1
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8

augustus 2024

2024/2025

A+
RECEPTOR ON CELL PROLIFERATION Mutations in the receptor were made to evaluate the roles of various tyrosine residues in receptor function (Roussel and Sherr, 1993; Roussel, 1994). Remarkably, the deletion of the kinase insert did not have any significant effect on the ability of the cell to respond to CSF-1, suggesting that the binding of all of the partners to the phosphorylated tyrosines in that domain was not essential for CSF-1-dependent proliferation. However, the substitution of ...
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